Absence of the adenosine A2A receptor or its chronic blockade decrease ethanol withdrawal-induced seizures in mice
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M. Parmentier | C. Ledent | J. Costentin | J. Vaugeois | M. Daoust | M. E. Yacoubi | Marc Parmentier | M. Yacoubi | Catherine Ledent | Jean Costentin | Martine Daoust
[1] M. Moskowitz,et al. A2A Adenosine Receptor Deficiency Attenuates Brain Injury Induced by Transient Focal Ischemia in Mice , 1999, The Journal of Neuroscience.
[2] R. Bertorelli,et al. Effects of adenosine receptor agonists and antagonists on audiogenic seizure-sensible DBA/2 mice. , 1999, European journal of pharmacology.
[3] M. Naassila,et al. Cyamemazine decreases ethanol intake in rats and convulsions during ethanol withdrawal syndrome in mice , 1998, Psychopharmacology.
[4] E. Ongini,et al. Blockade of adenosine A2A receptors by SCH 58261 results in neuroprotective effects in cerebral ischaemia in rats , 1998, Neuroreport.
[5] B. Fredholm,et al. Age-dependent changes in adenosine receptors are not modified by life-long intermittent alcohol administration , 1998, Brain Research.
[6] H. Becker,et al. Single and repeated episodes of ethanol withdrawal increase adenosine A1, but not A2A, receptor density in mouse brain , 1998, Brain Research.
[7] G. Biggio,et al. Rapid increase in basal acetylcholine release in the hippocampus of freely moving rats induced by withdrawal from long-term ethanol intoxication , 1998, Brain Research.
[8] M. J. Field,et al. Effect of chronic ethanol treatment in vivo on excitability in mouse cortical neurones in vitro , 1997, British journal of pharmacology.
[9] Sergi Ferré,et al. Adenosine–dopamine receptor–receptor interactions as an integrative mechanism in the basal ganglia , 1997, Trends in Neurosciences.
[10] S. Schiffmann,et al. Aggressiveness, hypoalgesia and high blood pressure in mice lacking the adenosine A2a receptor , 1997, Nature.
[11] B. Fredholm,et al. Long‐term Treatment with some Methylxanthines Decreases the Susceptibility to Bicuculline‐ and Pentylenetetrazol‐induced Seizures in Mice. Relationship to c‐ios Expression and Receptor Binding , 1996, The European journal of neuroscience.
[12] P. Svenningsson,et al. Adenosine (P1) receptor signalling , 1996 .
[13] B. Fredholm,et al. Pharmacology of adenosine A2A receptors. , 1996, Trends in pharmacological sciences.
[14] A. Gubitz,et al. Dual Signalling by the Adenosine A2a Receptor Involves Activation of Both N‐ and P‐Type Calcium Channels by Different G Proteins and Protein Kinases in the Same Striatal Nerve Terminals , 1996, Journal of neurochemistry.
[15] J. Ribeiro,et al. Adenosine A2 receptor-mediated excitatory actions on the nervous system , 1996, Progress in Neurobiology.
[16] A. Heinz,et al. Reduced dopaminergic function in alcoholics is related to severe dependence , 1996, Biological Psychiatry.
[17] P. Popoli,et al. Adenosine A2A receptor stimulation enhances striatal extracellular glutamate levels in rats. , 1995, European journal of pharmacology.
[18] Z. Rossetti,et al. Ethanol withdrawal is associated with increased extracellular glutamate in the rat striatum. , 1995, European journal of pharmacology.
[19] B. Fredholm,et al. Adenosine A2A receptors stimulate acetylcholine release from nerve terminals of the rat hippocampus , 1995, Neuroscience Letters.
[20] P. Singh,et al. The in vitro pharmacology of ZM 241385, a potent, non‐xanthine, A2a selective adenosine receptor antagonist , 1995, British journal of pharmacology.
[21] M. Mascia,et al. 2-Chloro-N6-cyclopentyladenosine (CCPA), an adenosine A1 receptor agonist, suppresses ethanol withdrawal syndrome in rats. , 1994, Alcohol and alcoholism.
[22] B. Fredholm,et al. Effects of adenosine A1 and A2 receptor activation on electrically evoked dopamine and acetylcholine release from rat striatal slices. , 1993, The Journal of pharmacology and experimental therapeutics.
[23] B. Fredholm,et al. The binding of the adenosine A2 receptor selective agonist [3H]CGS 21680 to rat cortex differs from its binding to rat striatum. , 1993, European journal of pharmacology.
[24] C. Thomsen,et al. Contrasting effects of adenosine A1 and A2 receptor ligands in different chemoconvulsive rodent models. , 1993, European journal of pharmacology.
[25] G. Gessa,et al. Alcohol withdrawal in rats is associated with a marked fall in extraneuronal dopamine. , 1992, Alcoholism, clinical and experimental research.
[26] J. Phillis,et al. Excitatory Transmitter Amino Acid Release from the Ischemic Rat Cerebral Cortex: Effects of Adenosine Receptor Agonists and Antagonists , 1992, Journal of neurochemistry.
[27] J. Phillis,et al. The selective A2 adenosine receptor agonist CGS 21680 enhances excitatory transmitter amino acid release from the ischemic rat cerebral cortex , 1992, Neuroscience Letters.
[28] J. Crabbe,et al. Acute dependence on depressant drugs is determined by common genes in mice. , 1991, The Journal of pharmacology and experimental therapeutics.
[29] P. Hoffman,et al. Opposite changes in turnover of noradrenaline and dopamine in the CNS of ethanol-dependent mice , 1990, Neuropharmacology.
[30] T. Dunwiddie,et al. In vivo pertussis toxin treatment attenuates some, but not all, adenosine A1 effects in slices of the rat hippocampus. , 1989, European journal of pharmacology.
[31] M. Dragunow. Purinergic mechanisms in epilepsy , 1988, Progress in Neurobiology.
[32] B. Fredholm,et al. How does adenosine inhibit transmitter release? , 1988, Trends in pharmacological sciences.
[33] M. Dragunow,et al. 8-Cyclopentyl 1,3-dimethylxanthine prolongs epileptic seizures in rats , 1987, Brain Research.
[34] A. Dolphin,et al. Pertussis toxin reverses adenosine inhibition of neuronal glutamate release , 1985, Nature.
[35] Shen Ww. Extrapyramidal symptoms associated with alcohol withdrawal. , 1984 .
[36] J. Crabbe,et al. Genetic selection for ethanol withdrawal severity: differences in replicate mouse lines. , 1983, Life sciences.
[37] V. Borg,et al. Bromocriptine in the treatment of the alcohol‐withdrawal syndrome , 1982, Acta psychiatrica Scandinavica.
[38] W. A. Hunt,et al. Reduction of striatal dopamine release during an ethanol withdrawal syndrome , 1977, Journal of neurochemistry.
[39] I. Diamond,et al. Cellular and molecular neuroscience of alcoholism. , 1997, Physiological reviews.
[40] R. Cunha,et al. Chapter 23 Purinergic regulation of acetylcholine release , 1996 .
[41] P F Morgan,et al. Caffeine-induced seizures: apparent proconvulsant action of N-ethyl carboxamidoadenosine (NECA). , 1990, Life sciences.
[42] M. Dragunow. Adenosine: the brain's natural anticonvulsant? , 1986 .
[43] T. Dunwiddie,et al. Sedative and anticonvulsant effects of adenosine analogs in mouse and rat. , 1982, The Journal of pharmacology and experimental therapeutics.