Absence of the adenosine A2A receptor or its chronic blockade decrease ethanol withdrawal-induced seizures in mice

[1]  M. Moskowitz,et al.  A2A Adenosine Receptor Deficiency Attenuates Brain Injury Induced by Transient Focal Ischemia in Mice , 1999, The Journal of Neuroscience.

[2]  R. Bertorelli,et al.  Effects of adenosine receptor agonists and antagonists on audiogenic seizure-sensible DBA/2 mice. , 1999, European journal of pharmacology.

[3]  M. Naassila,et al.  Cyamemazine decreases ethanol intake in rats and convulsions during ethanol withdrawal syndrome in mice , 1998, Psychopharmacology.

[4]  E. Ongini,et al.  Blockade of adenosine A2A receptors by SCH 58261 results in neuroprotective effects in cerebral ischaemia in rats , 1998, Neuroreport.

[5]  B. Fredholm,et al.  Age-dependent changes in adenosine receptors are not modified by life-long intermittent alcohol administration , 1998, Brain Research.

[6]  H. Becker,et al.  Single and repeated episodes of ethanol withdrawal increase adenosine A1, but not A2A, receptor density in mouse brain , 1998, Brain Research.

[7]  G. Biggio,et al.  Rapid increase in basal acetylcholine release in the hippocampus of freely moving rats induced by withdrawal from long-term ethanol intoxication , 1998, Brain Research.

[8]  M. J. Field,et al.  Effect of chronic ethanol treatment in vivo on excitability in mouse cortical neurones in vitro , 1997, British journal of pharmacology.

[9]  Sergi Ferré,et al.  Adenosine–dopamine receptor–receptor interactions as an integrative mechanism in the basal ganglia , 1997, Trends in Neurosciences.

[10]  S. Schiffmann,et al.  Aggressiveness, hypoalgesia and high blood pressure in mice lacking the adenosine A2a receptor , 1997, Nature.

[11]  B. Fredholm,et al.  Long‐term Treatment with some Methylxanthines Decreases the Susceptibility to Bicuculline‐ and Pentylenetetrazol‐induced Seizures in Mice. Relationship to c‐ios Expression and Receptor Binding , 1996, The European journal of neuroscience.

[12]  P. Svenningsson,et al.  Adenosine (P1) receptor signalling , 1996 .

[13]  B. Fredholm,et al.  Pharmacology of adenosine A2A receptors. , 1996, Trends in pharmacological sciences.

[14]  A. Gubitz,et al.  Dual Signalling by the Adenosine A2a Receptor Involves Activation of Both N‐ and P‐Type Calcium Channels by Different G Proteins and Protein Kinases in the Same Striatal Nerve Terminals , 1996, Journal of neurochemistry.

[15]  J. Ribeiro,et al.  Adenosine A2 receptor-mediated excitatory actions on the nervous system , 1996, Progress in Neurobiology.

[16]  A. Heinz,et al.  Reduced dopaminergic function in alcoholics is related to severe dependence , 1996, Biological Psychiatry.

[17]  P. Popoli,et al.  Adenosine A2A receptor stimulation enhances striatal extracellular glutamate levels in rats. , 1995, European journal of pharmacology.

[18]  Z. Rossetti,et al.  Ethanol withdrawal is associated with increased extracellular glutamate in the rat striatum. , 1995, European journal of pharmacology.

[19]  B. Fredholm,et al.  Adenosine A2A receptors stimulate acetylcholine release from nerve terminals of the rat hippocampus , 1995, Neuroscience Letters.

[20]  P. Singh,et al.  The in vitro pharmacology of ZM 241385, a potent, non‐xanthine, A2a selective adenosine receptor antagonist , 1995, British journal of pharmacology.

[21]  M. Mascia,et al.  2-Chloro-N6-cyclopentyladenosine (CCPA), an adenosine A1 receptor agonist, suppresses ethanol withdrawal syndrome in rats. , 1994, Alcohol and alcoholism.

[22]  B. Fredholm,et al.  Effects of adenosine A1 and A2 receptor activation on electrically evoked dopamine and acetylcholine release from rat striatal slices. , 1993, The Journal of pharmacology and experimental therapeutics.

[23]  B. Fredholm,et al.  The binding of the adenosine A2 receptor selective agonist [3H]CGS 21680 to rat cortex differs from its binding to rat striatum. , 1993, European journal of pharmacology.

[24]  C. Thomsen,et al.  Contrasting effects of adenosine A1 and A2 receptor ligands in different chemoconvulsive rodent models. , 1993, European journal of pharmacology.

[25]  G. Gessa,et al.  Alcohol withdrawal in rats is associated with a marked fall in extraneuronal dopamine. , 1992, Alcoholism, clinical and experimental research.

[26]  J. Phillis,et al.  Excitatory Transmitter Amino Acid Release from the Ischemic Rat Cerebral Cortex: Effects of Adenosine Receptor Agonists and Antagonists , 1992, Journal of neurochemistry.

[27]  J. Phillis,et al.  The selective A2 adenosine receptor agonist CGS 21680 enhances excitatory transmitter amino acid release from the ischemic rat cerebral cortex , 1992, Neuroscience Letters.

[28]  J. Crabbe,et al.  Acute dependence on depressant drugs is determined by common genes in mice. , 1991, The Journal of pharmacology and experimental therapeutics.

[29]  P. Hoffman,et al.  Opposite changes in turnover of noradrenaline and dopamine in the CNS of ethanol-dependent mice , 1990, Neuropharmacology.

[30]  T. Dunwiddie,et al.  In vivo pertussis toxin treatment attenuates some, but not all, adenosine A1 effects in slices of the rat hippocampus. , 1989, European journal of pharmacology.

[31]  M. Dragunow Purinergic mechanisms in epilepsy , 1988, Progress in Neurobiology.

[32]  B. Fredholm,et al.  How does adenosine inhibit transmitter release? , 1988, Trends in pharmacological sciences.

[33]  M. Dragunow,et al.  8-Cyclopentyl 1,3-dimethylxanthine prolongs epileptic seizures in rats , 1987, Brain Research.

[34]  A. Dolphin,et al.  Pertussis toxin reverses adenosine inhibition of neuronal glutamate release , 1985, Nature.

[35]  Shen Ww Extrapyramidal symptoms associated with alcohol withdrawal. , 1984 .

[36]  J. Crabbe,et al.  Genetic selection for ethanol withdrawal severity: differences in replicate mouse lines. , 1983, Life sciences.

[37]  V. Borg,et al.  Bromocriptine in the treatment of the alcohol‐withdrawal syndrome , 1982, Acta psychiatrica Scandinavica.

[38]  W. A. Hunt,et al.  Reduction of striatal dopamine release during an ethanol withdrawal syndrome , 1977, Journal of neurochemistry.

[39]  I. Diamond,et al.  Cellular and molecular neuroscience of alcoholism. , 1997, Physiological reviews.

[40]  R. Cunha,et al.  Chapter 23 Purinergic regulation of acetylcholine release , 1996 .

[41]  P F Morgan,et al.  Caffeine-induced seizures: apparent proconvulsant action of N-ethyl carboxamidoadenosine (NECA). , 1990, Life sciences.

[42]  M. Dragunow Adenosine: the brain's natural anticonvulsant? , 1986 .

[43]  T. Dunwiddie,et al.  Sedative and anticonvulsant effects of adenosine analogs in mouse and rat. , 1982, The Journal of pharmacology and experimental therapeutics.