Small artery remodeling is the most prevalent (earliest?) form of target organ damage in mild essential hypertension
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[1] E. Schiffrin,et al. Correlation of endothelial function in large and small arteries in human essential hypertension. , 2001, Journal of hypertension.
[2] E. Schiffrin,et al. Correction of arterial structure and endothelial dysfunction in human essential hypertension by the angiotensin receptor antagonist losartan. , 2000, Circulation.
[3] S. Higano,et al. Long-term follow-up of patients with mild coronary artery disease and endothelial dysfunction. , 2000, Circulation.
[4] E. Schiffrin,et al. System Journal of Renin-angiotensin-aldosterone Journal of Renin-angiotensin-aldosterone System Essential Hypertension Reduction of Resistance Artery Stiffness by Treatment with the At1-receptor Antagonist Losartan in Journal of Renin-angiotensin-aldosterone System Additional Services and Informatio , 2022 .
[5] R. Cannon,et al. Vascular effects of estrogen and vitamin E therapies in postmenopausal women. , 1999, Circulation.
[6] M. O'Rourke,et al. Pulse pressure: Is this a clinically useful risk factor? , 1999, Hypertension.
[7] E. Schiffrin,et al. Relationship between small-artery structure and systolic, diastolic and pulse pressure in essential hypertension. , 1999, Journal of hypertension.
[8] E. Schiffrin,et al. Mechanics and composition of human subcutaneous resistance arteries in essential hypertension. , 1999, Hypertension.
[9] P. Ganz,et al. Estradiol therapy combined with progesterone and endothelium-dependent vasodilation in postmenopausal women. , 1998, Circulation.
[10] E. Schiffrin,et al. Apoptosis in vasculature of spontaneously hypertensive rats: effect of an angiotensin converting enzyme inhibitor and a calcium channel antagonist. , 1998, American journal of hypertension.
[11] K. Kanmatsuse,et al. [Pathogenesis of acute coronary syndromes]. , 1998, Nihon rinsho. Japanese journal of clinical medicine.
[12] M. Safar,et al. Pulse pressure and cardiovascular mortality in normotensive and hypertensive subjects. , 1998, Hypertension.
[13] Giuseppe Mancia,et al. Risk factors associated with alterations in carotid intima—media thickness in hypertension: baseline data from the European Lacidipine Study on Atherosclerosis , 1998, Journal of hypertension.
[14] M. Omata,et al. Ostensible day-night difference of QT prolongation during long-term treatment with antiarrhythmic drugs: reappraisal of the law of "regression to the mean". , 1998, Journal of cardiovascular pharmacology.
[15] V. Miller,et al. The Effect of Estrogen Replacement Therapy on Plasma Nitric Oxide and Endothelin-1 Levels in Postmenopausal Women , 1998, Annals of Internal Medicine.
[16] T. Koob,et al. Contribution of chondroitin-dermatan sulfate-containing proteoglycans to the function of rat mesenteric arteries. , 1997, The American journal of physiology.
[17] A. Heagerty,et al. Blood pressure parameters as determinants of small artery structure in human essential hypertension. , 1997, Clinical science.
[18] D. Hayoz,et al. Time course changes of the mechanical properties of the carotid artery in renal hypertensive rats. , 1997, Hypertension.
[19] Detection. The sixth report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure (JNC VI) , 1997 .
[20] J. Mallion,et al. Left ventricular hypertrophy and arterial hypertrophy. , 1997, Advances in experimental medicine and biology.
[21] M. Mulvany,et al. Some pharmacological and elastic characteristics of isolated subcutaneous small arteries from patients with essential hypertension , 1996, Journal of hypertension.
[22] E. Schiffrin,et al. Effect of calcium channel blockade or angiotensin-converting enzyme inhibition on structure of coronary, renal, and other small arteries in spontaneously hypertensive rats. , 1996, Journal of cardiovascular pharmacology.
[23] B. Strauer,et al. Improvement of coronary flow reserve after long-term therapy with enalapril. , 1996, Hypertension.
[24] M. Creager,et al. Aging progressively impairs endothelium-dependent vasodilation in forearm resistance vessels of humans. , 1996, Hypertension.
[25] T. Lüscher,et al. Direct in situ measurement of nitric oxide in mesenteric resistance arteries. Increased decomposition by superoxide in hypertension. , 1996, Hypertension.
[26] B. Folkow. Hypertensive structural changes in systemic precapillary resistance vessels: how important are they for in vivo haemodynamics? , 1995, Journal of hypertension.
[27] A. Yeung,et al. Close relation of endothelial function in the human coronary and peripheral circulations. , 1995, Journal of the American College of Cardiology.
[28] R. Wilson,et al. Direct effects of smooth muscle relaxation and contraction on in vivo human brachial artery elastic properties. , 1995, Circulation research.
[29] J. Schwartz,et al. Prevalence and determinants of cardiac and vascular hypertrophy in hypertension. , 1995, Hypertension.
[30] E. Schiffrin,et al. Endothelium-dependent relaxation of small arteries from essential hypertensive patients: mechanisms and comparison with normotensive subjects and with responses of vessels from spontaneously hypertensive rats. , 1995, Clinical science.
[31] A. Izzard,et al. Hypertension and the vasculature: arterioles and the myogenic response , 1995, Journal of hypertension.
[32] D. Spiegelhalter,et al. Aging is associated with endothelial dysfunction in healthy men years before the age-related decline in women. , 1994, Journal of the American College of Cardiology.
[33] J. Ritter,et al. Preserved endothelium-dependent vasodilatation in patients with essential hypertension. , 1994, The New England journal of medicine.
[34] S. Kaul,et al. Functional improvement precedes structural regression of atherosclerosis. , 1994, Circulation.
[35] E. Porteri,et al. Vascular structural and functional alterations before and after the development of hypertension in SHR. , 1994, American journal of hypertension.
[36] E. Schiffrin,et al. Effects of a beta-blocker or a converting enzyme inhibitor on resistance arteries in essential hypertension. , 1994, Hypertension.
[37] M. Mulvany,et al. Histology of subcutaneous small arteries from patients with essential hypertension. , 1993, Hypertension.
[38] E. Schiffrin,et al. Morphology of resistance arteries and comparison of effects of vasoconstrictors in mild essential hypertensive patients. , 1993, Clinical and investigative medicine. Medecine clinique et experimentale.
[39] M. Mulvany,et al. Small artery structure in hypertension. Dual processes of remodeling and growth. , 1993, Hypertension.
[40] M. Mulvany,et al. Mesenteric arcade arteries contribute substantially to vascular resistance in conscious rats. , 1993, Journal of Vascular Research.
[41] V. Fuster,et al. The pathogenesis of coronary artery disease and the acute coronary syndromes (2). , 1992, The New England journal of medicine.
[42] M. Mulvany,et al. Structure and function of small arteries. , 1990, Physiological reviews.
[43] A. Quyyumi,et al. Abnormal endothelium-dependent vascular relaxation in patients with essential hypertension. , 1990, The New England journal of medicine.
[44] R. Bonow,et al. Angina due to coronary microvascular disease in hypertensive patients without left ventricular hypertrophy. , 1988, The New England journal of medicine.
[45] A. Heagerty,et al. Evidence for increased media thickness, increased neuronal amine uptake, and depressed excitation--contraction coupling in isolated resistance vessels from essential hypertensives. , 1987, Circulation research.
[46] O. Carretero,et al. The Loss of Endothelium‐Dependent Vascular Relaxation in Hypertension , 1986, Hypertension.
[47] N. Reichek,et al. Echocardiographic assessment of left ventricular hypertrophy: comparison to necropsy findings. , 1986, The American journal of cardiology.
[48] B. Folkow. Physiological aspects of primary hypertension. , 1982, Physiological reviews.