The Role of Programmed Cell Death in Cerebral Ischemia

[1]  K. Hossmann,et al.  Induction of protein inhibitor of neuronal nitric oxide synthase/cytoplasmic dynein light chain following cerebral ischemia , 1998, Neuroscience.

[2]  L. Rubin,et al.  Phosphorylation of c-Jun Is Necessary for Apoptosis Induced by Survival Signal Withdrawal in Cerebellar Granule Neurons , 1998, The Journal of Neuroscience.

[3]  K. Hossmann,et al.  Expression of nuclear redox factor ref-1 in the rat hippocampus following global ischemia induced by cardiac arrest. , 1997, Brain research. Molecular brain research.

[4]  P. Rakic,et al.  Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene , 1997, Nature.

[5]  K. Hossmann,et al.  Activation of CPP-32 protease in hippocampal neurons following ischemia and epilepsy. , 1997, Brain research. Molecular brain research.

[6]  M. Linnik,et al.  Gene Expression Induced by Cerebral Ischemia: An Apoptotic Perspective , 1997, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[7]  D. Israeli,et al.  A novel p53‐inducible gene, PAG608, encodes a nuclear zinc finger protein whose overexpression promotes apoptosis , 1997, The EMBO journal.

[8]  Guy S. Salvesen,et al.  X-linked IAP is a direct inhibitor of cell-death proteases , 1997, Nature.

[9]  Guido Kroemer,et al.  The proto-oncogene Bcl-2 and its role in regulating apoptosis , 1997, Nature Medicine.

[10]  M. Deshmukh,et al.  Programmed cell death in neurons: focus on the pathway of nerve growth factor deprivation-induced death of sympathetic neurons. , 1997, Molecular pharmacology.

[11]  C. Iadecola Bright and dark sides of nitric oxide in ischemic brain injury , 1997, Trends in Neurosciences.

[12]  L. Schwartz,et al.  Cold thoughts of death: the role of ICE proteases in neuronal cell death , 1996, Trends in Neurosciences.

[13]  C. Thompson,et al.  Apoptosis Meets Signal Transduction: Elimination of a BAD Influence , 1996, Cell.

[14]  S. Snyder,et al.  PIN: An Associated Protein Inhibitor of Neuronal Nitric Oxide Synthase , 1996, Science.

[15]  J. Avruch,et al.  Sounding the Alarm: Protein Kinase Cascades Activated by Stress and Inflammation* , 1996, The Journal of Biological Chemistry.

[16]  H. Lander,et al.  Differential Activation of Mitogen-activated Protein Kinases by Nitric Oxide-related Species* , 1996, The Journal of Biological Chemistry.

[17]  Michael E. Greenberg,et al.  Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis , 1995, Science.

[18]  Y. Itoyama,et al.  Ischemic delayed neuronal death. A mitochondrial hypothesis. , 1995, Stroke.

[19]  A. Harris,et al.  A role for the human DNA repair enzyme HAP1 in cellular protection against DNA damaging agents and hypoxic stress. , 1994, Nucleic acids research.

[20]  Jean-Claude Martinou,et al.  Overexpression of BCL-2 in transgenic mice protects neurons from naturally occurring cell death and experimental ischemia , 1994, Neuron.

[21]  T. Neumann-Haefelin,et al.  Differential Expression of the Immediate Early Genes c-Fos, c-Jun, JunB, and NGFI-B in the Rat Brain following Transient Forebrain Ischemia , 1994, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.