The tight junction protein CAR regulates cardiac conduction and cell–cell communication
暂无分享,去创建一个
Chen Chen | Alexander Schirdewan | A. Schirdewan | W. Poller | F. Rathjen | M. Gotthardt | M. Radke | R. Fischer | Michael Gotthardt | Robert Fischer | Rene Jüttner | Yu Shi | Ulrike Lisewski | Uta Wrackmeyer | Fritz Rathjen | Wolfgang Poller | Michael H. Radke | Ulrike Lisewski | R. Jüttner | Yu Shi | Uta Wrackmeyer | Cheng Chen
[1] Jamie I Vandenberg,et al. Slowed conduction and ventricular tachycardia after targeted disruption of the cardiac sodium channel gene Scn5a , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[2] S. Butz,et al. Coxsackievirus-adenovirus receptor (CAR) is essential for early embryonic cardiac development , 2005, Journal of Cell Science.
[3] J E Saffitz,et al. Connexin expression and turnover : implications for cardiac excitability. , 2000, Circulation research.
[4] J. Bergelson,et al. Isolation of a Common Receptor for Coxsackie B Viruses and Adenoviruses 2 and 5 , 1997, Science.
[5] K. Otsu,et al. Direct Association of the Gap Junction Protein Connexin-43 with ZO-1 in Cardiac Myocytes* , 1998, The Journal of Biological Chemistry.
[6] J. Seidman,et al. The T-Box transcription factor Tbx5 is required for the patterning and maturation of the murine cardiac conduction system , 2004, Development.
[7] Yiming Wu,et al. Targeted deletion of titin N2B region leads to diastolic dysfunction and cardiac atrophy , 2007, Proceedings of the National Academy of Sciences.
[8] Yuan-Tsong Chen,et al. ENU mutagenesis identifies mice with cardiac fibrosis and hepatic steatosis caused by a mutation in the mitochondrial trifunctional protein beta-subunit. , 2006, Human molecular genetics.
[9] D. Roden,et al. Nkx2-5 mutation causes anatomic hypoplasia of the cardiac conduction system. , 2004, The Journal of clinical investigation.
[10] D. Gros,et al. Mouse Model of SCN5A-Linked Hereditary Lenègre’s Disease: Age-Related Conduction Slowing and Myocardial Fibrosis , 2005, Circulation.
[11] K. Willecke,et al. Connexin-mediated cardiac impulse propagation: connexin 30.2 slows atrioventricular conduction in mouse heart. , 2006, Trends in cardiovascular medicine.
[12] Colin McKerlie,et al. A Gja1 missense mutation in a mouse model of oculodentodigital dysplasia , 2005, Development.
[13] G. Lemke,et al. A critical role for the EphA3 receptor tyrosine kinase in heart development. , 2007, Developmental biology.
[14] L. Philipson,et al. The Coxsackievirus and Adenovirus Receptor (CAR) Forms a Complex with the PDZ Domain-containing Protein Ligand-of-Numb Protein-X (LNX)* , 2003, The Journal of Biological Chemistry.
[15] A. Matsumori,et al. Electrocardiographic findings in experimental myocarditis in DBA/2 mice: complete atrioventricular block in the acute stage, low voltage of the QRS complex in the subacute stage and arrhythmias in the chronic stage. , 1984, Journal of the American College of Cardiology.
[16] B. Erdmann,et al. M line–deficient titin causes cardiac lethality through impaired maturation of the sarcomere , 2006, The Journal of cell biology.
[17] G. Fishman,et al. Electrical remodeling contributes to complex tachyarrhythmias in connexin43‐deficient mouse hearts , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[18] P. Carmeliet,et al. Overlap Syndrome of Cardiac Sodium Channel Disease in Mice Carrying the Equivalent Mutation of Human SCN5A-1795insD , 2006, Circulation.
[19] B L Langille,et al. Cardiac malformation in neonatal mice lacking connexin43. , 1995, Science.
[20] L. Philipson,et al. HCAR and MCAR: the human and mouse cellular receptors for subgroup C adenoviruses and group B coxsackieviruses. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[21] K. Nishii,et al. Mice Lacking Connexin45 Conditionally in Cardiac Myocytes Display Embryonic Lethality Similar to That of Germline Knockout Mice Without Endocardial Cushion Defect , 2003, Cell communication & adhesion.
[22] Christian C Witt,et al. Conditional Expression of Mutant M-line Titins Results in Cardiomyopathy with Altered Sarcomere Structure* , 2003, The Journal of Biological Chemistry.
[23] J. Seidman,et al. Electrophysiological abnormalities and arrhythmias in alpha MHC mutant familial hypertrophic cardiomyopathy mice. , 1997, The Journal of clinical investigation.
[24] H. Musa,et al. Connexin43 Remodeling Caused by Inhibition of Plakophilin-2 Expression in Cardiac Cells , 2007, Circulation research.
[25] I. Larre,et al. New diseases derived or associated with the tight junction. , 2007, Archives of medical research.
[26] M. Franz,et al. Targeted disruption of the Kcnq1 gene produces a mouse model of Jervell and Lange– Nielsen Syndrome , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[27] W. Edwards,et al. Pathology of the cardiac conduction system in myotonic dystrophy: a study of 12 cases. , 1988, Journal of the American College of Cardiology.
[28] Yiming Wu,et al. Cardiac Hypertrophy and Reduced Contractility in Hearts Deficient in the Titin Kinase Region , 2007, Circulation.
[29] D. Kass,et al. Sudden cardiac death in heart failure. The role of abnormal repolarization. , 1994, Circulation.
[30] Stephen N. Jones,et al. Coxsackievirus and adenovirus receptor is essential for cardiomyocyte development , 2005, Genesis.
[31] M. Welsh,et al. Adenovirus Fiber Disrupts CAR-Mediated Intercellular Adhesion Allowing Virus Escape , 2002, Cell.
[32] T. Magnuson,et al. Mice mutant for Egfr and Shp2 have defective cardiac semilunar valvulogenesis , 2000, Nature Genetics.
[33] E. Stanley,et al. Cardiac Septal and Valvular Dysmorphogenesis in Mice Heterozygous for Mutations in the Homeobox Gene Nkx2-5 , 2000, Circulation research.
[34] K. Willecke,et al. Reduced cardiac conduction velocity and predisposition to arrhythmias in connexin40-deficient mice , 1998, Current Biology.
[35] S. Varnous,et al. Mouse model carrying H222P-Lmna mutation develops muscular dystrophy and dilated cardiomyopathy similar to human striated muscle laminopathies. , 2005, Human molecular genetics.
[36] Misao Suzuki,et al. Loss of connexin45 causes a cushion defect in early cardiogenesis. , 2000, Development.
[37] Michael D. Schneider,et al. Cardiomyocyte-Specific Deletion of the Coxsackievirus and Adenovirus Receptor Results in Hyperplasia of the Embryonic Left Ventricle and Abnormalities of Sinuatrial Valves , 2006, Circulation research.
[38] E. Schenk,et al. HISTOCHEMICAL METHODS FOR SEPARATE, CONSECUTIVE AND SIMULTANEOUS DEMONSTRATION OF ACETYLCHOLINESTERASE AND NOREPINEPHRINE IN CRYOSTAT SECTIONS , 1967, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[39] B. Persson,et al. Dye‐coupling between term pregnant human myometrial cells before labor: Carboxyfluorescein versus lucifer yellow , 1995, Cell biology international.
[40] Tsuyoshi Saito,et al. Possible involvement of gap junctions in the barrier function of tight junctions of brain and lung endothelial cells , 2006, Journal of cellular physiology.
[41] H. Yagi,et al. Complete atrioventricular block associated with regional myocardial scarring in a patient with Coxsackie B2 myocarditis. , 1989, Japanese heart journal.
[42] K. Alitalo,et al. Vascular Endothelial Growth Factor-B–Deficient Mice Display an Atrial Conduction Defect , 2001, Circulation.
[43] N. Peters. Gap junctions: clarifying the complexities of connexins and conduction. , 2006, Circulation research.
[44] J. Bergelson,et al. The Coxsackievirus and Adenovirus Receptor Interacts with the Multi-PDZ Domain Protein-1 (MUPP-1) within the Tight Junction* , 2004, Journal of Biological Chemistry.
[45] Kazuko Koshiba-Takeuchi,et al. Tbx5-dependent rheostatic control of cardiac gene expression and morphogenesis. , 2006, Developmental biology.
[46] Paul J. Wang,et al. Assessment of Atrioventricular Nodal Physiology in the Mouse , 1999, Journal of Interventional Cardiac Electrophysiology.
[47] G. Morley,et al. Cardiac-Specific Loss of N-Cadherin Leads to Alteration in Connexins With Conduction Slowing and Arrhythmogenesis , 2005, Circulation research.
[48] J. Bergelson,et al. The coxsackievirus and adenovirus receptor is a transmembrane component of the tight junction , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[49] J. Nam,et al. Internalization and trafficking mechanisms of coxsackievirus B3 in HeLa cells. , 2005, Virology.
[50] G. Barsh,et al. Overexpression of angiotensin AT1 receptor transgene in the mouse myocardium produces a lethal phenotype associated with myocyte hyperplasia and heart block. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[51] M. Van Ranst,et al. Identification of alternative splice products encoded by the human coxsackie-adenovirus receptor gene. , 2001, Biochemical and biophysical research communications.
[52] E. Herlenius,et al. Ro/SSA autoantibodies directly bind cardiomyocytes, disturb calcium homeostasis, and mediate congenital heart block , 2005, The Journal of experimental medicine.
[53] G. Tomaselli,et al. Sudden Cardiac Death in Heart Failure , 1998 .
[54] David L. Paul,et al. Mice lacking connexin40 have cardiac conduction abnormalities characteristic of atrioventricular block and bundle branch block , 1998, Current Biology.
[55] J. Engel,et al. Congenital Deafness and Sinoatrial Node Dysfunction in Mice Lacking Class D L-Type Ca2+ Channels , 2000, Cell.
[56] G. Salama,et al. Transcription Enhancer Factor-1–Related Factor–Transgenic Mice Develop Cardiac Conduction Defects Associated With Altered Connexin Phosphorylation , 2004, Circulation.
[57] A. Dörner,et al. Alternatively Spliced Soluble Coxsackie-adenovirus Receptors Inhibit Coxsackievirus Infection* , 2004, Journal of Biological Chemistry.
[58] D. Roden,et al. Nkx 2-5 mutation causes anatomic hypoplasia of the cardiac conduction system , 2022 .
[59] B. A. French,et al. Gene recombination in postmitotic cells. Targeted expression of Cre recombinase provokes cardiac-restricted, site-specific rearrangement in adult ventricular muscle in vivo. , 1997, The Journal of clinical investigation.
[60] O. Berenfeld,et al. Altered Right Atrial Excitation and Propagation in Connexin40 Knockout Mice , 2005, Circulation.
[61] I. Efimov,et al. Cx43 and Dual-Pathway Electrophysiology of the Atrioventricular Node and Atrioventricular Nodal Reentry , 2003, Circulation research.