Cigarette smoke causes acute airway disease and exacerbates chronic obstructive lung disease in neonatal mice.
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N. Weissmann | O. Eickelberg | T. Conlon | A. Yildirim | M. Mall | M. Seimetz | Z. Zhou-Suckow | Jie Jia | Mariola Bednorz | Carolina Ballester Lopez | Zhou-Suckow Zhe | Oliver Eickelberg | Ali Önder Yildirim | Norbert Weissmann | Marcus A. Mall | Jie Jia | Carolina Ballester Lopez
[1] M. Mall. Unplugging Mucus in Cystic Fibrosis and Chronic Obstructive Pulmonary Disease. , 2016, Annals of the American Thoracic Society.
[2] J. Füllekrug,et al. Generation and functional characterization of epithelial cells with stable expression of SLC26A9 Cl- channels. , 2016, American journal of physiology. Lung cellular and molecular physiology.
[3] P. Gluckman,et al. Tobacco smoke exposure and respiratory morbidity in young children , 2015, Tobacco Control.
[4] J. Sterne,et al. Childhood Wheezing, Asthma, Allergy, Atopy, and Lung Function: Different Socioeconomic Patterns for Different Phenotypes , 2015, American journal of epidemiology.
[5] M. Ochs,et al. A review of recent developments and applications of morphometry/stereology in lung research. , 2015, American journal of physiology. Lung cellular and molecular physiology.
[6] G. Joos,et al. Airway Surface Dehydration Aggravates Cigarette Smoke-Induced Hallmarks of COPD in Mice , 2015, PloS one.
[7] S. Matalon,et al. CFTR and lung homeostasis. , 2014, American journal of physiology. Lung cellular and molecular physiology.
[8] O. Sommerburg,et al. Airway mucus obstruction triggers macrophage activation and matrix metalloproteinase 12-dependent emphysema. , 2014, American journal of respiratory cell and molecular biology.
[9] C. Schultz,et al. Lack of neutrophil elastase reduces inflammation, mucus hypersecretion, and emphysema, but not mucus obstruction, in mice with cystic fibrosis-like lung disease. , 2014, American journal of respiratory and critical care medicine.
[10] O. Schmid,et al. The composition of cigarette smoke determines inflammatory cell recruitment to the lung in COPD mouse models , 2013, Clinical science.
[11] C. Schultz,et al. CFTR Regulates Early Pathogenesis of Chronic Obstructive Lung Disease in βENaC-Overexpressing Mice , 2012, PloS one.
[12] Hong Wang,et al. Etanercept attenuates short-term cigarette-smoke-exposure-induced pulmonary arterial remodelling in rats by suppressing the activation of TNF-a/NF-kB signal and the activities of MMP-2 and MMP-9. , 2012, Pulmonary pharmacology & therapeutics.
[13] S. De Flora,et al. Differential carcinogenicity of cigarette smoke in mice exposed either transplacentally, early in life or in adulthood , 2012, International journal of cancer.
[14] W. Seeger,et al. Inducible NOS Inhibition Reverses Tobacco-Smoke-Induced Emphysema and Pulmonary Hypertension in Mice , 2011, Cell.
[15] H. Kauczor,et al. In vivo monitoring of cystic fibrosis-like lung disease in mice by volumetric computed tomography , 2011, European Respiratory Journal.
[16] Jilly F. Evans,et al. Pharmacological Blockade of the DP2 Receptor Inhibits Cigarette Smoke-Induced Inflammation, Mucus Cell Metaplasia, and Epithelial Hyperplasia in the Mouse Lung , 2010, Journal of Pharmacology and Experimental Therapeutics.
[17] S. Randell,et al. Airway and Lung Pathology Due to Mucosal Surface Dehydration in β-Epithelial Na+ Channel-Overexpressing Mice: Role of TNF-α and IL-4Rα Signaling, Influence of Neonatal Development, and Limited Efficacy of Glucocorticoid Treatment1 , 2009, The Journal of Immunology.
[18] V. Peinado,et al. Pulmonary vascular involvement in COPD. , 2008, Chest.
[19] B. Fischer,et al. Proteases and cystic fibrosis. , 2008, The international journal of biochemistry & cell biology.
[20] M. Cosio,et al. Mechanisms of cigarette smoke-induced COPD: insights from animal models. , 2008, American journal of physiology. Lung cellular and molecular physiology.
[21] A. Livraghi,et al. Development of chronic bronchitis and emphysema in beta-epithelial Na+ channel-overexpressing mice. , 2008, American journal of respiratory and critical care medicine.
[22] Shyam Biswal,et al. Impaired lung homeostasis in neonatal mice exposed to cigarette smoke. , 2008, American journal of respiratory cell and molecular biology.
[23] M. McGuckin,et al. Structure and function of the polymeric mucins in airways mucus. , 2008, Annual review of physiology.
[24] S. De Flora,et al. Potent carcinogenicity of cigarette smoke in mice exposed early in life. , 2007, Carcinogenesis.
[25] S. Lazarus,et al. Epithelial mucin stores are increased in the large airways of smokers with airflow obstruction. , 2006, Chest.
[26] M. De Sario,et al. Parental smoking and children’s respiratory health: independent effects of prenatal and postnatal exposure , 2006, Tobacco Control.
[27] G. Joos,et al. Cigarette smoke-induced pulmonary emphysema in scid-mice. Is the acquired immune system required? , 2005, Respiratory research.
[28] R. Pauwels,et al. Time course of cigarette smoke-induced pulmonary inflammation in mice , 2005, European Respiratory Journal.
[29] M. Eppihimer,et al. Cigarette smoke condensate induces MMP-12 gene expression in airway-like epithelia. , 2005, Biochemical and biophysical research communications.
[30] M. Cosio,et al. The development of emphysema in cigarette smoke-exposed mice is strain dependent. , 2004, American journal of respiratory and critical care medicine.
[31] J. Hogg,et al. Pathophysiology of airflow limitation in chronic obstructive pulmonary disease , 2004, The Lancet.
[32] P. Paré,et al. The nature of small-airway obstruction in chronic obstructive pulmonary disease. , 2004, The New England journal of medicine.
[33] Richard C Boucher,et al. Increased airway epithelial Na+ absorption produces cystic fibrosis-like lung disease in mice , 2004, Nature Medicine.
[34] A. M. Houghton,et al. Neutrophil elastase contributes to cigarette smoke-induced emphysema in mice. , 2003, The American journal of pathology.
[35] A. Churg,et al. A neutrophil elastase inhibitor reduces cigarette smoke-induced remodelling of lung vessels , 2003, European Respiratory Journal.
[36] L. Fabbri,et al. Goblet cell hyperplasia and epithelial inflammation in peripheral airways of smokers with both symptoms of chronic bronchitis and chronic airflow limitation. , 2000, American journal of respiratory and critical care medicine.
[37] P. Robinson,et al. Maternal cigarette smoking is associated with increased inner airway wall thickness in children who die from sudden infant death syndrome. , 1998, American journal of respiratory and critical care medicine.
[38] S. Shapiro,et al. Requirement for macrophage elastase for cigarette smoke-induced emphysema in mice. , 1997, Science.
[39] J. D. de Monchy,et al. Desensitization of the adenylyl cyclase system in peripheral blood mononuclear cells from patients with asthma three hours after allergen challenge. , 1993, The Journal of allergy and clinical immunology.
[40] J. Bousquet,et al. Eosinophilic and neutrophilic inflammation in asthma, chronic bronchitis, and chronic obstructive pulmonary disease. , 1993, The Journal of allergy and clinical immunology.
[41] L. Fabbri,et al. Activated T-lymphocytes and macrophages in bronchial mucosa of subjects with chronic bronchitis. , 1993, The American review of respiratory disease.
[42] A. Churg,et al. Effect of long-term cigarette smoke exposure on pulmonary vascular structure and function in the guinea pig. , 1991, Experimental lung research.
[43] L. Boon,et al. Dendritic cells inversely regulate airway inflammation in cigarette smoke-exposed mice. , 2016, American journal of physiology. Lung cellular and molecular physiology.
[44] S. Matalon,et al. CALL FOR PAPERS Ion Channels and Transporters in Lung Function and Disease Role of epithelial sodium channels in the regulation of lung fluid homeostasis , 2015 .
[45] L. C. Pôrto,et al. Immunohistochemical study of tumor necrosis factor-a, matrix metalloproteinase-12, and tissue inhibitor of metalloproteinase-2 on alveolar macrophages of BALB/c mice exposed to short-term cigarette smoke , 2005 .
[46] Oliver Eickelberg,et al. Cigarette smoke-induced iBALT mediates macrophage activation in a B cell-dependent manner in COPD. , 2014, American journal of physiology. Lung cellular and molecular physiology.