Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency
暂无分享,去创建一个
J. Puck | Joie Davis | R. Siegel | H. Chun | M. Lenardo | S. Skoda-Smith | Manzoor Ahmad | C. Speirs | S. Straus | J. Dale | Lixin Zheng | C. Hall | T. Atkinson | Jin Wang | Joie N. Davis
[1] J. Blenis,et al. Death Receptor Recruitment of Endogenous Caspase-10 and Apoptosis Initiation in the Absence of Caspase-8* , 2001, The Journal of Biological Chemistry.
[2] H. Chun,et al. Caspase-10 is an initiator caspase in death receptor signaling , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[3] T. Tuschl,et al. Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cells , 2001, Nature.
[4] H. Chun,et al. Autoimmune lymphoproliferative syndrome: types I, II and beyond. , 2001, Advances in experimental medicine and biology.
[5] R. Morgan,et al. Lentiviral-mediated gene transfer into human lymphocytes: role of HIV-1 accessory proteins. , 2000, Blood.
[6] R. Sékaly,et al. Early Activation of Caspases during T Lymphocyte Stimulation Results in Selective Substrate Cleavage in Nonapoptotic Cells , 1999, The Journal of experimental medicine.
[7] J. Tschopp,et al. Caspase Activation Is Required for T Cell Proliferation , 1999, The Journal of experimental medicine.
[8] J. Puck,et al. Inherited Human Caspase 10 Mutations Underlie Defective Lymphocyte and Dendritic Cell Apoptosis in Autoimmune Lymphoproliferative Syndrome Type II , 1999, Cell.
[9] M. Peter,et al. Defective CD95/APO-1/Fas signal complex formation in the human autoimmune lymphoproliferative syndrome, type Ia. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[10] R. Siegel,et al. Mature T lymphocyte apoptosis--immune regulation in a dynamic and unpredictable antigenic environment. , 1999, Annual review of immunology.
[11] R. Gilbertsen,et al. Caspase-3-like activity is necessary for IL-2 release in activated Jurkat T-cells. , 1998, Experimental cell research.
[12] J. Blenis,et al. Essential requirement for caspase-8/FLICE in the initiation of the Fas-induced apoptotic cascade , 1998, Current Biology.
[13] R. Siegel,et al. Membrane Oligomerization and Cleavage Activates the Caspase-8 (FLICE/MACHα1) Death Signal* , 1998, The Journal of Biological Chemistry.
[14] T. Miyawaki,et al. Novel Fas (CD95/APO-1) mutations in infants with a lymphoproliferative disorder. , 1998, International immunology.
[15] E. Jaffe,et al. Clincal, immunologic, and genetic features of an autoimmune lymphoproliferative syndrome associated with abnormal lymphocyte apoptosis. , 1997, Blood.
[16] L. Notarangelo,et al. Missense mutations in the Fas gene resulting in autoimmune lymphoproliferative syndrome: a molecular and immunological analysis. , 1997, Blood.
[17] K. Sullivan,et al. Fas gene mutations in the Canale-Smith syndrome, an inherited lymphoproliferative disorder associated with autoimmunity. , 1996, The New England journal of medicine.
[18] P. Schur,et al. Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease. , 1996, The Journal of clinical investigation.
[19] A. Abbas,et al. Die and Let Live: Eliminating Dangerous Lymphocytes , 1996, Cell.
[20] Warren Strober,et al. Dominant interfering fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome , 1995, Cell.
[21] F. Rieux-Laucat,et al. Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity. , 1995, Science.
[22] T. Mustelin,et al. Triggering of human natural killer cells through CD16 induces tyrosine phosphorylation of the p72syk kinase , 1994, European journal of immunology.
[23] M. Birrer,et al. Dominant negative mutant of c-Jun inhibits NF-AT transcriptional activity and prevents IL-2 gene transcription. , 1994, Journal of immunology.
[24] T. Dull,et al. kat: A high-efficiency retroviral transduction system for primary human T lymphocytes , 1994 .
[25] A. Purohit,et al. A novel function-associated molecule related to non-MHC-restricted cytotoxicity mediated by activated natural killer cells and T cells. , 1993, Journal of immunology.
[26] B. Bloom,et al. Role of NK cells in protection of mice against herpes simplex virus-1 infection. , 1987, Journal of immunology.