Rgs9-2–controlled Adaptations in the Striatum Determine the Onset of Action and Efficacy of Antidepressants in Neuropathic Pain States Accessed Terms of Use Detailed Terms
暂无分享,去创建一个
V. Zachariou | Sevasti Gaspari | Lefteris Manouras | Dimitra Terzi | Immanuel Purushothaman | Rachael L. Neve | Li Shen | Citation Mitsi | Vasiliki | Maria Stratinaki | Jian Feng
[1] G. Descalzi,et al. RGS9-2 modulates sensory and mood related symptoms of neuropathic pain , 2014, Neurobiology of Learning and Memory.
[2] M. Barrot,et al. The anxiodepressive comorbidity in chronic pain , 2014, Current opinion in anaesthesiology.
[3] R. Huganir,et al. GluA1 Phosphorylation Contributes to Postsynaptic Amplification of Neuropathic Pain in the Insular Cortex , 2014, The Journal of Neuroscience.
[4] B. Lim,et al. Decreased motivation during chronic pain requires long-term depression in the nucleus accumbens , 2014, Science.
[5] Massimo Contini,et al. Role of nucleus accumbens in neuropathic pain: Linked multi-scale evidence in the rat transitioning to neuropathic pain , 2014, PAIN®.
[6] E. Capobianco,et al. Identification of potential therapeutic targets in a model of neuropathic pain , 2014, Front. Genet..
[7] E. Nestler,et al. Prefrontal Cortical Circuit for Depression- and Anxiety-Related Behaviors Mediated by Cholecystokinin: Role of ΔFosB , 2014, The Journal of Neuroscience.
[8] K. Deisseroth,et al. Nucleus Accumbens-Specific Interventions in RGS9-2 Activity Modulate Responses to Morphine , 2014, Neuropsychopharmacology.
[9] N. Attal,et al. Psychiatric co‐morbidities in patients with chronic peripheral neuropathic pain: A multicentre cohort study , 2013, European journal of pain.
[10] L. Becerra,et al. Analogous responses in the nucleus accumbens and cingulate cortex to pain onset (aversion) and offset (relief) in rats and humans. , 2013, Journal of neurophysiology.
[11] Xue-Jun Song,et al. WNT signaling underlies the pathogenesis of neuropathic pain in rodents. , 2013, The Journal of clinical investigation.
[12] E. Nestler,et al. Regulator of G protein signaling 4 is a crucial modulator of antidepressant drug action in depression and neuropathic pain models , 2013, Proceedings of the National Academy of Sciences.
[13] Cole Trapnell,et al. TopHat2: accurate alignment of transcriptomes in the presence of insertions, deletions and gene fusions , 2013, Genome Biology.
[14] Z. Duan,et al. Interaction of G-Protein βγ Complex with Chromatin Modulates GPCR-Dependent Gene Regulation , 2013, PloS one.
[15] David G Hendrickson,et al. Differential analysis of gene regulation at transcript resolution with RNA-seq , 2012, Nature Biotechnology.
[16] Yoko Furukawa-Hibi,et al. Transcriptional suppression of the neuronal PAS domain 4 (Npas4) gene by stress via the binding of agonist‐bound glucocorticoid receptor to its promoter , 2012, Journal of neurochemistry.
[17] Damian Szklarczyk,et al. STRING v9.1: protein-protein interaction networks, with increased coverage and integration , 2012, Nucleic Acids Res..
[18] A. Abdel-Zaher,et al. Comparative evaluation of the effect of tricyclic antidepressants on inducible nitric oxide synthase expression in neuropathic pain model. , 2012, Nitric oxide : biology and chemistry.
[19] J. Micó,et al. Analgesic antidepressants promote the responsiveness of locus coeruleus neurons to noxious stimulation: Implications for neuropathic pain , 2012, PAIN.
[20] Nanxin Li,et al. Neuritin produces antidepressant actions and blocks the neuronal and behavioral deficits caused by chronic stress , 2012, Proceedings of the National Academy of Sciences.
[21] Ralf Baron,et al. Deconstructing the Neuropathic Pain Phenotype to Reveal Neural Mechanisms , 2012, Neuron.
[22] R. Neve,et al. Histone Deacetylase 5 Limits Cocaine Reward through cAMP-Induced Nuclear Import , 2012, Neuron.
[23] Yunjia Chen,et al. The Antidepressant Desipramine Is an Arrestin-biased Ligand at the α2A-Adrenergic Receptor Driving Receptor Down-regulation in Vitro and in Vivo* , 2011, The Journal of Biological Chemistry.
[24] D. Ferguson,et al. A Unique Role of RGS9-2 in the Striatum as a Positive or Negative Regulator of Opiate Analgesia , 2011, The Journal of Neuroscience.
[25] A. Vania Apkarian,et al. Pain and the brain: Specificity and plasticity of the brain in clinical chronic pain , 2011, PAIN.
[26] José María Delgado-García,et al. Associative Learning and CA3–CA1 Synaptic Plasticity Are Impaired in D1R Null, Drd1a−/− Mice and in Hippocampal siRNA Silenced Drd1a Mice , 2010, The Journal of Neuroscience.
[27] Cristina Murga,et al. The complex G protein‐coupled receptor kinase 2 (GRK2) interactome unveils new physiopathological targets , 2010, British journal of pharmacology.
[28] Román A. Corfas,et al. Loss of Inhibitory Interneurons in the Dorsal Spinal Cord and Elevated Itch in Bhlhb5 Mutant Mice , 2010, Neuron.
[29] R. Hashimoto,et al. Depression‐like behavior in the forced swimming test in PACAP‐deficient mice: amelioration by the atypical antipsychotic risperidone , 2009, Journal of neurochemistry.
[30] J. O'Donnell,et al. Postsynaptic α-2 Adrenergic Receptors are Critical for the Antidepressant-Like Effects of Desipramine on Behavior , 2009, Neuropsychopharmacology.
[31] J. Traynor,et al. RGS9-2: probing an intracellular modulator of behavior as a drug target. , 2009, Trends in pharmacological sciences.
[32] M. Barrot,et al. β2-adrenoceptors are essential for desipramine, venlafaxine or reboxetine action in neuropathic pain , 2009, Neurobiology of Disease.
[33] M. L. Macdonald,et al. Desipramine Reduces Stress-Activated Dynorphin Expression and CREB Phosphorylation in NAc Tissue , 2009, Molecular Pharmacology.
[34] Deanna L. Wallace,et al. CREB regulation of nucleus accumbens excitability mediates social isolation–induced behavioral deficits , 2009, Nature Neuroscience.
[35] P. Greengard,et al. Cocaine Regulates MEF2 to Control Synaptic and Behavioral Plasticity , 2008, Neuron.
[36] N. Gardiner,et al. Neuritin Mediates Nerve Growth Factor–Induced Axonal Regeneration and Is Deficient in Experimental Diabetic Neuropathy , 2008, Diabetes.
[37] Guanghua Xiao,et al. Histone Deacetylase 5 Epigenetically Controls Behavioral Adaptations to Chronic Emotional Stimuli , 2007, Neuron.
[38] G. Cruccu. Treatment of painful neuropathy , 2007, Current opinion in neurology.
[39] E. Nestler,et al. The Mesolimbic Dopamine Reward Circuit in Depression , 2006, Biological Psychiatry.
[40] J. Blendy. The Role of CREB in Depression and Antidepressant Treatment , 2006, Biological Psychiatry.
[41] M. Picciotto,et al. Galanin: a novel therapeutic target for depression, anxiety disorders and drug addiction? , 2006, CNS & neurological disorders drug targets.
[42] J. Blendy,et al. Antidepressant action: to the nucleus and beyond. , 2005, Trends in pharmacological sciences.
[43] M. Narita,et al. Direct evidence for the involvement of brain‐derived neurotrophic factor in the development of a neuropathic pain‐like state in mice , 2005, Journal of neurochemistry.
[44] Johannes Schwarz,et al. D2 Dopamine Receptors Colocalize Regulator of G-Protein Signaling 9-2 (RGS9-2) via the RGS9 DEP Domain, and RGS9 Knock-Out Mice Develop Dyskinesias Associated with Dopamine Pathways , 2005, The Journal of Neuroscience.
[45] S. Sands,et al. Relationship between the antinociceptive response to desipramine and changes in GABAB receptor function and subunit expression in the dorsal horn of the rat spinal cord. , 2004, Biochemical pharmacology.
[46] W. Katon,et al. Depression and pain comorbidity: a literature review. , 2003, Archives of internal medicine.
[47] E. Nestler,et al. Essential role for RGS9 in opiate action , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[48] A. Basbaum,et al. Spared nerve injury model of neuropathic pain in the mouse: a behavioral and anatomic analysis. , 2003, The journal of pain : official journal of the American Pain Society.
[49] V. Pachnis,et al. Transgenic overexpression of galanin in the dorsal root ganglia modulates pain-related behavior , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[50] H. Bading,et al. Neuronal activity‐dependent nucleocytoplasmic shuttling of HDAC4 and HDAC5 , 2003, Journal of neurochemistry.
[51] M. Zhuo,et al. Genetic Elimination of Behavioral Sensitization in Mice Lacking Calmodulin-Stimulated Adenylyl Cyclases , 2002, Neuron.
[52] R. Quirion,et al. The Neuropeptide Y (NPY) Y1 Receptor Subtype Mediates NPY-induced Antidepressant-like Activity in the Mouse Forced Swimming Test , 2002, Neuropsychopharmacology.
[53] Feng juan Wei,et al. Oxytocin mediates stress‐induced analgesia in adult mice , 2002, The Journal of physiology.
[54] E. Olson,et al. Identification of a Signal-Responsive Nuclear Export Sequence in Class II Histone Deacetylases , 2001, Molecular and Cellular Biology.
[55] P. Ernfors,et al. Reduced antinociception and plasma extravasation in mice lacking a neuropeptide Y receptor , 2001, Nature.
[56] R. Stoffel,et al. Receptor and G betagamma isoform-specific interactions with G protein-coupled receptor kinases. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[57] R. Huganir,et al. Characterization of Multiple Phosphorylation Sites on the AMPA Receptor GluR1 Subunit , 1996, Neuron.
[58] R. Dubner,et al. Efficacy of desipramine in painful diabetic neuropathy: a placebo-controlled trial , 1991, Pain.
[59] K. Blumer,et al. R9AP and R7BP: traffic cops for the RGS7 family in phototransduction and neuronal GPCR signaling. , 2009, Trends in pharmacological sciences.
[60] H. Hamm,et al. G betagamma binds histone deacetylase 5 (HDAC5) and inhibits its transcriptional co-repression activity. , 2005, The Journal of biological chemistry.
[61] S. Matsumura,et al. Development / Plasticity / Repair Pituitary Adenylate Cyclase-Activating Polypeptide Is Required for the Development of Spinal Sensitization and Induction of Neuropathic Pain , 2004 .
[62] G. Reynolds,et al. Epstein-Barr virus-encoded latent infection membrane protein 1 regulates the processing of p100 NF-kappaB2 to p52 via an IKKgamma/NEMO-independent signalling pathway. , 2003, Oncogene.
[63] J. Sondek,et al. Ggamma-like (GGL) domains: new frontiers in G-protein signaling and beta-propeller scaffolding. , 2001, Biochemical pharmacology.