C-terminal short arginine/serine repeat sequence-dependent regulation of Y14 (RBM8A) localization
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[1] P. Jennings,et al. Redirecting SR Protein Nuclear Trafficking through an Allosteric Platform. , 2017, Journal of molecular biology.
[2] S. Gygi,et al. Evidence that C9ORF72 Dipeptide Repeat Proteins Associate with U2 snRNP to Cause Mis-splicing in ALS/FTD Patients. , 2017, Cell reports.
[3] Xiang-Dong Fu,et al. Release of SR Proteins from CLK1 by SRPK1: A Symbiotic Kinase System for Phosphorylation Control of Pre-mRNA Splicing. , 2016, Molecular cell.
[4] Benjamin J. Blencowe,et al. Alternative Splicing in the Mammalian Nervous System: Recent Insights into Mechanisms and Functional Roles , 2015, Neuron.
[5] Y. Ishigaki,et al. Phosphorylation status of human RNA-binding protein 8A in cells and its inhibitory regulation by Magoh , 2015, Experimental biology and medicine.
[6] D. Ito,et al. Characterization of the dipeptide repeat protein in the molecular pathogenesis of c9FTD/ALS. , 2015, Human molecular genetics.
[7] Zhouteng Tao,et al. Nucleolar stress and impaired stress granule formation contribute to C9orf72 RAN translation-induced cytotoxicity. , 2015, Human molecular genetics.
[8] S. McKnight,et al. Poly-dipeptides encoded by the C9orf72 repeats bind nucleoli, impede RNA biogenesis, and kill cells , 2014, Science.
[9] M. Passos-Bueno,et al. A noncoding expansion in EIF4A3 causes Richieri-Costa-Pereira syndrome, a craniofacial disorder associated with limb defects. , 2014, American journal of human genetics.
[10] J. Gécz,et al. The UPF3B gene, implicated in intellectual disability, autism, ADHD and childhood onset schizophrenia regulates neural progenitor cell behaviour and neuronal outgrowth. , 2013, Human molecular genetics.
[11] H. Le Hir,et al. Crystal structure of the human eIF4AIII–CWC22 complex shows how a DEAD-box protein is inhibited by a MIF4G domain , 2013, Proceedings of the National Academy of Sciences.
[12] Kuniyoshi Iwabuchi,et al. Depletion of RNA-binding protein RBM8A (Y14) causes cell cycle deficiency and apoptosis in human cells , 2013, Experimental biology and medicine.
[13] J. Rosenfeld,et al. Contribution of copy number variants involving nonsense-mediated mRNA decay pathway genes to neuro-developmental disorders. , 2013, Human molecular genetics.
[14] Wei-Lun Chang,et al. The RNA-binding protein Y14 inhibits mRNA decapping and modulates processing body formation , 2013, Molecular biology of the cell.
[15] J. Steitz,et al. Human spliceosomal protein CWC22 plays a role in coupling splicing to exon junction complex deposition and nonsense-mediated decay , 2012, Proceedings of the National Academy of Sciences.
[16] Allan Jacobson,et al. NMD: a multifaceted response to premature translational termination , 2012, Nature Reviews Molecular Cell Biology.
[17] Niels H. Gehring,et al. CWC22 connects pre-mRNA splicing and exon junction complex assembly. , 2012, Cell reports.
[18] M. Blanchette,et al. Human CWC22 escorts the helicase eIF4AIII to spliceosomes and promotes exon junction complex assembly , 2012, Nature Structural &Molecular Biology.
[19] H. Le Hir,et al. Perispeckles are major assembly sites for the exon junction core complex , 2012, Molecular biology of the cell.
[20] H. Urlaub,et al. The cryo-EM structure of the UPF–EJC complex shows UPF1 poised toward the RNA 3′ end , 2012, Nature Structural &Molecular Biology.
[21] C. Kratz,et al. Faculty Opinions recommendation of Compound inheritance of a low-frequency regulatory SNP and a rare null mutation in exon-junction complex subunit RBM8A causes TAR syndrome. , 2012 .
[22] M. Fornerod. RS and RGG repeats as primitive proteins at the transition between the RNA and RNP worlds , 2012, Nucleus.
[23] G. Dreyfuss,et al. Specific Y14 domains mediate its nucleo-cytoplasmic shuttling and association with spliced mRNA , 2011, Scientific reports.
[24] Y. Murakami,et al. Genetic analyses using a mouse cell cycle mutant identifies magoh as a novel gene involved in Cdk regulation , 2011, Genes to cells : devoted to molecular & cellular mechanisms.
[25] E. Sakashita,et al. SR and SR-related proteins redistribute to segregated fibrillar components of nucleoli in a response to DNA damage , 2010, Nucleus.
[26] G. Biamonti,et al. Nuclear stress bodies. , 2010, Cold Spring Harbor perspectives in biology.
[27] M. Hentze,et al. The Hierarchy of Exon-Junction Complex Assembly by the Spliceosome Explains Key Features of Mammalian Nonsense-Mediated mRNA Decay , 2009, PLoS biology.
[28] A. Nanbo,et al. The exon-junction complex proteins, Y14 and MAGOH regulate STAT3 activation. , 2009, Biochemical and biophysical research communications.
[29] A. Kakita,et al. Activation of Signal Transducer and Activator of Transcription-3 in the Spinal Cord of Sporadic Amyotrophic Lateral Sclerosis Patients , 2009, Neurodegenerative Diseases.
[30] R. Loomis,et al. Chromatin binding of SRp20 and ASF/SF2 and dissociation from mitotic chromosomes is modulated by histone H3 serine 10 phosphorylation. , 2009, Molecular cell.
[31] Melissa J. Moore,et al. Pre-mRNA Processing Reaches Back toTranscription and Ahead to Translation , 2009, Cell.
[32] K. Martin,et al. mRNA Localization: Gene Expression in the Spatial Dimension , 2009, Cell.
[33] J. Gécz,et al. Mutations in UPF3B, a member of the nonsense-mediated mRNA decay complex, cause syndromic and nonsyndromic mental retardation , 2007, Nature Genetics.
[34] D. Cane,et al. The nonsense-mediated decay RNA surveillance pathway. , 2007, Annual review of biochemistry.
[35] J. Ebert,et al. The Crystal Structure of the Exon Junction Complex Reveals How It Maintains a Stable Grip on mRNA , 2006, Cell.
[36] G. Dreyfuss,et al. Binding of a novel SMG-1-Upf1-eRF1-eRF3 complex (SURF) to the exon junction complex triggers Upf1 phosphorylation and nonsense-mediated mRNA decay. , 2006, Genes & development.
[37] W. Tarn,et al. Phosphorylation of Y14 Modulates Its Interaction with Proteins Involved in mRNA Metabolism and Influences Its Methylation* , 2005, Journal of Biological Chemistry.
[38] B. Séraphin,et al. The exon junction core complex is locked onto RNA by inhibition of eIF4AIII ATPase activity , 2005, Nature Structural &Molecular Biology.
[39] M. Mann,et al. Proteomic analysis of the Arabidopsis nucleolus suggests novel nucleolar functions. , 2004, Molecular biology of the cell.
[40] B. Graveley,et al. Arginine/serine repeats are sufficient to constitute a splicing activation domain. , 2003, Nucleic acids research.
[41] A. Krainer,et al. Nuclear Export and Retention Signals in the RS Domain of SR Proteins , 2002, Molecular and Cellular Biology.
[42] A. Krainer,et al. Role of the Modular Domains of SR Proteins in Subnuclear Localization and Alternative Splicing Specificity , 1997, The Journal of cell biology.
[43] Y. Lam,et al. A new rapid method for isolating nucleoli. , 2015, Methods in molecular biology.
[44] Kuniyoshi Iwabuchi,et al. RNA-binding protein RBM8A (Y14) and MAGOH localize to centrosome in human A549 cells , 2013, Histochemistry and Cell Biology.
[45] Diamantis Sellis,et al. Phosphorylation of the arginine/serine repeats of lamin B receptor by SRPK1-insights from molecular dynamics simulations. , 2012, Biochimica et biophysica acta.