Thiol-mediated redox regulation of neutrophil apoptosis.

[1]  J. Wang,et al.  Neutrophils undergo apoptosis following ingestion of Escherichia coli. , 1996, Journal of immunology.

[2]  K. Hellstrand,et al.  Induction of apoptosis in NK cells by monocyte-derived reactive oxygen metabolites. , 1996, Journal of immunology.

[3]  J. Yodoi,et al.  Thiol-mediated redox regulation of apoptosis. Possible roles of cellular thiols other than glutathione in T cell apoptosis. , 1995, Journal of immunology.

[4]  John A. Smith.  Neutrophils, host defense, and inflammation: a double‐edged sword , 1994, Journal of leukocyte biology.

[5]  D. Green,et al.  Protein-tyrosine phosphorylation regulates apoptosis in human eosinophils and neutrophils. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[6]  S. Nagata,et al.  No requirement of reactive oxygen intermediates in Fas‐mediated apoptosis , 1994, FEBS letters.

[7]  S. Venuta,et al.  Inhibition of the differentiation of human myeloid cell lines by redox changes induced through glutathione depletion. , 1994, The Biochemical journal.

[8]  M. Mannie,et al.  Inhibition of activation‐induced death in T cell hybridomas by thiol antioxidants: oxidative stress as a mediator of apoptosis , 1994, Journal of leukocyte biology.

[9]  S Grinstein,et al.  Regulation of tyrosine phosphorylation in neutrophils by the NADPH oxidase. Role of reactive oxygen intermediates. , 1993, The Journal of biological chemistry.

[10]  S. Shah,et al.  Endonuclease-induced DNA damage and cell death in oxidant injury to renal tubular epithelial cells. , 1992, The Journal of clinical investigation.

[11]  D. Männel,et al.  Dithiocarbamates as potent inhibitors of nuclear factor kappa B activation in intact cells , 1992, The Journal of experimental medicine.

[12]  G. Rao,et al.  Active oxygen species stimulate vascular smooth muscle cell growth and proto-oncogene expression. , 1992, Circulation research.

[13]  N. Christou,et al.  Critically III Anergic Patients Demonstrate Polymorphonuclear Neutrophil Activation in the Intravascular Compartment With Decreased Cell Delivery to Inflammatory Focci , 1991, Journal of leukocyte biology.

[14]  I Nicoletti,et al.  A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry. , 1991, Journal of immunological methods.

[15]  M. Bilzer,et al.  Glutathione metabolism in activated human neutrophils: stimulation of glutathione synthesis and consumption of glutathione by reactive oxygen species , 1991, European journal of clinical investigation.

[16]  Seamus J. Martin,et al.  Ultraviolet B irradiation of human leukaemia HL-60 cells in vitro induces apoptosis. , 1991, International journal of radiation biology.

[17]  Demling Rh.  Current concepts on the adult respiratory distress syndrome. , 1990 .

[18]  M. Walport,et al.  Macrophage phagocytosis of aging neutrophils in inflammation. Programmed cell death in the neutrophil leads to its recognition by macrophages. , 1989, The Journal of clinical investigation.

[19]  L. Harker,et al.  Glutathione redox cycle protects cultured endothelial cells against lysis by extracellularly generated hydrogen peroxide. , 1984, The Journal of clinical investigation.

[20]  B. Kinon,et al.  Glutathione. VII. Differentiation among substrates by the thiol-oxidizing agent, diamide. , 1972, Biochimica et biophysica acta.

[21]  T. Buttke,et al.  Oxidative stress as a mediator of apoptosis. , 1994, Immunology today.

[22]  J. Marshall,et al.  Microbial infection and the septic response in critical surgical illness. Sepsis, not infection, determines outcome. , 1990, Archives of surgery.

[23]  P. Jocelyn.  Spectrophotometric assay of thiols. , 1987, Methods in enzymology.