Adhesion attenuates respiratory burst induced by different modes of triggering in resting or LPS-primed neutrophils.
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[1] T. Fukuda,et al. Disaggregation of lipopolysaccharide by albumin, hemoglobin or high-density lipoprotein, forming complexes that prime neutrophils for enhanced release of superoxide. , 2016, Pathogens and disease.
[2] G. Nash,et al. The Roles of Integrins in Function of Human Neutrophils after Their Migration through Endothelium into Interstitial Matrix , 2015, PloS one.
[3] E. Rock,et al. Phagocyte priming by low magnesium status: input to the enhanced inflammatory and oxidative stress responses. , 2010, Magnesium research.
[4] E. Moore,et al. Structural organization of the neutrophil NADPH oxidase: phosphorylation and translocation during priming and activation , 2005, Journal of leukocyte biology.
[5] E. Ligeti,et al. Regulation and termination of NADPH oxidase activity , 2005, Cellular and Molecular Life Sciences CMLS.
[6] A. Mócsai,et al. Syk is required for integrin signaling in neutrophils. , 2002, Immunity.
[7] M. Pabst,et al. Cross‐Linking of β2 Integrins Caused Diminished Responses of Neutrophils to Priming Agents Like Lipopolysaccharide or Tumor Necrosis Factor‐α: Possible Involvement of Tyrosine Kinase Syk , 2001, Microbiology and immunology.
[8] Mark A. Williams,et al. Integrin‐mediated signaling in human neutrophil functioning , 1999, Journal of leukocyte biology.
[9] J. Klein,et al. Extracellular matrix regulates apoptosis in human neutrophils. , 1999, Kidney international.
[10] K. Kusumoto,et al. Neutrophils responded to immobilized lipopolysaccharide in the absence of lipopolysaccharide‐binding protein , 1998, Journal of leukocyte biology.
[11] S. Suchard,et al. Syk activation is required for spreading and H2O2 release in adherent human neutrophils. , 1998, Journal of immunology.
[12] T. B. Morrison,et al. Borrelia burgdorferi outer surface protein A (OspA) activates and primes human neutrophils. , 1997, Journal of immunology.
[13] C. Cairns,et al. Magnesium attenuates the neutrophil respiratory burst in adult asthmatic patients. , 1996, Academic emergency medicine : official journal of the Society for Academic Emergency Medicine.
[14] M. Pabst,et al. Lipopolysaccharides from periodontal pathogens prime neutrophils for enhanced respiratory burst: differential effect of a synthetic lipid a precursor IVA (LA-14-PP). , 1995, Journal of periodontal research.
[15] K. Matsushima,et al. Stimulation and priming of human neutrophils by interleukin-8: cooperation with tumor necrosis factor and colony-stimulating factors. , 1991, Blood.
[16] M. Pabst,et al. Neutrophil responses to lipopolysaccharide. Effect of adherence on triggering and priming of the respiratory burst. , 1991, Journal of immunology.
[17] M. Pabst,et al. Priming of neutrophils by lipopolysaccharide for enhanced release of superoxide. Requirement for plasma but not for tumor necrosis factor-alpha. , 1990, Journal of immunology.
[18] A. Tauber,et al. Activation mechanisms of adherent human neutrophils , 1990 .
[19] C. Nathan,et al. Cytokine-induced respiratory burst of human neutrophils: dependence on extracellular matrix proteins and CD11/CD18 integrins , 1989, The Journal of cell biology.
[20] S. Weiss. Tissue destruction by neutrophils. , 1989, The New England journal of medicine.
[21] L. Akard,et al. Rapid deactivation of NADPH oxidase in neutrophils: continuous replacement by newly activated enzyme sustains the respiratory burst. , 1988, Blood.
[22] C. Nathan. Neutrophil activation on biological surfaces. Massive secretion of hydrogen peroxide in response to products of macrophages and lymphocytes. , 1987, The Journal of clinical investigation.
[23] J. Fehr,et al. Antiadhesive properties of biological surfaces are protective against stimulated granulocytes. , 1985, The Journal of clinical investigation.
[24] S. Hoffstein,et al. Surface contact inhibits neutrophil superoxide generation induced by soluble stimuli. , 1985, Laboratory investigation; a journal of technical methods and pathology.
[25] L. Mcphail,et al. Priming of neutrophils for enhanced release of oxygen metabolites by bacterial lipopolysaccharide. Evidence for increased activity of the superoxide-producing enzyme , 1984, The Journal of experimental medicine.
[26] J. Fehr,et al. Role of cell surface contact in the kinetics of superoxide production by granulocytes. , 1983, The Journal of clinical investigation.
[27] G. Barlow,et al. Studies on a lipopolysaccharide from Escherichia coli. Heterogeneity and mechanism of reversible inactivation by sodium deoxycholate. , 1969, Biochemistry.
[28] E. Kownatzki,et al. Adherence-induced enhancement of the oxidative burst of human neutrophilic granulocytes: Effects of the surface coat and of divalent cations , 2005, Agents and Actions.