Cardiovascular disease and risk of incident diabetes mellitus: Findings from the Hispanic Community Health Study/Study of Latinos (HCHS/SOL)

Studies have reported an association between prevalent cardiovascular disease (CVD) and risk of diabetes mellitus (DM). However, factors that may explain the association remain unclear. We examined the association of prevalent CVD with incident DM and assessed whether weight gain and medication use may explain the association.

[1]  Kim M Pfotenhauer,et al.  Classification and Diagnosis of Diabetes. , 2022, Primary care.

[2]  N. Schneiderman Hispanic Community Health Study/Study of Latinos , 2020, Encyclopedia of Behavioral Medicine.

[3]  T. Biering-Sørensen,et al.  Left ventricular concentric geometry predicts incident diabetes mellitus independent of established risk factors in the general population: the Copenhagen City Heart Study , 2019, Cardiovascular Diabetology.

[4]  E. T. Tchetgen Tchetgen,et al.  Evaluation of Medication-mediated Effects in Pharmacoepidemiology , 2017, Epidemiology.

[5]  2. Classification and Diagnosis of Diabetes , 2016, Diabetes Care.

[6]  O. Ganda Statin-induced diabetes: incidence, mechanisms, and implications , 2016, F1000Research.

[7]  S. Yusuf,et al.  Cholesterol Lowering in Intermediate-Risk Persons without Cardiovascular Disease. , 2016, The New England journal of medicine.

[8]  J. Larsen,et al.  Post-Transplant Diabetes Mellitus: Causes, Treatment, and Impact on Outcomes. , 2016, Endocrine reviews.

[9]  2. Classification and Diagnosis of Diabetes , 2015, Diabetes Care.

[10]  Peter Diem,et al.  Role of diuretics, β blockers, and statins in increasing the risk of diabetes in patients with impaired glucose tolerance: reanalysis of data from the NAVIGATOR study , 2014, BMJ : British Medical Journal.

[11]  J. Krischer,et al.  Heart Failure as a Risk Factor for Diabetes Mellitus , 2013, Cardiology.

[12]  Peter Diem,et al.  Role of diuretics, β blockers, and statins in increasing the risk of diabetes in patients with impaired glucose tolerance: reanalysis of data from the NAVIGATOR study , 2013, BMJ.

[13]  E. Rimm,et al.  Alternative dietary indices both strongly predict risk of chronic disease. , 2012, The Journal of nutrition.

[14]  Ilya Shpitser,et al.  Semiparametric Theory for Causal Mediation Analysis: efficiency bounds, multiple robustness, and sensitivity analysis. , 2012, Annals of statistics.

[15]  J. Manson,et al.  Statin use and risk of diabetes mellitus in postmenopausal women in the Women's Health Initiative. , 2012, Archives of internal medicine.

[16]  A. Vaag,et al.  Heart failure severity, as determined by loop diuretic dosages, predicts the risk of developing diabetes after myocardial infarction: a nationwide cohort study , 2010, European journal of heart failure.

[17]  E. Moler,et al.  Cardiovascular disease, heart failure, chronic kidney disease and depression independently increase the risk of incident diabetes , 2010, Diabetologia.

[18]  Lloyd E Chambless,et al.  Design and implementation of the Hispanic Community Health Study/Study of Latinos. , 2010, Annals of epidemiology.

[19]  Yasuo Ohashi,et al.  Statins and risk of incident diabetes: a collaborative meta-analysis of randomised statin trials , 2010, The Lancet.

[20]  W. Elliott Changes in Serum Potassium Mediate Thiazide-Induced Diabetes , 2010 .

[21]  S. Grundy,et al.  Harmonizing the metabolic syndrome: a joint interim statement of the International Diabetes Federation Task Force on Epidemiology and Prevention; National Heart, Lung, and Blood Institute; American Heart Association; World Heart Federation; International Atherosclerosis Society; and International As , 2009, Circulation.

[22]  E. Boerwinkle,et al.  Lack of Correlation Between Thiazide‐Induced Hyperglycemia and Hypokalemia: Subgroup Analysis of Results from the Pharmacogenomic Evaluation of Antihypertensive Responses (PEAR) Study , 2009, Pharmacotherapy.

[23]  F. Koehler,et al.  Daily walking performance as an independent predictor of advanced heart failure: Prediction of exercise capacity in chronic heart failure. , 2009, American heart journal.

[24]  L. Appel,et al.  Changes in Serum Potassium Mediate Thiazide-Induced Diabetes , 2008, Hypertension.

[25]  Jackson T. Wright,et al.  Thiazide-induced dysglycemia: call for research from a working group from the national heart, lung, and blood institute. , 2008, Hypertension.

[26]  Peter C Austin,et al.  Propensity-score matching in the cardiovascular surgery literature from 2004 to 2006: a systematic review and suggestions for improvement. , 2007, The Journal of thoracic and cardiovascular surgery.

[27]  S. Bangalore,et al.  A meta-analysis of 94,492 patients with hypertension treated with beta blockers to determine the risk of new-onset diabetes mellitus. , 2007, The American journal of cardiology.

[28]  Benjamin J. Epstein,et al.  Seventh Report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure , 2007 .

[29]  M. Nakata,et al.  Effects of statins on the adipocyte maturation and expression of glucose transporter 4 (SLC2A4): implications in glycaemic control , 2006, Diabetologia.

[30]  V. Roger,et al.  Prevalence and secular trends of excess body weight and impact on outcomes after myocardial infarction in the community. , 2004, Chest.

[31]  D. Rubin Using Propensity Scores to Help Design Observational Studies: Application to the Tobacco Litigation , 2001, Health Services and Outcomes Research Methodology.

[32]  Daniel W. Jones,et al.  Seventh report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure. , 2003, Hypertension.

[33]  James P. Boyle,et al.  A Dynamic Markov Model for Forecasting Diabetes Prevalence in the United States through 2050 , 2003, Health care management science.

[34]  P D Cleary,et al.  Validating recommendations for coronary angiography following acute myocardial infarction in the elderly: a matched analysis using propensity scores. , 2001, Journal of clinical epidemiology.

[35]  D S Bell,et al.  Hypertension and antihypertensive therapy as risk factors for type 2 diabetes mellitus. , 2000, The New England journal of medicine.

[36]  H. King,et al.  Global Burden of Diabetes, 1995–2025: Prevalence, numerical estimates, and projections , 1998, Diabetes Care.

[37]  A. Amos,et al.  The Rising Global Burden of Diabetes and its Complications: Estimates and Projections to the Year 2010 , 1997, Diabetic medicine : a journal of the British Diabetic Association.

[38]  M. Farrer,et al.  Patients undergoing coronary artery bypass graft surgery are at high risk of impaired glucose tolerance and diabetes mellitus during the first postoperative year. , 1995, Metabolism: clinical and experimental.

[39]  H. Lithell,et al.  Metabolic effects of pindolol and propranolol in a double-blind cross-over study in hypertensive patients. , 1992, Blood pressure.

[40]  Megan Gershon,et al.  Innervation of the pancreas by neurons in the gut , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[41]  K. Ratzmann,et al.  [Adrenergic modulation of basal insulin secretion in obesity]. , 1974, Zeitschrift fur die gesamte innere Medizin und ihre Grenzgebiete.

[42]  D. Porte,et al.  Adrenergic Modulation of Basal Insulin Secretion in Man , 1973, Diabetes.

[43]  E. Cerasi,et al.  Effect of adrenergic blocking agents on insulin response to glucose infusion in man. , 1972, Acta endocrinologica.