Stimulation by Dendritic Cells That Binds B7-DC and Potentiates T Cell Naturally Occurring Human IgM Antibody

[1]  Gerold Schuler,et al.  Dendritic Cells as Vectors for Therapy , 2001, Cell.

[2]  Aaron J. Johnson,et al.  Monomeric Class I Molecules Mediate TCR/CD3ε/CD8 Interaction on the Surface of T Cells , 2001, The Journal of Immunology.

[3]  D. Pardoll,et al.  B7-Dc, a New Dendritic Cell Molecule with Potent Costimulatory Properties for T Cells , 2001, The Journal of experimental medicine.

[4]  G. Zhu,et al.  B7-H1 costimulation preferentially enhances CD28-independent T-helper cell function. , 2001, Blood.

[5]  G. Freeman,et al.  PD-L2 is a second ligand for PD-1 and inhibits T cell activation , 2001, Nature Immunology.

[6]  J. Gutiérrez-Ramos,et al.  The expanding B7 superfamily: Increasing complexity in costimulatory signals regulating T cell function , 2001, Nature Immunology.

[7]  B. Riggs,et al.  Regulation of osteoclastogenesis and RANK expression by TGF‐β1 , 2001 .

[8]  L. Pease,et al.  Human monoclonal antibodies reactive to oligodendrocytes promote remyelination in a model of multiple sclerosis. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[9]  C. Figdor,et al.  Identification of DC-SIGN, a Novel Dendritic Cell–Specific ICAM-3 Receptor that Supports Primary Immune Responses , 2000, Cell.

[10]  R. Steinman,et al.  Trance, a Tumor Necrosis Factor Family Member, Enhances the Longevity and Adjuvant Properties of Dendritic Cells in Vivo , 2000, The Journal of experimental medicine.

[11]  Y. Niitsu,et al.  A case of malignant lymphoma producing autoantibody against platelet glycoprotein Ib. , 1999, International journal of hematology.

[12]  R. Steinman,et al.  TRANCE, a Tumor Necrosis Factor Family Member Critical for CD40 Ligand–independent T Helper Cell Activation , 1999, The Journal of experimental medicine.

[13]  O. Bairey,et al.  Cutaneous necrosis as a terminal paraneoplastic thromboembolic event in a patient with non‐Hodgkin’s lymphoma , 1999, Journal of internal medicine.

[14]  R. Steinman,et al.  TRANCE, a TNF family member, is differentially expressed on T cell subsets and induces cytokine production in dendritic cells. , 1999, Journal of immunology.

[15]  M. Davis,et al.  A receptor/cytoskeletal movement triggered by costimulation during T cell activation. , 1998, Science.

[16]  T. Jakob,et al.  Activation of cutaneous dendritic cells by CpG-containing oligodeoxynucleotides: a role for dendritic cells in the augmentation of Th1 responses by immunostimulatory DNA. , 1998, Journal of immunology.

[17]  Colin R. F. Monks,et al.  Three-dimensional segregation of supramolecular activation clusters in T cells , 1998, Nature.

[18]  R. Jonsson,et al.  Transfer of human serum IgG to nonobese diabetic Igmu null mice reveals a role for autoantibodies in the loss of secretory function of exocrine tissues in Sjögren's syndrome. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[19]  R. Vabulas,et al.  Bacterial DNA and immunostimulatory CpG oligonucleotides trigger maturation and activation of murine dendritic cells , 1998, European journal of immunology.

[20]  R. Steinman,et al.  Dendritic cells and the control of immunity , 1998, Nature.

[21]  R. Steinman,et al.  TRANCE (Tumor Necrosis Factor [TNF]-related Activation-induced Cytokine), a New TNF Family Member Predominantly Expressed in T cells, Is a Dendritic Cell–specific Survival Factor , 1997, The Journal of experimental medicine.

[22]  Antonio Lanzavecchia,et al.  Inflammatory stimuli induce accumulation of MHC class II complexes on dendritic cells , 1997, Nature.

[23]  S. Kaveri,et al.  Pooled normal human polyspecific IgM contains neutralizing anti-idiotypes to IgG autoantibodies of autoimmune patients and protects from experimental autoimmune disease. , 1997, Blood.

[24]  E. Simpson,et al.  B7-1 and B7-2 have overlapping, critical roles in immunoglobulin class switching and germinal center formation. , 1997, Immunity.

[25]  J. Bluestone,et al.  The Complexities of T‐Cell Co‐stimulation: CD28 and Beyond , 1996, Immunological reviews.

[26]  K. Shortman,et al.  Thymic dendritic cell precursors: relationship to the T lymphocyte lineage and phenotype of the dendritic cell progeny , 1996, The Journal of experimental medicine.

[27]  J. Bluestone,et al.  CD28/B7 system of T cell costimulation. , 1996, Annual review of immunology.

[28]  M. Rodriguez,et al.  A monoclonal autoantibody that promotes central nervous system remyelination in a model of multiple sclerosis is a natural autoantibody encoded by germline immunoglobulin genes. , 1995, Journal of immunology.

[29]  P. Linsley,et al.  CD80 (B7) and CD86 (B70) provide similar costimulatory signals for T cell proliferation, cytokine production, and generation of CTL. , 1995, Journal of immunology.

[30]  R. Steinman,et al.  The tissue distribution of the B7-2 costimulator in mice: abundant expression on dendritic cells in situ and during maturation in vitro , 1994, The Journal of experimental medicine.

[31]  M. Rodriguez,et al.  Monoclonal autoantibodies promote central nervous system repair in an animal model of multiple sclerosis , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[32]  P. Linsley,et al.  Regulation of immunostimulatory function and costimulatory molecule (B7-1 and B7-2) expression on murine dendritic cells. , 1994, Journal of immunology.

[33]  Kristin A. Hogquist,et al.  T cell receptor antagonist peptides induce positive selection , 1994, Cell.

[34]  J. Gribben,et al.  Murine B7-2, an alternative CTLA4 counter-receptor that costimulates T cell proliferation and interleukin 2 production , 1993, The Journal of experimental medicine.

[35]  J. Gribben,et al.  Cloning of B7-2: a CTLA-4 counter-receptor that costimulates human T cell proliferation. , 1993, Science.

[36]  R. Steinman,et al.  Generation of large numbers of dendritic cells from mouse bone marrow cultures supplemented with granulocyte/macrophage colony-stimulating factor , 1992, The Journal of experimental medicine.

[37]  R. Kyle.  Monoclonal proteins in neuropathy. , 1992, Neurologic clinics.

[38]  P. Dyck,et al.  Neuropathy associated with monoclonal gammopathies of undetermined significance , 1991, Annals of neurology.

[39]  A. Heimberger,et al.  Induction by antigen of intrathymic apoptosis of CD4+CD8+TCRlo thymocytes in vivo. , 1990, Science.

[40]  J. Campbell,et al.  Ataxic sensory neuropathy and dorsal root ganglionitis associated with Sjögren's syndrome , 1990, Annals of neurology.

[41]  Moses Rodriguez,et al.  Immunoglobulins promote remyelination in the central nervous system , 1990, Annals of neurology.

[42]  R. W. Plunkett,et al.  Complement-mediated autoimmune thrombocytopenia. Monoclonal IgM antiplatelet antibody associated with lymphoreticular malignant disease. , 1987, The New England journal of medicine.

[43]  Moses Rodriguez,et al.  Dysglobulinemic neuropathy: Absence of immunoglobulin within myelin sheaths , 1986, Annals of neurology.

[44]  S. B. Wilson,et al.  Platelet antibodies of the IgM class in immune thrombocytopenic purpura. , 1985, The Journal of clinical investigation.

[45]  R. Hughes,et al.  Microangiopathy of vasa nervorum in dysglobulinemic neuropathy , 1984, Annals of neurology.

[46]  P. Dyck,et al.  Prevalence of monoclonal protein in peripheral neuropathy , 1981, Neurology.