HDAC3 overexpression and colon cancer cell proliferation and differentiation
暂无分享,去创建一个
Charles Giardina | E. Noonan | Cassandra A. Godman | C. Giardina | D. Rosenberg | Theodore P Rasmussen | Daniel W Rosenberg | Colleen C Spurling | Cassandra A Godman | Emily J Noonan | T. Rasmussen | C. Godman
[1] M. Fishman,et al. Phase I trial of histone deacetylase inhibition by valproic acid followed by the topoisomerase II inhibitor epirubicin in advanced solid tumors: a clinical and translational study. , 2007, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[2] E. Sausville,et al. Phase 1 and pharmacologic study of MS-275, a histone deacetylase inhibitor, in adults with refractory and relapsed acute leukemias. , 2007, Blood.
[3] Y. Iwamoto,et al. The Effects of Histone Deacetylase Inhibitors on the Induction of Differentiation in Chondrosarcoma Cells , 2007, Clinical Cancer Research.
[4] B. Hinnebusch,et al. Transcriptional activation of the enterocyte differentiation marker intestinal alkaline phosphatase is associated with changes in the acetylation state of histone H3 at a specific site within its promoter region in vitro , 2003, Journal of Gastrointestinal Surgery.
[5] Yi Tang,et al. Tip60-dependent acetylation of p53 modulates the decision between cell-cycle arrest and apoptosis. , 2006, Molecular cell.
[6] Ronen Marmorstein,et al. Acetylation of the p53 DNA-binding domain regulates apoptosis induction. , 2006, Molecular cell.
[7] C. Pérez-Plasencia,et al. A Proof-Of-Principle Study of Epigenetic Therapy Added to Neoadjuvant Doxorubicin Cyclophosphamide for Locally Advanced Breast Cancer , 2006, PloS one.
[8] O. O’Connor. Clinical experience with the novel histone deacetylase inhibitor vorinostat (suberoylanilide hydroxamic acid) in patients with relapsed lymphoma , 2006, British Journal of Cancer.
[9] Michael Rytting,et al. Phase 1/2 study of the combination of 5-aza-2'-deoxycytidine with valproic acid in patients with leukemia. , 2006, Blood.
[10] E. Noonan,et al. Circumvention and reactivation of the p53 oncogene checkpoint in mouse colon tumors. , 2006, Biochemical pharmacology.
[11] P. Houghton,et al. Phase I study of depsipeptide in pediatric patients with refractory solid tumors: a Children's Oncology Group report. , 2006, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[12] Andrew J. Wilson,et al. Histone Deacetylase 3 (HDAC3) and Other Class I HDACs Regulate Colon Cell Maturation and p21 Expression and Are Deregulated in Human Colon Cancer* , 2006, Journal of Biological Chemistry.
[13] M. Esteller,et al. New therapeutic targets in cancer: the epigenetic connection , 2006, Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico.
[14] C. Steidl,et al. The histone deacetylase (HDAC) inhibitor valproic acid as monotherapy or in combination with all‐trans retinoic acid in patients with acute myeloid leukemia , 2006, Cancer.
[15] L. Schwartz,et al. Potential role of histone deacetylase inhibitors in mesothelioma: clinical experience with suberoylanilide hydroxamic acid. , 2006, Clinical lung cancer.
[16] S. Minucci,et al. Histone deacetylase inhibitors and the promise of epigenetic (and more) treatments for cancer , 2006, Nature Reviews Cancer.
[17] M. Duvic,et al. Selective induction of apoptosis by histone deacetylase inhibitor SAHA in cutaneous T-cell lymphoma cells: relevance to mechanism of therapeutic action. , 2005, The Journal of investigative dermatology.
[18] Hiroko Yamashita,et al. Quantitation of HDAC1 mRNA Expression in Invasive Carcinoma of the Breast* , 2005, Breast Cancer Research and Treatment.
[19] M. Dokmanovic,et al. Prospects: Histone deacetylase inhibitors , 2005, Journal of cellular biochemistry.
[20] K. Subbaramaiah,et al. Histone Deacetylase Inhibitors Suppress the Induction of c-Jun and Its Target Genes Including COX-2* , 2005, Journal of Biological Chemistry.
[21] R. Casadio,et al. Histone deacetylase 1: a target of 9-hydroxystearic acid in the inhibition of cell growth in human colon cancer Published, JLR Papers in Press, February 16, 2005. DOI 10.1194/jlr.M400424-JLR200 , 2005, Journal of Lipid Research.
[22] Gesine Hansen,et al. Comparative application of antibody and gene array for expression profiling in human squamous cell lung carcinoma. , 2005, Lung cancer.
[23] F. Dequiedt,et al. Effect of inhibitors of histone deacetylase on the induction of cell differentiation in murine and human erythroleukemia cell lines , 2005, Anti-cancer drugs.
[24] Li Yu,et al. [DNA methylation and cancer]. , 2005, Zhonghua nei ke za zhi.
[25] L. Yin,et al. Cyclooxygenase-2 Regulation in Colon Cancer Cells , 2005, Journal of Biological Chemistry.
[26] Suk Woo Nam,et al. Increased expression of histone deacetylase 2 is found in human gastric cancer , 2005, APMIS : acta pathologica, microbiologica, et immunologica Scandinavica.
[27] M. Salto‐Tellez,et al. Inhibition of histone deacetylase 2 increases apoptosis and p21Cip1/WAF1 expression, independent of histone deacetylase 1 , 2005, Cell Death and Differentiation.
[28] S. Voelter-Mahlknecht,et al. The histone deacetylase inhibitor suberoylanilide hydroxamic acid down-regulates expression levels of Bcr-abl, c-Myc and HDAC3 in chronic myeloid leukemia cell lines. , 2005, International journal of molecular medicine.
[29] C. Benz,et al. Clinical development of histone deacetylase inhibitors as anticancer agents. , 2005, Annual review of pharmacology and toxicology.
[30] Jing Lin,et al. Trefoil factor family-3 is associated with aggressive behavior of colon cancer cells , 2005, Clinical & Experimental Metastasis.
[31] Xinxing Wu,et al. Histone deacetylase inhibitor, Trichostatin A, activates p21WAF1/CIP1 expression through downregulation of c-myc and release of the repression of c-myc from the promoter in human cervical cancer cells. , 2004, Biochemical and biophysical research communications.
[32] C. Pilarsky,et al. Identification and validation of commonly overexpressed genes in solid tumors by comparison of microarray data. , 2004, Neoplasia.
[33] L. Yin,et al. The p50-p50 NF-κB complex as a stimulus-specific repressor of gene activation , 2004, Molecular and Cellular Biochemistry.
[34] M. Gorospe,et al. Acquisition of Resistance to Butyrate Enhances Survival after Stress and Induces Malignancy of Human Colon Carcinoma Cells , 2004, Cancer Research.
[35] M. Romero,et al. The human tumour suppressor gene SLC5A8 expresses a Na+–monocarboxylate cotransporter , 2004, The Journal of physiology.
[36] Hing Y Leung,et al. Upregulation and Nuclear Recruitment of HDAC1 in Hormone Refractory Prostate Cancer , 2004, The Prostate.
[37] Ping Zhu,et al. Induction of HDAC2 expression upon loss of APC in colorectal tumorigenesis. , 2004, Cancer cell.
[38] L. Yin,et al. Butyrate suppresses Cox-2 activation in colon cancer cells through HDAC inhibition. , 2004, Biochemical and biophysical research communications.
[39] L. Yin,et al. The p50-p50 NF-kappaB complex as a stimulus-specific repressor of gene activation. , 2004, Molecular and cellular biochemistry.
[40] J. Mariadason,et al. Repression of MUC2 gene expression by butyrate, a physiological regulator of intestinal cell maturation , 2003, Oncogene.
[41] M. Lazar,et al. The N-CoR/Histone Deacetylase 3 Complex Is Required for Repression by Thyroid Hormone Receptor , 2003, Molecular and Cellular Biology.
[42] C. Plass,et al. SLC5A8, a sodium transporter, is a tumor suppressor gene silenced by methylation in human colon aberrant crypt foci and cancers , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[43] D. Faller,et al. Short-chain fatty acid inhibitors of histone deacetylases: promising anticancer therapeutics? , 2003, Current cancer drug targets.
[44] Julia Tischler,et al. The Tumor Suppressor p53 and Histone Deacetylase 1 Are Antagonistic Regulators of the Cyclin-Dependent Kinase Inhibitor p21/WAF1/CIP1 Gene , 2003, Molecular and Cellular Biology.
[45] A. V. van Kuilenburg,et al. Histone deacetylases (HDACs): characterization of the classical HDAC family. , 2003, The Biochemical journal.
[46] Peter A. Jones,et al. Epigenetics in Carcinogenesis and Cancer Prevention , 2003, Annals of the New York Academy of Sciences.
[47] P. Marks,et al. Histone deacetylase inhibitors: from target to clinical trials , 2002, Expert opinion on investigational drugs.
[48] D. O’Carroll,et al. Essential function of histone deacetylase 1 in proliferation control and CDK inhibitor repression , 2002, The EMBO journal.
[49] B. Hinnebusch,et al. The effects of short-chain fatty acids on human colon cancer cell phenotype are associated with histone hyperacetylation. , 2002, The Journal of nutrition.
[50] Kan Yang,et al. Colorectal Cancer in Mice Genetically Deficient in the Mucin Muc2 , 2002, Science.
[51] H. Zoghbi,et al. Requirement of Math1 for Secretory Cell Lineage Commitment in the Mouse Intestine , 2001, Science.
[52] H. Kwon,et al. Expression Profile of Histone Deacetylase 1 in Gastric Cancer Tissues , 2001, Japanese journal of cancer research : Gann.
[53] S. Berger,et al. Acetylation of p53 activates transcription through recruitment of coactivators/histone acetyltransferases. , 2001, Molecular cell.
[54] N. Rosen,et al. The histone deacetylase inhibitor suberoylanilide hydroxamic acid induces differentiation of human breast cancer cells. , 2001, Cancer research.
[55] R. Dahiya,et al. Histone deacetylase and DNA methyltransferase in human prostate cancer. , 2001, Biochemical and biophysical research communications.
[56] B. Hinnebusch,et al. Transient vs. prolonged histone hyperacetylation: effects on colon cancer cell growth, differentiation, and apoptosis. , 2001, American journal of physiology. Gastrointestinal and liver physiology.
[57] W. D. Cress,et al. Histone deacetylases, transcriptional control, and cancer , 2000, Journal of cellular physiology.
[58] L. Yin,et al. The luminal short-chain fatty acid butyrate modulates NF-kappaB activity in a human colonic epithelial cell line. , 2000, Gastroenterology.
[59] D. Podolsky. Mechanisms of regulatory peptide action in the gastrointestinal tract: trefoil peptides. , 2000, Journal of gastroenterology.
[60] T. Fujioka. Clarithromycin resistance in Helicobacter pylori: implications for therapy , 2000, Journal of Gastroenterology.
[61] S. Archer,et al. Histone acetylation and cancer. , 1999, Current opinion in genetics & development.
[62] R. Bresalier,et al. Liver colonization by human colon cancer cells is reduced by antisense inhibition of MUC2 mucin synthesis. , 1999, Gastroenterology.
[63] R. Whitehead,et al. Short-chain fatty acids inhibit intestinal trefoil factor gene expression in colon cancer cells. , 1998, American journal of physiology. Gastrointestinal and liver physiology.
[64] A. Eastman,et al. Apoptotic death in adenocarcinoma cell lines induced by butyrate and other histone deacetylase inhibitors. , 1997, Biochemical pharmacology.
[65] L. Augenlicht,et al. Patterns of expression of lineage-specific markers during the in vitro-induced differentiation of HT29 colon carcinoma cells. , 1995, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[66] R. Dahiya,et al. Mucin production by human colonic carcinoma cells correlates with their metastatic potential in animal models of colon cancer metastasis. , 1991, The Journal of clinical investigation.
[67] I. M. Neiman,et al. [Inflammation and cancer]. , 1974, Patologicheskaia fiziologiia i eksperimental'naia terapiia.