Ischemia/reperfusion injury in human kidney transplantation: an immunohistochemical analysis of changes after reperfusion.
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[1] M. Al-Qattan. Ischaemia-Reperfusion Injury , 1998 .
[2] T. Aw,et al. Molecular mechanisms of anoxia/reoxygenation-induced neutrophil adherence to cultured endothelial cells. , 1997, Circulation research.
[3] A. Marchevsky,et al. Accumulation of platelets in rat syngeneic lung transplants: a potential factor responsible for preservation-reperfusion injury. , 1997, Transplantation.
[4] K. Nadeau,et al. The role of the B7 costimulatory pathway in experimental cold ischemia/reperfusion injury. , 1997, The Journal of clinical investigation.
[5] K. Salmela,et al. Long-term graft outcome is not necessarily affected by delayed onset of graft function and early acute rejection. , 1997, Transplantation.
[6] K. Nadeau,et al. The cytokine-adhesion molecule cascade in ischemia/reperfusion injury of the rat kidney. Inhibition by a soluble P-selectin ligand. , 1997, The Journal of clinical investigation.
[7] R. Wolfe,et al. Delayed graft function: risk factors and implications for renal allograft survival. , 1997, Transplantation.
[8] J J Zwaginga,et al. Platelet and fibrin deposition at the damaged vessel wall: cooperative substrates for neutrophil adhesion under flow conditions. , 1997, Blood.
[9] D. Durand,et al. A randomized multicenter trial comparing leukocyte function-associated antigen-1 monoclonal antibody with rabbit antithymocyte globulin as induction treatment in first kidney transplantations. , 1996, Transplantation.
[10] B. Brenner,et al. Antigen-independent determinants of cadaveric kidney transplant failure. , 1996, JAMA.
[11] F. Belloc,et al. Influence of hypoxia and hypoxia-reoxygenation on endothelial P-selectin expression. , 1996, Haemostasis.
[12] M. Nicholson,et al. The relative influence of delayed graft function and acute rejection on renal transplant survival , 1996, Transplant international : official journal of the European Society for Organ Transplantation.
[13] T. Springer,et al. Neutrophil rolling, arrest, and transmigration across activated, surface-adherent platelets via sequential action of P-selectin and the beta 2-integrin CD11b/CD18. , 1996, Blood.
[14] G. Nash,et al. Continuous activation and deactivation of integrin CD11b/CD18 during de novo expression enables rolling neutrophils to immobilize on platelets. , 1996, Blood.
[15] P. Halloran,et al. Delayed graft function in renal transplantation: etiology, management and long-term significance. , 1996, The Journal of urology.
[16] D. Dunn,et al. Delayed graft function in the absence of rejection has no long-term impact. A study of cadaver kidney recipients with good graft function at 1 year after transplantation. , 1996, Transplantation.
[17] K. Messmer,et al. The Impact of Ischemia/Reperfusion Injury on Specific and Non-Specific, Early and Late Chronic Events After Organ Transplantation , 1996 .
[18] C. Vesin,et al. Activated human platelets express β2 integrin , 1996 .
[19] T. Mayadas,et al. Hypoxia-induced exocytosis of endothelial cell Weibel-Palade bodies. A mechanism for rapid neutrophil recruitment after cardiac preservation. , 1996, The Journal of clinical investigation.
[20] E. Tremoli,et al. Platelet-neutrophil interaction and superoxide anion generation: involvement of purine nucleotides. , 1996, Free radical biology & medicine.
[21] G. Nash,et al. Adhesion of flowing neutrophils to cultured endothelial cells after hypoxia and reoxygenation in vitro. , 1995, The American journal of physiology.
[22] A. Matas,et al. Delayed graft function, acute rejection, and outcome after cadaver renal transplantation. The multivariate analysis. , 1995, Transplantation.
[23] P. Halloran,et al. Ischemic acute tubular necrosis induces an extensive local cytokine response. Evidence for induction of interferon-gamma, transforming growth factor-beta 1, granulocyte-macrophage colony-stimulating factor, interleukin-2, and interleukin-10. , 1995, Transplantation.
[24] S. Tullius,et al. Both alloantigen-dependent and -independent factors influence chronic allograft rejection. , 1995, Transplantation.
[25] T. Peters,et al. Cold Ischemia And Outcome In 17,937 Cadaveric Kidney Transplants , 1995, Transplantation.
[26] A. Davenport,et al. Measurement of malondialdehyde as a marker of oxygen free radical production during renal allograft transplantation and the effect on early graft function. , 1995, Clinical transplantation.
[27] J. Remacle,et al. Hypoxic human umbilical vein endothelial cells induce activation of adherent polymorphonuclear leukocytes. , 1994, Blood.
[28] I. Feuerstein,et al. Role of P-selectin and leukocyte activation in polymorphonuclear cell adhesion to surface adherent activated platelets under physiologic shear conditions (an injury vessel wall model) , 1994 .
[29] J. Hakim,et al. Reactive oxygen species rapidly increase endothelial ICAM-1 ability to bind neutrophils without detectable upregulation. , 1994, Blood.
[30] P. Grace,et al. Ischemia-reperfusion injury , 1994 .
[31] Takaaki Kobayashi,et al. Effect of prolonged delayed graft function on long-term graft outcome in cadaveric kidney transplantation. , 1994, Clinical transplantation.
[32] A. Matas,et al. THE IMPACT OF THE QUALITY OF INITIAL GRAFT FUNCTION ON CADAVER KIDNEY TRANSPLANTS1,2 , 1994, Transplantation.
[33] K. Messmer,et al. The beneficial effect of human recombinant superoxide dismutase on acute and chronic rejection events in recipients of cadaveric renal transplants. , 1994, Transplantation.
[34] I. Feuerstein,et al. Role of P-selectin and leukocyte activation in polymorphonuclear cell adhesion to surface adherent activated platelets under physiologic shear conditions (an injury vessel wall model). , 1994, Blood.
[35] L. McIntire,et al. E-selectin supports neutrophil rolling in vitro under conditions of flow. , 1993, The Journal of clinical investigation.
[36] T. Springer,et al. Neutrophils roll on E-selectin. , 1993, Journal of immunology.
[37] T. Irimura,et al. Activated platelets induce superoxide anion release by monocytes and neutrophils through P-selectin (CD62). , 1993, Journal of immunology.
[38] J. Sanabria,et al. Role of platelets in hepatic allograft preservation injury in the rat , 1993, Hepatology.
[39] M. Lamy,et al. Evidence for free radical formation during human kidney transplantation. , 1993, Free radical biology & medicine.
[40] G. Nash,et al. Selectin-mediated rolling of neutrophils on immobilized platelets. , 1993, Blood.
[41] Y. Nishizawa,et al. Clinicopathology of kidneys from brain-dead patients treated with vasopressin and epinephrine. , 1993, Kidney international.
[42] R. Korthuis,et al. Reactive oxygen metabolites, neutrophils, and the pathogenesis of ischemic‐tissue/reperfusion , 1993, Clinical cardiology.
[43] R. Colvin,et al. A phase I trial of immunosuppression with anti-ICAM-1 (CD54) mAb in renal allograft recipients. , 1993, Transplantation.
[44] S. Fuggle,et al. VARIATION IN EXPRESSION OF ENDOTHELIAL ADHESION MOLECULES IN PRETRANSPLANT AND TRANSPLANTED KIDNEYS‐CORRELATION WITH INTRAGRAFT EVENTS , 1993, Transplantation.
[45] D. Granger,et al. Leukocyte--endothelial cell adhesion induced by ischemia and reperfusion. , 1993, Canadian journal of physiology and pharmacology.
[46] A. Malik,et al. Thrombin-induced expression of endothelial P-selectin and intercellular adhesion molecule-1: a mechanism for stabilizing neutrophil adhesion , 1992, The Journal of cell biology.
[47] C. Benjamin,et al. Leukocyte accumulation promoting fibrin deposition is mediated in vivo by P-selectin on adherent platelets , 1992, Nature.
[48] H. Esterbauer,et al. Human plasma lipid peroxide levels show a strong transient increase after successful revascularization operations. , 1992, Free radical biology & medicine.
[49] A. Gaber,et al. Prediction by postrevascularization biopsies of cadaveric kidney allografts of rejection, graft loss, and preservation nephropathy. , 1992, Transplantation.
[50] G. Danovitch,et al. The high cost of delayed graft function in cadaveric renal transplantation. , 1991, Transplantation.
[51] G. Zimmerman,et al. Oxygen radicals induce human endothelial cells to express GMP-140 and bind neutrophils , 1991, The Journal of cell biology.
[52] Rodger P. McEver,et al. Rapid neutrophil adhesion to activated endothelium mediated by GMP-140 , 1990, Nature.
[53] P. Halloran,et al. Increased major histocompatibility complex antigen expression in unilateral ischemic acute tubular necrosis in the mouse. , 1990, Transplantation.
[54] P. Sims,et al. Stimulated secretion of endothelial von Willebrand factor is accompanied by rapid redistribution to the cell surface of the intracellular granule membrane protein GMP-140. , 1989, The Journal of biological chemistry.
[55] J. McGee,et al. Monoclonal antibody EBM/11: high cellular specificity for human macrophages. , 1988, Journal of clinical pathology.
[56] W. Bennett,et al. SIGNIFICANCE OF DELAYED GRAFT FUNCTION IN CYCLOSPORINE‐TREATED RECIPIENTS OF CADAVER KIDNEY TRANSPLANTS , 1988, Transplantation.
[57] R. Allen,et al. Triple therapy in cadaver renal transplantation , 1988, The British journal of surgery.
[58] S. Fuggle,et al. SEQUENTIAL ANALYSIS OF HLA‐CLASS II ANTIGEN EXPRESSION IN HUMAN RENAL ALLOGRAFTS Induction of Tubular class II Antigens and Correlation with Clinical Parameters , 1986, Transplantation.
[59] V. Nachmias,et al. Platelet activation. , 2020, Arteriosclerosis.
[60] N. Hogg,et al. Monoclonal antibodies specific for human monocytes, granulocytes and endothelium. , 1984, Immunology.
[61] W K Vaughn,et al. THE DETRIMENTAL EFFECTS OF DELAYED GRAFT FUNCTION IN CADAVER DONOR RENAL TRANSPLANTATION , 1984, Transplantation.
[62] R. McEver,et al. A monoclonal antibody to a membrane glycoprotein binds only to activated platelets. , 1984, The Journal of biological chemistry.
[63] R. Andrews,et al. Normal and malignant human myelocytic and monocytic cells identified by monoclonal antibodies. , 1982, Journal of immunology.
[64] J. McKenzie,et al. STUDIES WITH A MONOCLONAL ANTIBODY ON THE DISTRIBUTION OF THY‐1 IN THE LYMPHOID AND EXTRACELLULAR CONNECTIVE TISSUES OF THE DOG , 1981, Transplantation.
[65] R. Callard,et al. Distinctive functional characteristics of human „T”︁ lymphocytes defined by E rosetting or a monoclonal anti‐T cell antibody , 1981, European journal of immunology.
[66] J. Selkon,et al. β-LACTAMASE-PRODUCING ANÆROBES , 1980, The Lancet.
[67] J. Fabre,et al. Monoclonal antibody to a human leukocyte‐specific membrane glycoprotein probably homologous to the leukocyte‐common (L‐C) antigen of the rat , 1980, European journal of immunology.
[68] P. Parham,et al. A monoclonal antibody that recognizes an antigenic determinant shared by HLA A2 and B17. , 1980, Human immunology.
[69] D. G. Osborne,et al. Histologic, ultrastructural, and immunomicroscopic findings in 96 one hour human renal allograft biopsy specimens. Immunologic and clinical significance. , 1980, Human pathology.
[70] C. Barnstable,et al. Monoclonal Antibodies for Analysis of the HLA System , 1979, Immunological reviews.
[71] D. L. Westbroek,et al. Prognostic value for immediate function of one-hour renal allograft biopsy. , 1975, British medical journal.
[72] L. Perloff,et al. Value of one-hour renal-allograft biopsy. , 1973, Lancet.
[73] P. Kincaid‐smith,et al. Immediate renal-graft biopsy and subsequent rejection. , 1968, Lancet.
[74] P. Morris,et al. "Hyperacute" renal-homograft rejection in man. , 1968, The New England journal of medicine.
[75] N. L. Johnson,et al. Multivariate Analysis , 1958, Nature.