A Subset of TREM2+ Dermal Macrophages Secretes Oncostatin M to Maintain Hair Follicle Stem Cell Quiescence and Inhibit Hair Growth.
暂无分享,去创建一个
C. Drake | A. Christiano | A. Ferrante | Z. Dai | E. Wang
[1] F. Azuaje,et al. Single‐cell transcriptomics reveals distinct inflammation‐induced microglia signatures , 2018, EMBO reports.
[2] D. Artis,et al. Beyond Host Defense: Emerging Functions of the Immune System in Regulating Complex Tissue Physiology , 2018, Cell.
[3] H. Blau,et al. Induction of muscle stem cell quiescence by the secreted niche factor Oncostatin M , 2018, Nature Communications.
[4] Qingwen Cheng,et al. TREM2-activating antibodies abrogate the negative pleiotropic effects of the Alzheimer's disease variant Trem2R47H on murine myeloid cell function , 2018, The Journal of Biological Chemistry.
[5] Dennis Wolf,et al. Single-Cell RNA-Seq Reveals the Transcriptional Landscape and Heterogeneity of Aortic Macrophages in Murine Atherosclerosis , 2018, Circulation research.
[6] S. Rose-John. Interleukin-6 Family Cytokines. , 2018, Cold Spring Harbor perspectives in biology.
[7] Flores Aimee,et al. Lactate dehydrogenase activity drives hair follicle stem cell activation , 2017, Nature cell biology.
[8] F. Nestle,et al. Regulatory T Cells in Skin Facilitate Epithelial Stem Cell Differentiation , 2017, Cell.
[9] Andrej J. Savol,et al. Macrophages Facilitate Electrical Conduction in the Heart , 2017, Cell.
[10] D. Holtzman,et al. Elucidating the Role of TREM2 in Alzheimer’s Disease , 2017, Neuron.
[11] R. Yi,et al. Signaling Networks among Stem Cell Precursors, Transit-Amplifying Progenitors, and their Niche in Developing Hair Follicles. , 2016, Cell reports.
[12] Y. Niitsu,et al. Vitamin A and insulin are required for the maintenance of hepatic stellate cell quiescence. , 2016, Experimental cell research.
[13] A. Christiano,et al. Pharmacologic inhibition of JAK-STAT signaling promotes hair growth , 2015, Science Advances.
[14] Avi Ma’ayan,et al. An Integrated Transcriptome Atlas of Embryonic Hair Follicle Progenitors, Their Niche, and the Developing Skin. , 2015, Developmental cell.
[15] M. Colonna,et al. TREM2 sustains microglial expansion during aging and response to demyelination. , 2015, The Journal of clinical investigation.
[16] R. Paus,et al. Macrophages Contribute to the Cyclic Activation of Adult Hair Follicle Stem Cells , 2014, PLoS biology.
[17] Kaibo Duan,et al. Human Dermal CD14+ Cells Are a Transient Population of Monocyte-Derived Macrophages , 2014, Immunity.
[18] V. Horsley,et al. Calcineurin/Nfatc1 signaling links skin stem cell quiescence to hormonal signaling during pregnancy and lactation , 2014, Genes & development.
[19] N. Bausek. JAK-STAT signaling in stem cells and their niches in Drosophila , 2013, JAK-STAT.
[20] L. Sly,et al. Generation and characterization of murine alternatively activated macrophages. , 2013, Methods in molecular biology.
[21] L. Cozzuto,et al. Age-associated inflammation inhibits epidermal stem cell function. , 2012, Genes & development.
[22] C. Watson,et al. The spectrum of STAT functions in mammary gland development , 2012, JAK-STAT.
[23] M. Kim,et al. Dihydrotestosterone-inducible IL-6 inhibits elongation of human hair shafts by suppressing matrix cell proliferation and promotes regression of hair follicles in mice. , 2012, The Journal of investigative dermatology.
[24] E. Fuchs,et al. Dynamics between Stem Cells, Niche, and Progeny in the Hair Follicle , 2011, Cell.
[25] D. Hume,et al. An antibody against the colony-stimulating factor 1 receptor depletes the resident subset of monocytes and tissue- and tumor-associated macrophages but does not inhibit inflammation. , 2010, Blood.
[26] F. Ginhoux,et al. Fate Mapping Analysis Reveals That Adult Microglia Derive from Primitive Macrophages , 2010, Science.
[27] H. Pasolli,et al. A two-step mechanism for stem cell activation during hair regeneration. , 2009, Cell stem cell.
[28] P. Heinrich,et al. Oncostatin M-Induced and Constitutive Activation of the JAK2/STAT5/CIS Pathway Suppresses CCL1, but Not CCL7 and CCL8, Chemokine Expression1 , 2008, The Journal of Immunology.
[29] A. Uezumi,et al. Suppression of macrophage functions impairs skeletal muscle regeneration with severe fibrosis. , 2008, Experimental cell research.
[30] C. Evers,et al. Box 2 Region of the Oncostatin M Receptor Determines Specificity for Recruitment of Janus Kinases and STAT5 Activation* , 2008, Journal of Biological Chemistry.
[31] Ruth E. Baker,et al. Cyclic dermal BMP signalling regulates stem cell activation during hair regeneration , 2008, Nature.
[32] J. Shapiro,et al. Interleukin‐6 cytokine family member oncostatin M is a hair‐follicle‐expressed factor with hair growth inhibitory properties , 2007, Experimental dermatology.
[33] H. Kettenmann,et al. Microglia: active sensor and versatile effector cells in the normal and pathologic brain , 2007, Nature Neuroscience.
[34] H. Neumann,et al. TREM2-Transduced Myeloid Precursors Mediate Nervous Tissue Debris Clearance and Facilitate Recovery in an Animal Model of Multiple Sclerosis , 2007, PLoS medicine.
[35] A. Miyajima,et al. Oncostatin M Inhibits Adipogenesis through the RAS/ERK and STAT5 Signaling Pathways* , 2006, Journal of Biological Chemistry.
[36] F. Helmchen,et al. Resting Microglial Cells Are Highly Dynamic Surveillants of Brain Parenchyma in Vivo , 2005, Science.
[37] G. Müller-Newen. The Cytokine Receptor gp130: Faithfully Promiscuous , 2003, Science's STKE.
[38] R. Paus,et al. Prolactin and its receptor are expressed in murine hair follicle epithelium, show hair cycle-dependent expression, and induce catagen. , 2003, The American journal of pathology.
[39] Michael C. Ostrowski,et al. A macrophage colony-stimulating factor receptor-green fluorescent protein transgene is expressed throughout the mononuclear phagocyte system of the mouse. , 2003, Blood.
[40] D. Levy,et al. Deletion of Stat3 blocks mammary gland involution and extends functional competence of the secretory epithelium in the absence of lactogenic stimuli. , 2002, Endocrinology.
[41] R Paus,et al. A comprehensive guide for the accurate classification of murine hair follicles in distinct hair cycle stages. , 2001, The Journal of investigative dermatology.
[42] R Paus,et al. Controls of hair follicle cycling. , 2001, Physiological reviews.
[43] K. Yoshikawa,et al. Two distinct signaling pathways in hair cycle induction: Stat3-dependent and -independent pathways. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[44] H. Baumann,et al. Oncostatin M and Interleukin 6 inhibit cell cycle progression by prevention of p27kip1 degradation in HepG2 cells , 2000, Oncogene.
[45] T. Imamura,et al. Dual-mode regulation of hair growth cycle by two Fgf-5 gene products. , 2000, The Journal of investigative dermatology.
[46] M. Gómez-Lechón,et al. Oncostatin M: signal transduction and biological activity. , 1999, Life sciences.
[47] T. Imamura,et al. Localization of rat FGF-5 protein in skin macrophage-like cells and FGF-5S protein in hair follicle: possible involvement of two Fgf-5 gene products in hair growth cycle regulation. , 1998, The Journal of investigative dermatology.
[48] S. Eichmüller,et al. Generation and cyclic remodeling of the hair follicle immune system in mice. , 1998, The Journal of investigative dermatology.
[49] S. Eichmüller,et al. Clusters of Perifollicular Macrophages in Normal Murine Skin: Physiological Degeneration of Selected Hair Follicles by Programmed Organ Deletion , 1998, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[50] N. Platt,et al. Macrophage membrane molecules: markers of tissue differentiation and heterogeneity. , 1996, Immunobiology.
[51] T. Rose,et al. Oncostatin M is a member of a cytokine family that includes leukemia-inhibitory factor, granulocyte colony-stimulating factor, and interleukin 6. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[52] R. Paus,et al. Telogen skin contains an inhibitor of hair growth , 1990, The British journal of dermatology.
[53] H. Marquardt,et al. Molecular cloning, sequence analysis, and functional expression of a novel growth regulator, oncostatin M , 1989, Molecular and cellular biology.
[54] G. Todaro,et al. Oncostatin M: a growth regulator produced by differentiated histiocytic lymphoma cells. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[55] O. H. Iversen. The chalones. , 1973, Acta pathologica et microbiologica Scandinavica. Supplement.