Enhancement of the antiallodynic and antinociceptive efficacy of spinal morphine by antisera to dynorphin A (1–13) or MK-801 in a nerve-ligation model of peripheral neuropathy
暂无分享,去创建一个
F. Porreca | M. Ossipov | D. Bian | M. Nichols | Y. Lopez | M. L. Nichols
[1] G. Wilcox,et al. Single intrathecal injections of dynorphin A or des-Tyr-dynorphins produce long-lasting allodynia in rats: blockade by MK-801 but not naloxone , 1996, Pain.
[2] F. Porreca,et al. Regulation of morphine antiallodynic efficacy by cholecystokinin in a model of neuropathic pain in rats. , 1995, The Journal of pharmacology and experimental therapeutics.
[3] F. Porreca,et al. Inhibition by spinal morphine of the tail-flick response is attenuated in rats with nerve ligation injury , 1995, Neuroscience Letters.
[4] F. Porreca,et al. The loss of antinociceptive efficacy of spinal morphine in rats with nerve ligation injury is prevented by reducing spinal afferent drive , 1995, Neuroscience Letters.
[5] S. Harrison,et al. Peri-administration of clonidine or MK801 delays but does not prevent the development of mechanical hyperalgesia in a model of mononeuropathy in the rat , 1995, Neuroscience Letters.
[6] C. Woolf,et al. Noxious stimuli induce an N-methyl-d-aspartate receptor-dependent hypersensitivity of the flexion withdrawal reflex to touch: implications for the treatment of mechanical allodynia , 1995, Pain.
[7] L. Chen,et al. The mechanism of action for the block of NMDA receptor channels by the opioid peptide dynorphin , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[8] T. Yaksh,et al. Analysis of Drug Interaction between Intrathecal Clonidine and MK‐801 in Peripheral Neuropathic Pain Rat Model , 1995, Anesthesiology.
[9] S. Yom,et al. Norepinephrine Rekindles Mechanical Allodynia in Sympathectomized Neuropathic Rat , 1995 .
[10] T. Yaksh,et al. Reversal of nerve ligation-induced allodynia by spinal alpha-2 adrenoceptor agonists. , 1995, The Journal of pharmacology and experimental therapeutics.
[11] V. Shukla,et al. Non-opioid effects of dynorphins: possible role of the NMDA receptor. , 1994, Trends in pharmacological sciences.
[12] A. Yakovlev,et al. Sequential expression of c-fos protooncogene, TNF-alpha, and dynorphin genes in spinal cord following experimental traumatic injury. , 1994, Molecular and chemical neuropathology.
[13] T. Doubell,et al. The pathophysiology of chronic pain — increased sensitivity to low threshold Aβ-fibre inputs , 1994, Current Opinion in Neurobiology.
[14] T. Yaksh,et al. Quantitative assessment of tactile allodynia in the rat paw , 1994, Journal of Neuroscience Methods.
[15] G. Bennett,et al. Neuropathic pain sensations are differentially sensitive to dextrorphan , 1994, Neuroreport.
[16] Xiao-jun Xu,et al. Cholecystokinin and morphine analgesia: variations on a theme. , 1994, Trends in pharmacological sciences.
[17] Jin Mo Chung,et al. An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat , 1992, PAIN.
[18] R. Coggeshall,et al. Peripheral nerve injury triggers central sprouting of myelinated afferents , 1992, Nature.
[19] R. Dubner,et al. Up-regulation of opioid gene expression in spinal cord evoked by experimental nerve injuries and inflammation , 1991, Brain Research.
[20] Z. Seltzer,et al. Modulation of neuropathic pain behavior in rats by spinal disinhibition and NMDA receptor blockade of injury discharge , 1991, Pain.
[21] Clifford J. Woolf,et al. The induction and maintenance of central sensitization is dependent on N-methyl-d-aspartic acid receptor activation; implications for the treatment of post-injury pain hypersensitivity states , 1991, Pain.
[22] M. Iadarola,et al. Dynorphin increases in the dorsal spinal cord in rats with a painful peripheral neuropathy , 1990, Peptides.
[23] A. Faden. Opioid and nonopioid mechanisms may contribute to dynorphin's pathophysiological actions in spinal cord injury , 1990, Annals of neurology.
[24] D. Massardier,et al. A direct non-opiate interaction of dynorphin-(1-13) with the N-methyl-D-aspartate (NMDA) receptor. , 1989, European journal of pharmacology.
[25] J. Holaday,et al. Neurological dysfunction after intrathecal injection of dynorphin A (1-13) in the rat. II. Nonopioid mechanisms mediate loss of motor, sensory and autonomic function. , 1988, The Journal of pharmacology and experimental therapeutics.
[26] Gary J. Bennett,et al. A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man , 1988, Pain.
[27] B. Meyerson,et al. Lack of analgesic effect of opioids on neuropathic and idiopathic forms of pain , 1988, Pain.
[28] R. Caudle,et al. A novel interaction between dynorphin(1–13) and an N-methyl-d-aspartate site , 1988, Brain Research.
[29] D. Lodge,et al. Evidence for involvement ofN-methylaspartate receptors in ‘wind-up’ of class 2 neurones in the dorsal horn of the rat , 1987, Brain Research.
[30] G. Wilcox,et al. Nociceptive action of excitatory amino acids in the mouse: effects of spinally administered opioids, phencyclidine and sigma agonists. , 1987, The Journal of pharmacology and experimental therapeutics.
[31] G. Wilcox,et al. Phencyclidine selectively blocks a spinal action of N-methyl-d-aspartate in mice , 1986, Neuroscience Letters.
[32] R. Payne. Neuropathic Pain Syndromes, with Special Reference to Causalgia and Reflex Sympathetic Dystrophy , 1986 .
[33] A. Faden,et al. Effects of traumatic injury on dynorphin immunoreactivity in spinal cord , 1985, Neuropeptides.
[34] H. Akil,et al. Nonopiate effects of dynorphin and des-Tyr-dynorphin. , 1982, Science.
[35] R. Twycross,et al. Morphine and Diamorphine in the Terminally Ill Patient , 1982, Acta anaesthesiologica Scandinavica. Supplementum.
[36] T. Yaksh,et al. Chronic catheterization of the spinal subarachnoid space , 1976, Physiology & Behavior.