Decreases in Ca2+-Dependent K+-Currents Due to Cyclic Guanosine Monophosphate Are Not Dependent on Phosphorylation
暂无分享,去创建一个
[1] M. Kohlhardt,et al. Na+ channel blockade by cyclic AMP and other 6-aminopurines in neonatal rat heart , 2005, The Journal of Membrane Biology.
[2] B. Horowitz,et al. Cyclic GMP-dependent Protein Kinase Activates Cloned BKCa Channels Expressed in Mammalian Cells by Direct Phosphorylation at Serine 1072* , 1999, The Journal of Biological Chemistry.
[3] D DiFrancesco,et al. Dual allosteric modulation of pacemaker (f) channels by cAMP and voltage in rabbit SA node , 1999, The Journal of physiology.
[4] F. Hofmann,et al. The Large Conductance, Voltage-dependent, and Calcium-sensitive K+ Channel, Hslo, Is a Target of cGMP-dependent Protein Kinase Phosphorylation in Vivo * , 1998, The Journal of Biological Chemistry.
[5] A. De Luca,et al. Blockade by cAMP of native sodium channels of adult rat skeletal muscle fibers. , 1998, American journal of physiology. Cell physiology.
[6] N. Teramoto,et al. Cyclic GMP‐dependent but G‐kinase‐independent inhibition of Ca2+‐dependent Cl− currents by NO donors in cat tracheal smooth muscle , 1998, The Journal of physiology.
[7] J. Stockand,et al. Regulation of Large Calcium-activated Potassium Channels by Protein Phosphatase 2A* , 1997, The Journal of Biological Chemistry.
[8] Y. Kurachi,et al. Phosphorylation‐independent inhibition by intracellular cyclic nucleotides of brain inwardly rectifying K+ current expressed in Xenopus oocytes , 1997, FEBS letters.
[9] O. Borisova,et al. Cyclic AMP Does Not Induce the Down-Regulation of Calcium Current in Molluscan Neurons Through Kinase A Activation or Cytoplasmic Ca2+ Elevation , 1997 .
[10] F. Hofmann,et al. Protein Phosphatase 2A Is Essential for the Activation of Ca2+-activated K+ Currents by cGMP-dependent Protein Kinase in Tracheal Smooth Muscle and Chinese Hamster Ovary Cells* , 1996, The Journal of Biological Chemistry.
[11] G. Breitwieser. Mechanisms of K+ Channel Regulation , 1996, The Journal of Membrane Biology.
[12] M. Mattson,et al. Activation of K+ channels and suppression of neuronal activity by secreted β-amyloid-precursor protein , 1996, Nature.
[13] F. Murad,et al. Nitric Oxide and Cyclic GMP Signaling , 1996, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[14] H. Sontheimer,et al. β‐Adrenergic Modulation of Glial Inwardly Rectifying Potassium Channels , 1995, Journal of neurochemistry.
[15] Q.-Y. Liu,et al. cGMP inhibits L-type Ca2+ channel currents through protein phosphorylation in rat pinealocytes , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[16] J. Warmke,et al. A family of potassium channel genes related to eag in Drosophila and mammals. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[17] L. Pardo,et al. Ether-à-go-go encodes a voltage-gated channel permeable to K+ and Ca2+ and modulated by cAMP , 1993, Nature.
[18] Richard E. White,et al. Potassium channel stimulation by natriuretic peptides through cGMP-dependent dephosphorylation , 1993, Nature.
[19] Y. Zhong,et al. Alteration of four identified K+ currents in Drosophila muscle by mutations in eag , 1991, Science.
[20] Pharmacology of potassium channels and presynaptic regulation of excitatory amino acid release Siciliano J, Dajas F, and Barbeito L. Div. Neuroquimica, Instituto Clemente Estable, Montevideo, CP 11600, URUGUAY , 1990, Neurochemistry International.
[21] Kazuko Watanabe,et al. Two subtypes of C current in identifiedHelix neurons , 1990, Brain Research.
[22] J. Corbin,et al. Dual ion-channel regulation by cyclic GMP and cyclic GMP-dependent protein kinase , 1990, Nature.
[23] S. Kawamoto,et al. Isoquinolinesulfonamides, novel and potent inhibitors of cyclic nucleotide dependent protein kinase and protein kinase C. , 1984, Biochemistry.