p38α and p38δ mitogen-activated protein kinase isoforms regulate invasion and growth of head and neck squamous carcinoma cells
暂无分享,去创建一个
J. Westermarck | P. Jaakkola | R. Grenman | R. Ala-aho | V. Kähäri | J. Peltonen | M. Junttila | T. Jokilehto | M. Kallajoki | R. Grénman | R. Grénman | R. Ala‐aho
[1] K. Ang,et al. Head and Neck Cancers , 2011 .
[2] D. Brachman,et al. Occurrence of p 53 Gene Deletions and Human Papilloma Virus Infection in Human Head and Neck Cancer 1 , 2006 .
[3] G. Casey,et al. Frequent p 53 Mutations in Head and Neck Cancer ' , 2006 .
[4] P. Harrison,et al. Profiling early head and neck cancer , 2005, Nature Reviews Cancer.
[5] R. Ala-aho,et al. Targeted inhibition of human collagenase-3 (MMP-13) expression inhibits squamous cell carcinoma growth in vivo , 2004, Oncogene.
[6] P. Hofmann,et al. Protein phosphatase 2A-mediated cross-talk between p38 MAPK and ERK in apoptosis of cardiac myocytes. , 2004, American journal of physiology. Heart and circulatory physiology.
[7] J. Olson,et al. p38 MAP kinase: a convergence point in cancer therapy. , 2004, Trends in molecular medicine.
[8] K. Reddy,et al. Role of MAP kinase in tumor progression and invasion , 2003, Cancer and Metastasis Reviews.
[9] Jiahuai Han,et al. p38 Mitogen-activated protein kinase pathway suppresses cell survival by inducing dephosphorylation of mitogen-activated protein/extracellular signal-regulated kinase kinase1,2. , 2020, Cancer research.
[10] R. Eckert,et al. A Regulatory Role for p38δ MAPK in Keratinocyte Differentiation , 2003, Journal of Biological Chemistry.
[11] Jiahuai Han,et al. p38 Isoforms Have Opposite Effects on AP-1-dependent Transcription through Regulation of c-Jun , 2003, The Journal of Biological Chemistry.
[12] Emma Black,et al. Negative Feedback Regulation of MKK6 mRNA Stability by p38α Mitogen-Activated Protein Kinase , 2003, Molecular and Cellular Biology.
[13] Jiahuai Han,et al. p38 Mitogen-activated Protein Kinase Pathway Suppresses Cell Survival by Inducing Dephosphorylation of Mitogen-activated Protein/Extracellular , 2003 .
[14] Emma Black,et al. Negative feedback regulation of MKK6 mRNA stability by p38alpha mitogen-activated protein kinase. , 2003, Molecular and cellular biology.
[15] Xiang Guo,et al. Head and neck cancers. , 2003, Cancer chemotherapy and biological response modifiers.
[16] Jiahuai Han,et al. Activation of p38α MAPK Enhances Collagenase-1 (Matrix Metalloproteinase (MMP)-1) and Stromelysin-1 (MMP-3) Expression by mRNA Stabilization* , 2002, The Journal of Biological Chemistry.
[17] C. Der,et al. Raf-independent Deregulation of p38 and JNK Mitogen-activated Protein Kinases Are Critical for Ras Transformation* , 2002, The Journal of Biological Chemistry.
[18] Albert J. Fornace,et al. Amplification of PPM1D in human tumors abrogates p53 tumor-suppressor activity , 2002, Nature Genetics.
[19] V. Kähäri,et al. Matrix metalloproteinases in cancer: Prognostic markers and therapeutic targets , 2002, International journal of cancer.
[20] Z. Werb,et al. New functions for the matrix metalloproteinases in cancer progression , 2002, Nature Reviews Cancer.
[21] R. Grenman,et al. Adenoviral delivery of p53 gene suppresses expression of collagenase-3 (MMP-13) in squamous carcinoma cells , 2002, Oncogene.
[22] Jiahuai Han,et al. p38 Mitogen-Activated Protein Kinase-Dependent Activation of Protein Phosphatases 1 and 2A Inhibits MEK1 and MEK2 Activity and Collagenase 1 (MMP-1) Gene Expression , 2001, Molecular and Cellular Biology.
[23] J. Aguirre-Ghiso,et al. Urokinase receptor and fibronectin regulate the ERK(MAPK) to p38(MAPK) activity ratios that determine carcinoma cell proliferation or dormancy in vivo. , 2001, Molecular biology of the cell.
[24] J. Westermarck,et al. Activation of fibroblast collagenase-1 expression by tumor cells of squamous cell carcinomas is mediated by p38 mitogen-activated protein kinase and c-Jun NH2-terminal kinase-2. , 2000, Cancer research.
[25] G. Nemerow,et al. Urokinase Plasminogen Activator/Urokinase-specific Surface Receptor Expression and Matrix Invasion by Breast Cancer Cells Requires Constitutive p38α Mitogen-activated Protein Kinase Activity* , 2000, The Journal of Biological Chemistry.
[26] H. Enslen,et al. Molecular determinants that mediate selective activation of p38 MAP kinase isoforms , 2000, The EMBO journal.
[27] C. López-Otín,et al. Expression of collagenase-3 (MMP-13) and collagenase-1 (MMP-1) by transformed keratinocytes is dependent on the activity of p38 mitogen-activated protein kinase. , 2000, Journal of cell science.
[28] D. Hanahan,et al. The Hallmarks of Cancer , 2000, Cell.
[29] G. Nemerow,et al. Urokinase plasminogen activator/urokinase-specific surface receptor expression and matrix invasion by breast cancer cells requires constitutive p38alpha mitogen-activated protein kinase activity. , 2000, The Journal of biological chemistry.
[30] Jiahuai Han,et al. The p38 signal transduction pathway: activation and function. , 2000, Cellular signalling.
[31] Jiahuai Han,et al. Transforming Growth Factor-β Induces Collagenase-3 Expression by Human Gingival Fibroblasts via p38 Mitogen-activated Protein Kinase* , 1999, The Journal of Biological Chemistry.
[32] K. Sakaguchi,et al. Phosphorylation of human p53 by p38 kinase coordinates N‐terminal phosphorylation and apoptosis in response to UV radiation , 1999, The EMBO journal.
[33] C. Manthey,et al. Differential expression and activation of p38 mitogen-activated protein kinase alpha, beta, gamma, and delta in inflammatory cell lineages. , 1999, Journal of immunology.
[34] G L Johnson,et al. Organization and regulation of mitogen-activated protein kinase signaling pathways. , 1999, Current opinion in cell biology.
[35] D. Johnson,et al. Purification and activation of recombinant p38 isoforms alpha, beta, gamma, and delta. , 1998, Protein expression and purification.
[36] D. Boyd,et al. Inhibition of the p38 mitogen-activated protein kinase by SB 203580 blocks PMA-induced Mr 92,000 type IV collagenase secretion and in vitro invasion. , 1998, Cancer research.
[37] J Ross,et al. Cardiac Muscle Cell Hypertrophy and Apoptosis Induced by Distinct Members of the p38 Mitogen-activated Protein Kinase Family* , 1998, The Journal of Biological Chemistry.
[38] Roger J. Davis,et al. Selective Activation of p38 Mitogen-activated Protein (MAP) Kinase Isoforms by the MAP Kinase Kinases MKK3 and MKK6* , 1998, The Journal of Biological Chemistry.
[39] R. Grenman,et al. Expression of collagenase-3 (matrix metalloproteinase-13) in squamous cell carcinomas of the head and neck. , 1997, The American journal of pathology.
[40] C. López-Otín,et al. Expression of collagenase-3 in the rat ovary during the ovulatory process. , 1996, The Journal of endocrinology.
[41] Michael E. Greenberg,et al. Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis , 1995, Science.
[42] G. Casey,et al. Frequent p53 mutations in head and neck cancer. , 1992, Cancer research.
[43] D. Brachman,et al. Occurrence of p53 gene deletions and human papilloma virus infection in human head and neck cancer. , 1992, Cancer research.
[44] G. Wilkinson,et al. Constitutive and enhanced expression from the CMV major IE promoter in a defective adenovirus vector. , 1992, Nucleic acids research.
[45] P. Chomczyński,et al. Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction. , 1987, Analytical biochemistry.
[46] S. Boyce,et al. Calcium-regulated differentiation of normal human epidermal keratinocytes in chemically defined clonal culture and serum-free serial culture. , 1983, The Journal of investigative dermatology.
[47] G. Klein,et al. Cellular reactions against Burkitt lymphoma cells. III. Effector cell activity of leukocytes stimulated in vitro with autochthonous cultured lymphoma cells , 1972, International journal of cancer.