Bile Acids as Inducers of Protonophore and Ionophore Permeability of Biological and Artificial Membranes
暂无分享,去创建一个
[1] K. Belosludtsev,et al. The Effect of Uridine on the State of Skeletal Muscles and the Functioning of Mitochondria in Duchenne Dystrophy , 2022, International journal of molecular sciences.
[2] A. Elorza,et al. Bile Acids Induce Alterations in Mitochondrial Function in Skeletal Muscle Fibers , 2022, Antioxidants.
[3] Y. Antonenko,et al. Fifty Years of Research on Protonophores: Mitochondrial Uncoupling As a Basis for Therapeutic Action , 2022, Acta naturae.
[4] Huiping Zhou,et al. Key Signaling in Alcohol-Associated Liver Disease: The Role of Bile Acids , 2022, Cells.
[5] Xinchun Shen,et al. A Review of Bile Acid Metabolism and Signaling in Cognitive Dysfunction-Related Diseases , 2022, Oxidative medicine and cellular longevity.
[6] M. Camilleri. Bile Acid Detergency: Permeability, Inflammation and Effects of Sulfation. , 2022, American journal of physiology. Gastrointestinal and liver physiology.
[7] Tatyana V Butkova,et al. A Recent Ten-Year Perspective: Bile Acid Metabolism and Signaling , 2022, Molecules.
[8] M. Naud,et al. Comparative Analysis of Urso- and Tauroursodeoxycholic Acid Neuroprotective Effects on Retinal Degeneration Models , 2022, Pharmaceuticals.
[9] Wenxia Xu,et al. Research Progress of Bile Acids in Cancer , 2022, Frontiers in Oncology.
[10] P. Pinton,et al. Molecular mechanisms and consequences of mitochondrial permeability transition , 2021, Nature Reviews Molecular Cell Biology.
[11] Alessandra Del Giudice,et al. Condensed Supramolecular Helices: The Twisted Sisters of DNA , 2021, Angewandte Chemie.
[12] G. Lippe,et al. The mitochondrial permeability transition: Recent progress and open questions , 2021, The FEBS journal.
[13] N. Belosludtseva,et al. Uridine treatment prevents myocardial injury in rat models of acute ischemia and ischemia/reperfusion by activating the mitochondrial ATP-dependent potassium channel , 2021, Scientific Reports.
[14] G. Sukhikh,et al. Neuroprotective Potential of Mild Uncoupling in Mitochondria. Pros and Cons , 2021, Brain sciences.
[15] Alessandra Del Giudice,et al. Revealing the complex self-assembly behaviour of sodium deoxycholate in aqueous solution. , 2021, Journal of colloid and interface science.
[16] Edward Johnson,et al. Exploring the therapeutic potential of mitochondrial uncouplers in cancer , 2021, Molecular metabolism.
[17] G. Shulman,et al. Therapeutic potential of mitochondrial uncouplers for the treatment of metabolic associated fatty liver disease and NASH , 2021, Molecular metabolism.
[18] L. Galantini,et al. Physiology and Physical Chemistry of Bile Acids , 2021, International journal of molecular sciences.
[19] G. Mironova,et al. Mitochondrial Cyclosporine A-Independent Palmitate/Ca2+-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity , 2021, Cells.
[20] P. P. Zagoskin,et al. Bile Acids as a New Type of Steroid Hormones Regulating Nonspecific Energy Expenditure of the Body (Review) , 2020, Sovremennye tekhnologii v meditsine.
[21] K. Belosludtsev,et al. Diabetes Mellitus, Mitochondrial Dysfunction and Ca2+-Dependent Permeability Transition Pore , 2020, International journal of molecular sciences.
[22] S. DeMorrow,et al. Bile Acid Signaling in Neurodegenerative and Neurological Disorders , 2020, International journal of molecular sciences.
[23] P. Pinton,et al. Physiopathology of the Permeability Transition Pore: Molecular Mechanisms in Human Pathology , 2020, Biomolecules.
[24] M. Dubinin,et al. A Comparative Study of the Action of Protonophore Uncouplers and Decoupling Agents as Inducers of Free Respiration in Mitochondria in States 3 and 4: Theoretical and Experimental Approaches , 2020, Cell Biochemistry and Biophysics.
[25] E. Murphy,et al. Role of Mitochondrial Calcium and the Permeability Transition Pore in Regulating Cell Death. , 2020, Circulation research.
[26] M. Dubinin,et al. ω-Hydroxypalmitic and α,ω-Hexadecanedioic Acids As Activators of Free Respiration and Inhibitors of H2O2 Generation in Liver Mitochondria , 2020, Biochemistry (Moscow), Supplement Series A: Membrane and Cell Biology.
[27] Amy E. Chadwick,et al. Differential toxic effects of bile acid mixtures in isolated mitochondria and physiologically relevant HepaRG cells , 2019, Toxicology in vitro : an international journal published in association with BIBRA.
[28] C. Steer,et al. Ursodeoxycholic acid: A promising therapeutic target for inflammatory bowel diseases? , 2019, American journal of physiology. Gastrointestinal and liver physiology.
[29] Edward T Chouchani,et al. H+ Transport is an Integral Function of the Mitochondrial ADP/ATP Carrier , 2019, Nature.
[30] K. Belosludtsev,et al. Mitochondrial Ca2+ Transport: Mechanisms, Molecular Structures, and Role in Cells , 2019, Biochemistry (Moscow).
[31] P. Dawson,et al. Animal models to study bile acid metabolism. , 2019, Biochimica et biophysica acta. Molecular basis of disease.
[32] Luís M. S. Loura,et al. Interaction of Bile Salts With Lipid Bilayers: An Atomistic Molecular Dynamics Study , 2019, Front. Physiol..
[33] J. Geisler. 2,4 Dinitrophenol as Medicine , 2019, Cells.
[34] K. Belosludtsev,et al. Induction of the Ca2+-Dependent Permeability Transition in Liver Mitochondria by α,ω-Hexadecanedioic Acid is Blocked by Inorganic Phosphate in the Presence of Cyclosporin A , 2019, Biochemistry (Moscow), Supplement Series A: Membrane and Cell Biology.
[35] Alessandra Del Giudice,et al. Bile Salts: Natural Surfactants and Precursors of a Broad Family of Complex Amphiphiles. , 2018, Langmuir : the ACS journal of surfaces and colloids.
[36] M. Dubinin,et al. Bile Acid-Induced Ca2+ Efflux from Liver Mitochondria as a Factor Preventing the Formation of Mitochondrial Pores , 2018, Biochemistry (Moscow), Supplement Series A: Membrane and Cell Biology.
[37] J. Chiang,et al. Bile Acid Metabolism in Liver Pathobiology. , 2018, Gene expression.
[38] J. Bard,et al. Lithocholic bile acid inhibits lipogenesis and induces apoptosis in breast cancer cells , 2018, Cellular Oncology.
[39] J. Casadesús,et al. Interactions between Bacteria and Bile Salts in the Gastrointestinal and Hepatobiliary Tracts , 2017, Front. Med..
[40] M. Dubinin,et al. Lithocholic acid induces two different calcium-dependent inner membrane permeability systems in liver mitochondria , 2017, Biochemistry (Moscow), Supplement Series A: Membrane and Cell Biology.
[41] M. Dubinin,et al. Ursodeoxycholic acid, in contrast to other bile acids, induces Ca2+-dependent cyclosporin A-insensitive permeability transition in liver mitochondria in the presence of potassium chloride , 2016, Biochemistry (Moscow) Supplement Series A: Membrane and Cell Biology.
[42] R. Schubert,et al. Membrane/Water Partition Coefficients of Bile Salts Determined Using Laurdan as a Fluorescent Probe. , 2016, Biophysical journal.
[43] Tullio Pozzan,et al. Enjoy the Trip: Calcium in Mitochondria Back and Forth. , 2016, Annual review of biochemistry.
[44] Longkun Li,et al. Hydrophobic bile acids relax rat detrusor contraction via inhibiting the opening of the Na+/Ca2+ exchanger , 2016, Scientific Reports.
[45] M. Prieto,et al. Deoxycholic acid modulates cell death signaling through changes in mitochondrial membrane properties[S] , 2015, Journal of Lipid Research.
[46] E. Moghimipour,et al. Absorption-Enhancing Effects of Bile Salts , 2015, Molecules.
[47] P. Hylemon,et al. Bile acids are nutrient signaling hormones , 2014, Steroids.
[48] H. Jaeschke,et al. Lithocholic acid feeding results in direct hepato-toxicity independent of neutrophil function in mice. , 2014, Toxicology letters.
[49] S. Sollott,et al. Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release. , 2014, Physiological reviews.
[50] M. Zoratti,et al. Mitochondrial channels: ion fluxes and more. , 2014, Physiological reviews.
[51] A. Walch,et al. Progressive stages of mitochondrial destruction caused by cell toxic bile salts. , 2013, Biochimica et biophysica acta.
[52] M. Cicala,et al. Ursodeoxycholic acid therapy in gallbladder disease, a story not yet completed. , 2013, World journal of gastroenterology.
[53] V. Skulachev,et al. Principles of Bioenergetics , 2012 .
[54] C. Baines,et al. Phosphate is not an absolute requirement for the inhibitory effects of cyclosporin A or cyclophilin D deletion on mitochondrial permeability transition. , 2012, The Biochemical journal.
[55] Jianhua Zhang,et al. Autophagy, mitochondria and oxidative stress: cross-talk and redox signalling , 2011, The Biochemical journal.
[56] V. Titorenko,et al. Lithocholic bile acid selectively kills neuroblastoma cells, while sparing normal neuronal cells , 2011, Oncotarget.
[57] J. Sastre,et al. Mitochondrial biogenesis fails in secondary biliary cirrhosis in rats leading to mitochondrial DNA depletion and deletions. , 2011, American journal of physiology. Gastrointestinal and liver physiology.
[58] V. N. Samartsev,et al. Free fatty acids as inducers and regulators of uncoupling of oxidative phosphorylation in liver mitochondria with participation of ADP/ATP- and aspartate/glutamate-antiporter , 2011, Biochemistry (Moscow).
[59] D. Bourdette,et al. Activation of the mitochondrial permeability transition pore modulates Ca2+ responses to physiological stimuli in adult neurons , 2011, The European journal of neuroscience.
[60] J. Farber,et al. Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice. , 2010, The Journal of clinical investigation.
[61] G. Gores,et al. Hepatocyte death: a clear and present danger. , 2010, Physiological reviews.
[62] S. Egelhaaf,et al. Self-assembly in aqueous bile salt solutions , 2010 .
[63] V. Skulachev,et al. Penetrating cation/fatty acid anion pair as a mitochondria-targeted protonophore , 2009, Proceedings of the National Academy of Sciences.
[64] J. Marin,et al. Bile acids: chemistry, physiology, and pathophysiology. , 2009, World journal of gastroenterology.
[65] P. Bernardi,et al. Phosphate Is Essential for Inhibition of the Mitochondrial Permeability Transition Pore by Cyclosporin A and by Cyclophilin D Ablation* , 2008, Journal of Biological Chemistry.
[66] Yuan Zhao,et al. Computation of Octanol-Water Partition Coefficients by Guiding an Additive Model with Knowledge , 2007, J. Chem. Inf. Model..
[67] A. Blume,et al. Membranolytic activity of bile salts: influence of biological membrane properties and composition. , 2007, Molecules.
[68] K. Belosludtsev,et al. Mitochondrial Ca2+ cycle mediated by the palmitate-activated cyclosporin a-insensitive pore , 2007, Journal of bioenergetics and biomembranes.
[69] C. Hill,et al. The interaction between bacteria and bile. , 2005, FEMS microbiology reviews.
[70] Samrat Mukhopadhyay,et al. Chemistry and biology of bile acids , 2004 .
[71] P. Oliveira,et al. Chenodeoxycholate induction of mitochondrial permeability transition pore is associated with increased membrane fluidity and cytochrome c release: protective role of carvedilol. , 2003, Mitochondrion.
[72] P. M. Sokolove,et al. Palmitic acid opens a novel cyclosporin A-insensitive pore in the inner mitochondrial membrane. , 2001, Archives of biochemistry and biophysics.
[73] P. Oliveira,et al. Bile acids affect liver mitochondrial bioenergetics: possible relevance for cholestasis therapy. , 2000, Toxicological sciences : an official journal of the Society of Toxicology.
[74] P. Bernardi,et al. Mitochondrial transport of cations: channels, exchangers, and permeability transition. , 1999, Physiological reviews.
[75] G. Gores,et al. Induction of the mitochondrial permeability transition as a mechanism of liver injury during cholestasis: a potential role for mitochondrial proteases. , 1998, Biochimica et biophysica acta.
[76] V. Skulachev. Uncoupling: new approaches to an old problem of bioenergetics. , 1998, Biochimica et biophysica acta.
[77] R. Moreno-Sánchez,et al. On the Protection by Inorganic Phosphate of Calcium-Induced Membrane Permeability Transition , 1997, Journal of bioenergetics and biomembranes.
[78] A. A. D’Archivio,et al. Calcium Ion Binding to Bile Salts , 1997 .
[79] A. V. Smirnov,et al. Involvement of aspartate/glutamate antiporter in fatty acid-induced uncoupling of liver mitochondria. , 1997, Biochimica et biophysica acta.
[80] M. Carey,et al. Calcium affinity for biliary lipid aggregates in model biles: complementary importance of bile salts and lecithin. , 1994, Gastroenterology.
[81] H. Westerhoff,et al. Movement of fatty acids, fatty acid analogues, and bile acids across phospholipid bilayers. , 1993, Biochemistry.
[82] P. Bernardi,et al. Physiological effectors modify voltage sensing by the cyclosporin A-sensitive permeability transition pore of mitochondria. , 1993, The Journal of biological chemistry.
[83] K. J. Mysels,et al. Bile acid solubility and precipitation in vitro and in vivo: the role of conjugation, pH, and Ca2+ ions. , 1992, Journal of lipid research.
[84] K. J. Mysels,et al. Solubility of calcium salts of unconjugated and conjugated natural bile acids. , 1992, Journal of lipid research.
[85] V. Skulachev. Fatty acid circuit as a physiological mechanism of uncoupling of oxidative phosphorylation , 1991, FEBS letters.
[86] D. Oelberg,et al. Taurine-conjugated bile acids act as Ca2+ ionophores. , 1991, Biochemistry.
[87] B. Hirst,et al. Prostaglandin protects against bile salt induced increases in proton permeation of duodenal brush border membrane. , 1991, Gut.
[88] V. Skulachev,et al. Inhibitors of the ATP/ADP antiporter suppress stimulation of mitochondrial respiration and H+ permeability by palmitate and anionic detergents , 1990, FEBS letters.
[89] A. Roda,et al. Bile acid structure-activity relationship: evaluation of bile acid lipophilicity using 1-octanol/water partition coefficient and reverse phase HPLC. , 1990, Journal of lipid research.
[90] H. Terada. Uncouplers of oxidative phosphorylation. , 1990, Environmental health perspectives.
[91] P. Schönfeld,et al. Does the function of adenine nucleotide translocase in fatty acid uncoupling depend on the type of mitochondria? , 1990, FEBS letters.
[92] V. Skulachev,et al. The ATP/ADP-antiporter is involved in the uncoupling effect of fatty acids on mitochondria. , 1989, European journal of biochemistry.
[93] D. Oelberg,et al. Effect of Bile Acids on Calcium Efflux from Isolated Rat Hepatocytes and Perfused Rat Livers , 1989, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[94] D. Heuman. Quantitative estimation of the hydrophilic-hydrophobic balance of mixed bile salt solutions. , 1989, Journal of lipid research.
[95] M. Anwer,et al. Bile acids increase cellular free calcium in cultured kidney cells (LLC-PK1) , 1988, Pflügers Archiv.
[96] L. Combettes,et al. Release of calcium from the endoplasmic reticulum by bile acids in rat liver cells. , 1988, The Journal of biological chemistry.
[97] A. Roda,et al. Chemical properties of bile acids. IV. Acidity constants of glycine-conjugated bile acids. , 1987, Journal of lipid research.
[98] D. Oelberg,et al. Bile salts induce calcium uptake in vitro by human erythrocytes , 1987, Hepatology.
[99] J. Sunamoto,et al. Bile salt damage of egg phosphatidylcholine liposomes. , 1985, Biochimica et biophysica acta.
[100] A. Hofmann,et al. Physicochemical properties of bile acids and their relationship to biological properties: an overview of the problem. , 1984, Journal of lipid research.
[101] D. Oelberg,et al. Calcium binding by lithocholic acid derivatives. , 1984, The American journal of physiology.
[102] J. Schölmerich,et al. Influence of Hydroxylation and Conjugation of Bile Salts on Their Membrane‐Damaging Properties‐Studies on Isolated Hepatocytes and Lipid Membrane Vesicles , 1984, Hepatology.
[103] A. Shamoo,et al. Anionic detergents as divalent cation ionophores across black lipid membranes , 1979, The Journal of Membrane Biology.
[104] J. K. Thomas,et al. Kinetic studies in bile acid micelles. , 1975, Journal of the American Chemical Society.
[105] S. Schryver. Some investigations on the phenomena of "clot" formations. Part I.―On the clotting of milk , 1913 .
[106] Harpreet Singh,et al. Anion Channels of Mitochondria. , 2017, Handbook of experimental pharmacology.
[107] J. Sastre,et al. Mitochondrial dysfunction in cholestatic liver diseases. , 2012, Frontiers in bioscience.
[108] P. Oliveira,et al. Bile acids are toxic for isolated cardiac mitochondria , 2007, Cardiovascular Toxicology.
[109] G. Forsyth,et al. Ricinoleate and deoxycholate are calcium ionophores in jejunal brush border vesicles , 2005, The Journal of Membrane Biology.
[110] Marianne,et al. Ion Transport by Heart Mitochondria , 2003 .
[111] D. Oelberg,et al. Bile salt-induced calcium fluxes in artificial phospholipid vesicles. , 1988, Biochimica et biophysica acta.
[112] M. Armstrong,et al. The hydrophobic-hydrophilic balance of bile salts. Inverse correlation between reverse-phase high performance liquid chromatographic mobilities and micellar cholesterol-solubilizing capacities. , 1982, Journal of lipid research.
[113] G. Hunt,et al. A 1H-NMR investigation of the mechanism for the ionophore activity of the bile salts in phospholipid vesicular membranes and the effect of cholesterol. , 1980, Biochimica et biophysica acta.