Association of periodontitis with cognitive decline and its progression: Contribution of blood-based biomarkers of Alzheimer's disease to this relationship.
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T. Sobrino | J. Castillo | V. Machado | J. Botelho | Y. Leira | J. Blanco | I. López-Dequidt | J. Pías-Peleteiro | Antía Custodia | Marta Aramburu-Núñez | J. M. Aldrey | Laura Vázquez-Vázquez | A. Ouro | I. Jiménez-Martín | P. Aguiar | M. Rodríguez-Yáñez | Daniel Romaus-Sanjurjo | Á. Carballo
[1] T. Sobrino,et al. Periodontitis is associated with subclinical cerebral and carotid atherosclerosis in hypertensive patients: A cross-sectional study , 2023, Clinical Oral Investigations.
[2] R. Kelley,et al. Emerging Concepts in Vascular Dementia: A Review. , 2021, Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association.
[3] J. Valdés,et al. Alzheimer’s Disease-Like Pathology Triggered by Porphyromonas gingivalis in Wild Type Rats Is Serotype Dependent , 2020, Frontiers in Immunology.
[4] Queralt Miró Catalina,et al. Is systemic inflammation a missing link between periodontitis and hypertension? Results from two large population‐based surveys , 2020, Journal of internal medicine.
[5] D. Liang,et al. Effects of Porphyromonas gingivalis and Its Underlying Mechanisms on Alzheimer-Like Tau Hyperphosphorylation in Sprague-Dawley Rats , 2020, Journal of Molecular Neuroscience.
[6] T. Hortobágyi,et al. An update on blood-based biomarkers for non-Alzheimer neurodegenerative disorders , 2020, Nature Reviews Neurology.
[7] K. Blennow,et al. Plasma P-tau181 in Alzheimer’s disease: relationship to other biomarkers, differential diagnosis, neuropathology and longitudinal progression to Alzheimer’s dementia , 2020, Nature Medicine.
[8] T. Sobrino,et al. Periodontitis and Systemic Markers of Neurodegeneration. A case-control study. , 2020, Journal of clinical periodontology.
[9] K. Blennow,et al. Diagnostic value of plasma phosphorylated tau181 in Alzheimer’s disease and frontotemporal lobar degeneration , 2020, Nature Medicine.
[10] David A Bennett,et al. Diagnosis and Management of Dementia: Review. , 2019, JAMA.
[11] Cheng-En Sung,et al. Association between periodontitis and cognitive impairment: Analysis of national health and nutrition examination survey (NHANES) III. , 2019, Journal of clinical periodontology.
[12] T. Sobrino,et al. Porphyromonas gingivalis lipopolysaccharide-induced periodontitis and serum amyloid-beta peptides. , 2019, Archives of oral biology.
[13] D. Raha,et al. Porphyromonas gingivalis in Alzheimer’s disease brains: Evidence for disease causation and treatment with small-molecule inhibitors , 2019, Science Advances.
[14] T. Sobrino,et al. Periodontitis as a risk indicator and predictor of poor outcome for lacunar infarct , 2018, Journal of clinical periodontology.
[15] M. Ragozzino,et al. Chronic oral application of a periodontal pathogen results in brain inflammation, neurodegeneration and amyloid beta production in wild type mice , 2018, PloS one.
[16] J. Sanmartin Berglund,et al. Longitudinal evaluation of periodontitis and development of cognitive decline among older adults , 2018, Journal of clinical periodontology.
[17] C. Jack,et al. Plasma phospho-tau181 increases with Alzheimer's disease clinical severity and is associated with tau- and amyloid-positron emission tomography , 2018, Alzheimer's & Dementia.
[18] Takashi Kasai,et al. Quantification of plasma phosphorylated tau to use as a biomarker for brain Alzheimer pathology: pilot case-control studies including patients with Alzheimer’s disease and down syndrome , 2017, Molecular Neurodegeneration.
[19] H. Zetterberg,et al. Review: Tau in biofluids – relation to pathology, imaging and clinical features , 2017, Neuropathology and applied neurobiology.
[20] A. Yoshihara,et al. Longitudinal relationship of severe periodontitis with cognitive decline in older Japanese. , 2016, Journal of periodontal research.
[21] B. Choi,et al. Periodontitis Is Associated with Cognitive Impairment in Elderly Koreans: Results from the Yangpyeong Cohort Study , 2016, Journal of the American Geriatrics Society.
[22] P. Eke,et al. Update of the case definitions for population-based surveillance of periodontitis. , 2012, Journal of periodontology.
[23] L. Mucke,et al. Alzheimer Mechanisms and Therapeutic Strategies , 2012, Cell.
[24] A. Spiro,et al. Tooth Loss and Periodontal Disease Predict Poor Cognitive Function in Older Men , 2010, Journal of the American Geriatrics Society.
[25] S. Pocock,et al. The Strengthening the Reporting of Observational Studies in Epidemiology (STROBE) statement: guidelines for reporting observational studies. , 2007, Epidemiology.
[26] F. Manes,et al. Validation of the Spanish version of the Addenbrooke's Cognitive Examination in a rural community in Spain , 2006, International journal of geriatric psychiatry.
[27] J. C. de la Torre,et al. Alzheimer's disease is a vasocognopathy: a new term to describe its nature , 2004, Neurological research.
[28] George A. Carlson,et al. Exogenous Aβ1–40 Reproduces Cerebrovascular Alterations Resulting from Amyloid Precursor Protein Overexpression in Mice , 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[29] C. Iadecola,et al. Abeta 1-40-related reduction in functional hyperemia in mouse neocortex during somatosensory activation. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[30] S. Younkin. Evidence that Aβ42 is the real culprit in alzheimer's disease , 1995 .
[31] D. Mann,et al. Amyloid β protein (Aβ) deposition: Aβ42(43) precedes Aβ40 in down Syndrome , 1995, Annals of neurology.
[32] S. Folstein,et al. “Mini-mental state”: A practical method for grading the cognitive state of patients for the clinician , 1975 .