Epileptiform K Complexes

ABSTRACT. The term “dyshormia” implies paroxysmal responses to arousing stimuli, either electrographically [spiky K complexes in non-rapid eye movement (NREM) sleep] or with clinical seizures (Niedermeyer 1972). It has been shown, however, that focal temporal spikes may also occur in conjunction with K complexes. Accordingly, arousal-induced spiking may occur not only in primary generalized epilepsy (PGE) but also in focal forms of epilepsy. In PGE, the dyshormia phenomenon is obligatory and a basic element. K complexes with focal spikes in temporal lobe epilepsy are rarely observed and facultative, providing evidence of the powerful “drag” of the K complexes from the midline to the area of the focal spike.

[1]  J. Martinerie,et al.  Characterizing Neurodynamic Changes Before Seizures , 2001, Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society.

[2]  E. Niedermeyer Generalized Seizure Discharges and Possible Precipitating Mechanisms , 1966, Epilepsia.

[3]  E. Niedermeyer The Epileptic Pre-Aura , 2002, Clinical EEG.

[4]  Paul R. Carney,et al.  Focal Epileptiform Spikes in Conjuction With K-Complexes , 2006, Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society.

[5]  E. Niedermeyer,et al.  Primary (Idiopathic) Generalized Epilepsy and Underlying Mechanisms , 1996, Clinical EEG.

[6]  C. Panayiotopoulos Reflex Seizures and Reflex Epilepsies , 2005 .

[7]  P. Wolf [Juvenile myoclonic epilepsy]. , 2005, Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova.

[8]  Wieser Hg Temporal lobe epilepsy, sleep and arousal: stereo-EEG findings. , 1991 .