The EAE-associated behavioral syndrome I. Temporal correlation with inflammatory mediators

[1]  I. Goshen,et al.  Experimental autoimmune encephalomyelitis-associated behavioral syndrome as a model of ‘depression due to multiple sclerosis’ , 2002, Brain, Behavior, and Immunity.

[2]  A. Dunn,et al.  Behavioral Effects of Cytokines , 2001, Brain, Behavior, and Immunity.

[3]  R. Dantzer Cytokine-Induced Sickness Behavior: Where Do We Stand? , 2001, Brain, Behavior, and Immunity.

[4]  V. Perry,et al.  Interleukin-1β-Induced Changes in Blood–Brain Barrier Permeability, Apparent Diffusion Coefficient, and Cerebral Blood Volume in the Rat Brain: A Magnetic Resonance Study , 2000, The Journal of Neuroscience.

[5]  P. Deyn,et al.  Experimental autoimmune encephalomyelitis: An animal model for multiple sclerosis , 2000 .

[6]  M. Labow,et al.  The induction of EAE is only partially dependent on TNF receptor signaling but requires the IL-1 type I receptor. , 2000, Clinical immunology.

[7]  I. Goshen,et al.  Behavioral aspects of experimental autoimmune encephalomyelitis , 2000, Journal of Neuroimmunology.

[8]  M E Newman,et al.  Illness, Cytokines, and Depression , 2000, Annals of the New York Academy of Sciences.

[9]  L. Steinman,et al.  Assessment of Animal Models for MS and Demyelinating Disease in the Design of Rational Therapy , 1999, Neuron.

[10]  C. Dinarello Cytokines as endogenous pyrogens. , 1999, The Journal of infectious diseases.

[11]  H. Schluesener,et al.  Cyclooxygenases-1 and -2 are differentially localized to microglia and endothelium in rat EAE and glioma , 1999, Journal of Neuroimmunology.

[12]  K. Frei,et al.  Severity of symptoms and demyelination in MOG‐induced EAE depends on TNFR1 , 1999, European journal of immunology.

[13]  T. Owens,et al.  Immune cell entry to the CNS--a focus for immunoregulation of EAE. , 1998, Research in immunology.

[14]  A. Juedes,et al.  Cytokines and antibodies in myelin oligodendrocyte glycoprotein-induced experimental allergic encephalomyelitis. , 1998, Research in immunology.

[15]  W. Karpus,et al.  Chemokine regulation of CNS T-cell infiltration in experimental autoimmune encephalomyelitis. , 1998, Research in immunology.

[16]  S. Khoury,et al.  Kinetics of expression of costimulatory molecules and their ligands in murine relapsing experimental autoimmune encephalomyelitis in vivo. , 1998, Journal of immunology.

[17]  D. Danilenko,et al.  Combined Treatment of Acute EAE in Lewis Rats with TNF-Binding Protein and Interleukin-1 Receptor Antagonist , 1998, Experimental Neurology.

[18]  B. Leonard,et al.  Depression, stress and immunological activation: the role of cytokines in depressive disorders. , 1998, Life sciences.

[19]  Y. Pollak,et al.  Administration of interleukin-1 into the hypothalamic paraventricular nucleus induces febrile and behavioral effects. , 1997, Neuroimmunomodulation.

[20]  M. Cuzner,et al.  Apoptosis of T cells and macrophages in the central nervous system of intact and adrenalectomized Lewis rats during experimental allergic encephalomyelitis. , 1996, Journal of autoimmunity.

[21]  S. Sakoda,et al.  Expression of the inducible isoform of nitric oxide synthase in the central nervous system of mice correlates with the severity of actively induced experimental allergic encephalomyelitis , 1995, Journal of Neuroimmunology.

[22]  T. Olsson Cytokine-producing cells in experimental autoimmune encephalomyelitis and multiple sclerosis , 1995, Neurology.

[23]  H. Lassmann,et al.  Apoptosis of T lymphocytes in experimental autoimmune encephalomyelitis. Evidence for programmed cell death as a mechanism to control inflammation in the brain. , 1993, The American journal of pathology.

[24]  D. Schubert,et al.  Increased depression in multiple sclerosis patients. A meta-analysis. , 1993, Psychosomatics.

[25]  K. Mohler,et al.  Analysis of cytokine mRNA expression in the central nervous system of mice with experimental autoimmune encephalomyelitis reveals that IL-10 mRNA expression correlates with recovery. , 1992, Journal of immunology.

[26]  Y. Abu-Amer,et al.  Inhibition of prostaglandin synthesis in brain of rat by dexamethasone: Lack of effect of dexamethasone phosphate ester and various hormonal steroids , 1988, Neuropharmacology.

[27]  R. Schiffer,et al.  Behavioral disorders in multiple sclerosis, temporal lobe epilepsy, and amyotrophic lateral sclerosis. An epidemiologic study. , 1984, Archives of neurology.

[28]  F. A. Whitlock,et al.  Depression as a major symptom of multiple sclerosis. , 1980, Journal of neurology, neurosurgery, and psychiatry.

[29]  A. Clow Cytokines and depression. , 2002, International review of neurobiology.

[30]  A. Reichenberg,et al.  Cytokines, "depression due to a general medical condition," and antidepressant drugs. , 1999, Advances in experimental medicine and biology.

[31]  S. Maier,et al.  Cytokines for psychologists: implications of bidirectional immune-to-brain communication for understanding behavior, mood, and cognition. , 1998, Psychological review.

[32]  S. Krajewski,et al.  Tumor necrosis factor-alpha induced pathology in the rat brain: characterization of stereotaxic injection model. , 1998, Folia neuropathologica.

[33]  M. Hennerici,et al.  Mood disorders and dysfunction of the hypothalamic-pituitary-adrenal axis in multiple sclerosis: association with cerebral inflammation. , 1998, Archives of neurology.

[34]  S. Miller,et al.  Cytokines as intrinsic and exogenous regulators of pathogenesis in experimental autoimmune encephalomyelitis. , 1998, Research in immunology.