An HMGA2-IGF2BP2 axis regulates myoblast proliferation and myogenesis.
暂无分享,去创建一个
Yunyu Zhang | M. Goddeeris | K. Campbell | D. Kirsch | D. Glass | T. Shavlakadze | A. Brack | Zhizhong Li | J. Gilbert | J. Eash | Qiong Qiu | Minsi Zhang | K. Ramanujan | A. Scaramozza
[1] J. Avruch,et al. mTOR phosphorylates IMP2 to promote IGF2 mRNA translation by internal ribosomal entry. , 2011, Genes & development.
[2] Y. Yen,et al. High-Mobility Group A2 Protein Modulates hTERT Transcription To Promote Tumorigenesis , 2011, Molecular and Cellular Biology.
[3] D. Glass,et al. Molecular mechanisms and treatment options for muscle wasting diseases. , 2011, Annual review of pharmacology and toxicology.
[4] Valentina Proserpio,et al. TNF/p38α/polycomb signaling to Pax7 locus in satellite cells links inflammation to the epigenetic control of muscle regeneration. , 2010, Cell stem cell.
[5] G. Shefer,et al. The depletion of skeletal muscle satellite cells with age is concomitant with reduced capacity of single progenitors to produce reserve progeny. , 2010, Developmental biology.
[6] Scott B. Dewell,et al. Transcriptome-wide Identification of RNA-Binding Protein and MicroRNA Target Sites by PAR-CLIP , 2010, Cell.
[7] A. Fusco,et al. HMGA and cancer. , 2010, Biochimica et biophysica acta.
[8] K. Chada,et al. In vivo modulation of HMGA2 expression. , 2010, Biochimica et biophysica acta.
[9] T. Hansen,et al. IGF2 mRNA-binding protein 2: biological function and putative role in type 2 diabetes. , 2009, Journal of molecular endocrinology.
[10] J. Hescheler,et al. The High Mobility Group Protein HMGA2: A Co-Regulator of Chromatin Structure and Pluripotency in Stem Cells? , 2009, Stem Cell Reviews and Reports.
[11] John K. Hall,et al. Syndecan-4-expressing muscle progenitor cells in the SP engraft as satellite cells during muscle regeneration. , 2009, Cell stem cell.
[12] M. Nakao,et al. HMGA2 maintains oncogenic RAS-induced epithelial-mesenchymal transition in human pancreatic cancer cells. , 2009, The American journal of pathology.
[13] Sean J. Morrison,et al. Hmga2 Promotes Neural Stem Cell Self-Renewal in Young but Not Old Mice by Reducing p16Ink4a and p19Arf Expression , 2008, Cell.
[14] Helen M Blau,et al. Cell therapies for muscular dystrophy. , 2008, The New England journal of medicine.
[15] Y. Hiroi,et al. A crucial role of a high mobility group protein HMGA2 in cardiogenesis , 2008, Nature Cell Biology.
[16] M. Rudnicki,et al. The molecular regulation of muscle stem cell function. , 2008, Cold Spring Harbor symposia on quantitative biology.
[17] M. Rudnicki,et al. Skeletal muscle satellite cells and adult myogenesis. , 2007, Current opinion in cell biology.
[18] A. Uezumi,et al. Molecular Signature of Quiescent Satellite Cells in Adult Skeletal Muscle , 2007, Stem cells.
[19] T. Rando,et al. Intrinsic Changes and Extrinsic Influences of Myogenic Stem Cell Function During Aging , 2007, Stem Cell Reviews.
[20] R. Sciot,et al. HMGA2 Regulates Transcription of the Imp2 Gene via an Intronic Regulatory Element in Cooperation with Nuclear Factor-κB , 2007, Molecular Cancer Research.
[21] G. Viglietto,et al. Haploinsufficiency of the Hmga1 gene causes cardiac hypertrophy and myelo-lymphoproliferative disorders in mice. , 2006, Cancer research.
[22] F. Bost,et al. A new role for the oncogenic high-mobility group A2 transcription factor in myogenesis of embryonic stem cells , 2005, Oncogene.
[23] I. Weissman,et al. Rejuvenation of aged progenitor cells by exposure to a young systemic environment , 2005, Nature.
[24] B. Olwin,et al. Pax-7 up-regulation inhibits myogenesis and cell cycle progression in satellite cells: a potential mechanism for self-renewal. , 2004, Developmental biology.
[25] I. Weissman,et al. Isolation of Adult Mouse Myogenic Progenitors Functional Heterogeneity of Cells within and Engrafting Skeletal Muscle , 2004, Cell.
[26] T. Partridge,et al. Muscle satellite cells adopt divergent fates , 2004, The Journal of cell biology.
[27] Y. Tanabe,et al. Skeletal muscle pathology in X chromosome-linked muscular dystrophy (mdx) mouse , 2004, Acta Neuropathologica.
[28] M. Luster,et al. Physiological role of tumor necrosis factor α in traumatic muscle injury , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[29] D J Glass,et al. Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy , 2001, Science.
[30] J. Black,et al. Sp1 and krüppel‐like factor family of transcription factors in cell growth regulation and cancer , 2001, Journal of cellular physiology.
[31] J. Dimario,et al. Two distal Sp1-binding cis-elements regulate fibroblast growth factor receptor 1 (FGFR1) gene expression in myoblasts. , 2001, Gene.
[32] K. Chada,et al. In vivo modulation of Hmgic reduces obesity , 2000, Nature Genetics.
[33] N. Rosenthal,et al. Proliferation precedes differentiation in IGF-I-stimulated myogenesis , 1996, The Journal of cell biology.
[34] D. Gillespie,et al. c-Myc inhibits myogenic differentiation and myoD expression by a mechanism which can be dissociated from cell transformation. , 1994, Oncogene.
[35] M. Kaleko,et al. Type‐1 insulin‐like growth factor receptor overexpression produces dual effects on myoblast proliferation and differentiation , 1994, Journal of cellular physiology.
[36] J. Baker,et al. Mice carrying null mutations of the genes encoding insulin-like growth factor I (Igf-1) and type 1 IGF receptor (Igf1r) , 1993, Cell.
[37] L. S. Quinn,et al. Overexpression of the human type-1 insulin-like growth factor receptor in rat L6 myoblasts induces ligand-dependent cell proliferation and inhibition of differentiation. , 1993, Experimental cell research.
[38] B. Wold,et al. c-myc inhibition of MyoD and myogenin-initiated myogenic differentiation , 1991, Molecular and cellular biology.
[39] J. V. Van Wyk,et al. Biphasic concentration dependency of stimulation of myoblast differentiation by somatomedins. , 1986, The American journal of physiology.
[40] K. Moore,et al. X chromosome-linked muscular dystrophy (mdx) in the mouse. , 1984, Proceedings of the National Academy of Sciences of the United States of America.