Gene Polymorphism KIR3DL1 Defined by the DX9 Antibody Are Due to Different NK Cell Surface Phenotypes
暂无分享,去创建一个
P. Parham | L. Guethlein | R. Rajalingam | C. Gardiner | C. Vilches | M. Pando | W. Carr | Shilling
[1] P Parham,et al. Identification of seventeen novel KIR variants: fourteen of them from two non-Caucasian donors. , 2001, Tissue antigens.
[2] D Middleton,et al. Development of a PCR-SSOP approach capable of defining the natural killer cell inhibitory receptor (KIR) gene sequence repertoires. , 2000, Tissue antigens.
[3] Markus Uhrberg,et al. KIR2DL5, a Novel Killer-Cell Receptor with a D0-D2 Configuration of Ig-Like Domains1 2 , 2000, The Journal of Immunology.
[4] D. G. Muir,et al. Rapid evolution of NK cell receptor systems demonstrated by comparison of chimpanzees and humans. , 2000, Immunity.
[5] Jeffrey C. Boyington,et al. Crystal structure of an NK cell immunoglobulin-like receptor in complex with its class I MHC ligand , 2000, Nature.
[6] S. Beck,et al. Plasticity in the organization and sequences of human KIR/ILT gene families. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[7] Rolf Apweiler,et al. The SWISS-PROT protein sequence database and its supplement TrEMBL in 2000 , 2000, Nucleic Acids Res..
[8] C. Witt,et al. Population frequencies and putative haplotypes of the killer cell immunoglobulin-like receptor sequences and evidence for recombination. , 1999, Transplantation.
[9] J. Coligan,et al. Structure of CD94 reveals a novel C-type lectin fold: implications for the NK cell-associated CD94/NKG2 receptors. , 1999, Immunity.
[10] J. Coligan,et al. Specific recognition of HLA-E, but not classical, HLA class I molecules by soluble CD94/NKG2A and NK cells. , 1999, Journal of immunology.
[11] C. Biron,et al. Natural killer cells in antiviral defense: function and regulation by innate cytokines. , 1999, Annual review of immunology.
[12] A. Selvakumar,et al. Multiple transcripts of the killer cell immunoglobulin-like receptor family, KIR3DL1 (NKB1), are expressed by natural killer cells of a single individual. , 1998, Tissue antigens.
[13] John Trowsdale,et al. Isotypic variation of novel immunoglobulin‐like transcript/killer cell inhibitory receptor loci in the leukocyte receptor complex , 1998, European journal of immunology.
[14] P. Parham,et al. Evidence for recombination as a mechanism for KIR diversification , 1998, Immunogenetics.
[15] J. Bell,et al. HLA-E binds to natural killer cell receptors CD94/NKG2A, B and C , 1998, Nature.
[16] L. Lanier. NK cell receptors. , 1998, Annual review of immunology.
[17] P Parham,et al. Functionally and structurally distinct NK cell receptor repertoires in the peripheral blood of two human donors. , 1997, Immunity.
[18] P Parham,et al. Human diversity in killer cell inhibitory receptor genes. , 1997, Immunity.
[19] T. Ohta,et al. Episodic evolution and turnover of HLA-B in the indigenous human populations of the Americas. , 1997, Tissue antigens.
[20] W R Mayr,et al. Nomenclature for Factors of the HLA System, 1996 , 1997, European journal of immunogenetics : official journal of the British Society for Histocompatibility and Immunogenetics.
[21] W. Yokoyama,et al. The natural killer gene complex: a genetic basis for understanding natural killer cell function and innate immunity , 1997, Immunological reviews.
[22] Eric O Long,et al. Killer cell inhibitory receptors: diversity, specificity, and function , 1997, Immunological reviews.
[23] Eric O Long,et al. Binding of a soluble p70 killer cell inhibitory receptor to HLA‐B*5101: Requirement for all three p70 immunoglobulin domains , 1997, European journal of immunology.
[24] Rolf Apweiler,et al. The SWISS-PROT protein sequence data bank and its supplement TrEMBL , 1997, Nucleic Acids Res..
[25] R. Biassoni,et al. The natural killer cell receptor specific for HLA-A allotypes: a novel member of the p58/p70 family of inhibitory receptors that is characterized by three immunoglobulin-like domains and is expressed as a 140-kD disulphide-linked dimer , 1996, The Journal of experimental medicine.
[26] M. Colonna,et al. A human killer inhibitory receptor specific for HLA-A1,2. , 1996, Journal of immunology.
[27] P. Parham,et al. Heterogeneous phenotypes of expression of the NKB1 natural killer cell class I receptor among individuals of different human histocompatibility leukocyte antigens types appear genetically regulated, but not linked to major histocompatibililty complex haplotype , 1996, The Journal of experimental medicine.
[28] Eric O Long,et al. Inhibitory MHC class I receptors on NK and T cells: a standard nomenclature. , 1996, Immunology today.
[29] T. Mcclanahan,et al. Molecular cloning of NKB1. A natural killer cell receptor for HLA-B allotypes. , 1995, Journal of immunology.
[30] M. Colonna,et al. Cloning of immunoglobulin-superfamily members associated with HLA-C and HLA-B recognition by human natural killer cells. , 1995, Science.
[31] P. Parham,et al. The Bw4 public epitope of HLA-B molecules confers reactivity with natural killer cell clones that express NKB1, a putative HLA receptor , 1995, The Journal of experimental medicine.
[32] G. Ferrara,et al. NK3-specific natural killer cells are selectively inhibited by Bw4- positive HLA alleles with isoleucine 80 , 1994, The Journal of experimental medicine.
[33] P. Parham,et al. NKB1: a natural killer cell receptor involved in the recognition of polymorphic HLA-B molecules , 1994, The Journal of experimental medicine.
[34] G. Ferrara,et al. HLA-C is the inhibitory ligand that determines dominant resistance to lysis by NK1- and NK2-specific natural killer cells. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[35] E. Ciccone,et al. Identification of four subsets of human CD3-CD16+ natural killer (NK) cells by the expression of clonally distributed functional surface molecules: correlation between subset assignment of NK clones and ability to mediate specific alloantigen recognition , 1990, The Journal of experimental medicine.
[36] M. Torres,et al. Nomenclature for factors of the HLA system. , 2011, Bulletin of the World Health Organization.