Cardioprotective signaling to mitochondria.
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[1] L. Argaud,et al. PERSISTENT INHIBITION OF MITOCHONDRIAL PERMEABILITY TRANSITION BY PRECONDITIONING DURING THE FIRST HOURS OF REPERFUSION , 2008, Shock.
[2] R. Zucchi,et al. Cardioprotection by Ouabain and Digoxin in Perfused Rat Hearts , 2008, Journal of cardiovascular pharmacology.
[3] M. Disatnik,et al. Activation of Aldehyde Dehydrogenase-2 Reduces Ischemic Damage to the Heart , 2008, Science.
[4] P. dos Santos,et al. Conditioning the heart induces formation of signalosomes that interact with mitochondria to open mitoKATP channels. , 2008, American journal of physiology. Heart and circulatory physiology.
[5] K. Garlid,et al. Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT. , 2008, American journal of physiology. Heart and circulatory physiology.
[6] K. Garlid,et al. S3/7 Intramitochondrial signaling — Interactions among mitoKATP, PKCε, ROS, and MPT , 2008 .
[7] A. Wojtovich,et al. The endogenous mitochondrial complex II inhibitor malonate regulates mitochondrial ATP-sensitive potassium channels: implications for ischemic preconditioning. , 2008, Biochimica et biophysica acta.
[8] D. Stowe,et al. Differential Increase of Mitochondrial Matrix Volume by Sevoflurane in Isolated Cardiac Mitochondria , 2008, Anesthesia and analgesia.
[9] C. Hoppel,et al. Ischemic defects in the electron transport chain increase the production of reactive oxygen species from isolated rat heart mitochondria. , 2008, American journal of physiology. Cell physiology.
[10] Michael V. Cohen,et al. cGMP signalling in pre- and post-conditioning: the role of mitochondria. , 2008, Cardiovascular research.
[11] Haixia Huang,et al. Hydrogen sulfide contributes to cardioprotection during ischemia-reperfusion injury by opening K ATP channels. , 2007, Canadian journal of physiology and pharmacology.
[12] P. Ping,et al. Past and present course of cardioprotection against ischemia-reperfusion injury. , 2007, Journal of applied physiology.
[13] D. Mochly‐Rosen,et al. Mitochondrial protein kinase Cepsilon (PKCepsilon): emerging role in cardiac protection from ischaemic damage. , 2007, Biochemical Society transactions.
[14] V. Garg,et al. Protein kinase C isoform-dependent modulation of ATP-sensitive K+ channels in mitochondrial inner membrane. , 2007, American journal of physiology. Heart and circulatory physiology.
[15] Michael V. Cohen,et al. Signaling pathways in ischemic preconditioning , 2007, Heart Failure Reviews.
[16] P. Bernardi,et al. Mitochondria and cardioprotection , 2007, Heart Failure Reviews.
[17] P. Insel,et al. Mechanisms of cardiac protection from ischemia/reperfusion injury: a role for caveolae and caveolin‐1 , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[18] A. Hsu,et al. GSK3β inhibition and KATP channel opening mediate acute opioid-induced cardioprotection at reperfusion , 2007, Basic Research in Cardiology.
[19] A. Kowaltowski,et al. Mitochondrial ATP-sensitive K+ channels are redox-sensitive pathways that control reactive oxygen species production. , 2007, Free radical biology & medicine.
[20] P. Pasdois,et al. Ouabain protects rat hearts against ischemia-reperfusion injury via pathway involving src kinase, mitoKATP, and ROS. , 2007, American journal of physiology. Heart and circulatory physiology.
[21] E. Murphy,et al. Preconditioning: the mitochondrial connection. , 2007, Annual review of physiology.
[22] S. Pierre,et al. Ouabain triggers preconditioning through activation of the Na+,K+-ATPase signaling cascade in rat hearts. , 2007, Cardiovascular research.
[23] K. Fuxe,et al. Partners for adenosine A1 receptors , 2007, Journal of Molecular Neuroscience.
[24] C. Hoppel,et al. Reversible Blockade of Electron Transport during Ischemia Protects Mitochondria and Decreases Myocardial Injury following Reperfusion , 2006, Journal of Pharmacology and Experimental Therapeutics.
[25] I. West,et al. Opening mitoKATP increases superoxide generation from complex I of the electron transport chain. , 2006, American journal of physiology. Heart and circulatory physiology.
[26] K. Olson,et al. Hydrogen sulfide as an oxygen sensor/transducer in vertebrate hypoxic vasoconstriction and hypoxic vasodilation , 2006, Journal of Experimental Biology.
[27] N. Maniatis,et al. Novel Mechanism of Endothelial Nitric Oxide Synthase Activation Mediated by Caveolae Internalization in Endothelial Cells , 2006, Circulation research.
[28] M. Jabůrek,et al. Mitochondrial PKC&egr; and Mitochondrial ATP-Sensitive K+ Channel Copurify and Coreconstitute to Form a Functioning Signaling Module in Proteoliposomes , 2006, Circulation research.
[29] I. West,et al. The Mechanism by Which the Mitochondrial ATP-sensitive K+ Channel Opening and H2O2 Inhibit the Mitochondrial Permeability Transition* , 2006, Journal of Biological Chemistry.
[30] P. Puddu,et al. Inhibition of cardiac contractility by 5-hydroxydecanoate and tetraphenylphosphonium ion: a possible role of mitoKATP in response to inotropic stress. , 2006, American journal of physiology. Heart and circulatory physiology.
[31] P. Insel,et al. Protection of adult rat cardiac myocytes from ischemic cell death: role of caveolar microdomains and delta-opioid receptors. , 2006, American journal of physiology. Heart and circulatory physiology.
[32] P. Puddu,et al. Bimakalim: A Promising KATP Channel Activating Agent , 2006 .
[33] G. Gross,et al. Ligand triggers of classical preconditioning and postconditioning. , 2006, Cardiovascular research.
[34] P. Bernardi,et al. Mitochondria and ischemia-reperfusion injury of the heart: fixing a hole. , 2006, Cardiovascular research.
[35] L. Argaud,et al. Mitochondrial permeability transition pore and postconditioning. , 2006, Cardiovascular research.
[36] Z. Bosnjak,et al. Characterization of human cardiac mitochondrial ATP-sensitive potassium channel and its regulation by phorbol ester in vitro. , 2006, American journal of physiology. Heart and circulatory physiology.
[37] P. Pagliaro,et al. Post–conditioning induced cardioprotection requires signaling through a redox–sensitive mechanism, mitochondrial ATP–sensitive K+ channel and protein kinase C activation , 2006, Basic Research in Cardiology.
[38] Shufeng Zhou,et al. Role of Hydrogen Sulfide in the Cardioprotection Caused by Ischemic Preconditioning in the Rat Heart and Cardiac Myocytes , 2006, Journal of Pharmacology and Experimental Therapeutics.
[39] I. West,et al. The direct physiological effects of mitoK(ATP) opening on heart mitochondria. , 2006, American journal of physiology. Heart and circulatory physiology.
[40] Steven P Jones,et al. The ubiquitous role of nitric oxide in cardioprotection. , 2006, Journal of molecular and cellular cardiology.
[41] J. Downey,et al. Protein Kinase G Transmits the Cardioprotective Signal From Cytosol to Mitochondria , 2005, Circulation research.
[42] H. Hsieh,et al. Bradykinin‐induced p42/p44 MAPK phosphorylation and cell proliferation via Src, EGF receptors, and PI3‐K/Akt in vascular smooth muscle cells , 2005, Journal of cellular physiology.
[43] J. Downey,et al. Peptide blockers of PKG inhibit ROS generation by acetylcholine and bradykinin in cardiomyocytes but fail to block protection in the whole heart. , 2005, American journal of physiology. Heart and circulatory physiology.
[44] P. Ping,et al. Nitric oxide donors protect murine myocardium against infarction via modulation of mitochondrial permeability transition. , 2005, American journal of physiology. Heart and circulatory physiology.
[45] Q. Xia,et al. Calcium-Activated Potassium Channel Triggers Cardioprotection of Ischemic Preconditioning , 2005, Journal of Pharmacology and Experimental Therapeutics.
[46] H. Nakaya,et al. Mitochondrial Ca2+-Activated K+ Channels in Cardiac Myocytes: A Mechanism of the Cardioprotective Effect and Modulation by Protein Kinase A , 2005, Circulation.
[47] L. Argaud,et al. Postconditioning Inhibits Mitochondrial Permeability Transition , 2005, Circulation.
[48] G. Baxter,et al. Exogenous hydrogen sulfide (H2S) protects against regional myocardial ischemia–reperfusion injury , 2005, Basic Research in Cardiology.
[49] Shigetoshi Ohshima,et al. Nitric oxide: a signaling molecule against mitochondrial permeability transition- and pH-dependent cell death after reperfusion. , 2004, Free radical biology & medicine.
[50] S. Steinberg. Distinctive activation mechanisms and functions for protein kinase Cdelta. , 2004, The Biochemical journal.
[51] Xiaoying Wang,et al. Opening of Ca2+-activated K+ channels triggers early and delayed preconditioning against I/R injury independent of NOS in mice. , 2004, American journal of physiology. Heart and circulatory physiology.
[52] M. Ericsson,et al. Opening of mitochondrial KATP channels enhances cardioprotection through the modulation of mitochondrial matrix volume, calcium accumulation, and respiration. , 2004, American journal of physiology. Heart and circulatory physiology.
[53] Peipei Ping,et al. Scaffold proteins and assembly of multiprotein signaling complexes. , 2004, Journal of molecular and cellular cardiology.
[54] J. Shapiro,et al. Ouabain induces endocytosis of plasmalemmal Na/K-ATPase in LLC-PK1 cells by a clathrin-dependent mechanism. , 2004, Kidney international.
[55] B. Geng,et al. Endogenous hydrogen sulfide regulation of myocardial injury induced by isoproterenol. , 2004, Biochemical and biophysical research communications.
[56] Shigetoshi Ohshima,et al. Nitric oxide protects rat hepatocytes against reperfusion injury mediated by the mitochondrial permeability transition , 2004, Hepatology.
[57] E. Olson,et al. Glycogen synthase kinase-3β mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore , 2004 .
[58] W. Koch,et al. G Protein–Coupled Receptor Internalization Signaling Is Required for Cardioprotection in Ischemic Preconditioning , 2004, Circulation research.
[59] J. Downey,et al. Mitochondrial ROS generation following acetylcholine-induced EGF receptor transactivation requires metalloproteinase cleavage of proHB-EGF. , 2004, Journal of molecular and cellular cardiology.
[60] E. Olson,et al. Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore. , 2004, The Journal of clinical investigation.
[61] Jiang Tian,et al. Involvement of mitogen-activated protein kinases and reactive oxygen species in the inotropic action of ouabain on cardiac myocytes. A potential role for mitochondrial KATP channels , 2004, Molecular and Cellular Biochemistry.
[62] J. Downey,et al. Bradykinin induces mitochondrial ROS generation via NO, cGMP, PKG, and mitoKATP channel opening and leads to cardioprotection. , 2004, American journal of physiology. Heart and circulatory physiology.
[63] D. Mochly‐Rosen,et al. Opposing roles of δ and εPKC in cardiac ischemia and reperfusion: targeting the apoptotic machinery , 2003 .
[64] B. Nichols. Caveosomes and endocytosis of lipid rafts , 2003, Journal of Cell Science.
[65] J. Turrens,et al. Mitochondrial formation of reactive oxygen species , 2003, The Journal of physiology.
[66] Keith D Garlid,et al. Mitochondrial potassium transport: the K(+) cycle. , 2003, Biochimica et biophysica acta.
[67] Michael V. Cohen,et al. P1075 opens mitochondrial K(ATP) channels and generates reactive oxygen species resulting in cardioprotection of rabbit hearts. , 2003, Journal of molecular and cellular cardiology.
[68] Peipei Ping,et al. Role of the mitochondrial permeability transition in myocardial disease. , 2003, Circulation research.
[69] Michael V. Cohen,et al. Mitochondrial KATP channels in preconditioning , 2003 .
[70] J. Downey,et al. Acetylcholine-induced production of reactive oxygen species in adult rabbit ventricular myocytes is dependent on phosphatidylinositol 3- and Src-kinase activation and mitochondrial K(ATP) channel opening. , 2003, Journal of molecular and cellular cardiology.
[71] Zijian Xie,et al. Na+-K+--ATPase-mediated signal transduction: from protein interaction to cellular function. , 2003, Molecular interventions.
[72] F. Ciruela,et al. Ligand-induced caveolae-mediated internalization of A1 adenosine receptors: morphological evidence of endosomal sorting and receptor recycling. , 2003, Experimental cell research.
[73] L. Kevin,et al. Ischemic preconditioning alters real-time measure of O2 radicals in intact hearts with ischemia and reperfusion. , 2003, American journal of physiology. Heart and circulatory physiology.
[74] D. Mochly‐Rosen,et al. Opposing roles of delta and epsilonPKC in cardiac ischemia and reperfusion: targeting the apoptotic machinery. , 2003, Archives of biochemistry and biophysics.
[75] Jiang Tian,et al. Involvement of mitogen-activated protein kinases and reactive oxygen species in the inotropic action of ouabain on cardiac myocytes. A potential role for mitochondrial K ATP channels , 2003 .
[76] T. Vanden Hoek,et al. ROS and NO trigger early preconditioning: relationship to mitochondrial KATP channel. , 2003, American journal of physiology. Heart and circulatory physiology.
[77] J. Downey,et al. ACh and adenosine activate PI3-kinase in rabbit hearts through transactivation of receptor tyrosine kinases. , 2002, American journal of physiology. Heart and circulatory physiology.
[78] B. Hoyos,et al. Zinc Release from Protein Kinase C as the Common Event during Activation by Lipid Second Messenger or Reactive Oxygen* , 2002, The Journal of Biological Chemistry.
[79] Yongge Liu,et al. Cytoprotective Role of Ca2+- Activated K+ Channels in the Cardiac Inner Mitochondrial Membrane , 2002, Science.
[80] S. Woodman,et al. Caveolae: From Cell Biology to Animal Physiology , 2002, Pharmacological Reviews.
[81] K. Klotz,et al. Involvement of mitogen protein kinase cascade in agonist-mediated human A(3) adenosine receptor regulation. , 2002, Biochimica et biophysica acta.
[82] J. Krieglstein,et al. Serine/threonine protein phosphatases in apoptosis. , 2002, Current opinion in pharmacology.
[83] D. Yellon,et al. Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning? , 2002, Cardiovascular research.
[84] E. Murphy,et al. Phosphorylation of Glycogen Synthase Kinase-3&bgr; During Preconditioning Through a Phosphatidylinositol-3-Kinase–Dependent Pathway Is Cardioprotective , 2002, Circulation research.
[85] J. Weiss,et al. Protection of cardiac mitochondria by diazoxide and protein kinase C: Implications for ischemic preconditioning , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[86] R. Gottlieb,et al. Inhibition of mitochondrial calcium-independent phospholipase A2 (iPLA2) attenuates mitochondrial phospholipid loss and is cardioprotective. , 2002, The Biochemical journal.
[87] W. Dröge. Free radicals in the physiological control of cell function. , 2002, Physiological reviews.
[88] G. Gross,et al. Characteristics and Superoxide-Induced Activation of Reconstituted Myocardial Mitochondrial ATP-Sensitive Potassium Channels , 2001, Circulation research.
[89] J. Downey,et al. Acetylcholine, Bradykinin, Opioids, and Phenylephrine, but not Adenosine, Trigger Preconditioning by Generating Free Radicals and Opening Mitochondrial KATP Channels , 2001, Circulation research.
[90] T. Slabe,et al. Myocardial ischemia selectively depletes cardiolipin in rabbit heart subsarcolemmal mitochondria. , 2001, American journal of physiology. Heart and circulatory physiology.
[91] G. Grover,et al. Pharmacologic characterization of BMS-191095, a mitochondrial K(ATP) opener with no peripheral vasodilator or cardiac action potential shortening activity. , 2001, The Journal of pharmacology and experimental therapeutics.
[92] E. Murphy,et al. Diazoxide-Induced Cardioprotection Requires Signaling Through a Redox-Sensitive Mechanism , 2001, Circulation research.
[93] A. Kowaltowski,et al. Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria. , 2001, American journal of physiology. Heart and circulatory physiology.
[94] P. Bernardi,et al. Opening of the Mitochondrial Permeability Transition Pore Causes Depletion of Mitochondrial and Cytosolic NAD+and Is a Causative Event in the Death of Myocytes in Postischemic Reperfusion of the Heart* , 2001, The Journal of Biological Chemistry.
[95] W. Pierce,et al. Functional Proteomic Analysis of Protein Kinase C &egr; Signaling Complexes in the Normal Heart and During Cardioprotection , 2001, Circulation research.
[96] C. Moreau,et al. The molecular basis of the specificity of action of KATP channel openers , 2000, The EMBO journal.
[97] K. Klotz,et al. Agonist‐Induced Internalization and Recycling of the Human A3 Adenosine Receptors , 2000 .
[98] J. Headrick,et al. Cardioprotection by KATP channels in wild-type hearts and hearts overexpressing A1 adenosine receptors , 2000 .
[99] J. Downey,et al. Opening of Mitochondrial KATP Channels Triggers the Preconditioned State by Generating Free Radicals , 2000, Circulation research.
[100] E. Marbán,et al. ATP-Sensitive Potassium Channels , 2021, Encyclopedia of Molecular Pharmacology.
[101] G. Grover,et al. ATP-Sensitive potassium channels: a review of their cardioprotective pharmacology. , 2000, Journal of molecular and cellular cardiology.
[102] A. Reshef,et al. Opening of KATP channels is mandatory for acquisition of ischemic tolerance by adenosine , 2000, Neuroreport.
[103] E. Marbán,et al. Activation of mitochondrial ATP-dependent potassium channels by nitric oxide. , 2000, Circulation.
[104] K. Garlid. The state of water in biological systems. , 2000, International review of cytology.
[105] K. Klotz,et al. Agonist-induced internalization and recycling of the human A(3) adenosine receptors: role in receptor desensitization and resensitization. , 2000, Journal of Neurochemistry.
[106] J. Headrick,et al. Cardioprotection by K(ATP) channels in wild-type hearts and hearts overexpressing A(1)-adenosine receptors. , 2000, American journal of physiology. Heart and circulatory physiology.
[107] J. Engelman,et al. Caveolins, Liquid-Ordered Domains, and Signal Transduction , 1999, Molecular and Cellular Biology.
[108] N. Di Fonzo,et al. The Existence of the K+ Channel in Plant Mitochondria* , 1999, The Journal of Biological Chemistry.
[109] M Crompton,et al. The mitochondrial permeability transition pore and its role in cell death. , 1999, The Biochemical journal.
[110] J. Downey,et al. Signal Transduction in Ischemic Preconditioning: , 1999, Journal of cardiovascular electrophysiology.
[111] W. Müller-Esterl,et al. Bradykinin-induced Internalization of the Human B2Receptor Requires Phosphorylation of Three Serine and Two Threonine Residues at Its Carboxyl Tail* , 1999, The Journal of Biological Chemistry.
[112] D. Golan,et al. Receptor-regulated Translocation of Endothelial Nitric-oxide Synthase* , 1998, The Journal of Biological Chemistry.
[113] D. Mochly‐Rosen,et al. Peptide modulators of protein–protein interactions in intracellular signaling , 1998, Nature Biotechnology.
[114] V. Yarov-Yarovoy,et al. State-dependent inhibition of the mitochondrial KATP channel by glyburide and 5-hydroxydecanoate. , 1998, The Journal of biological chemistry.
[115] M. Haasemann,et al. Agonist-induced redistribution of bradykinin B2 receptor in caveolae. , 1998, Journal of cell science.
[116] M. Smith,et al. Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection. , 1997, Circulation research.
[117] R. Reneman,et al. Accumulation of arachidonic acid in ischemic/reperfused cardiac tissue: possible causes and consequences. , 1997, Prostaglandins, leukotrienes, and essential fatty acids.
[118] Tsuneya Ikezu,et al. Identification of Peptide and Protein Ligands for the Caveolin-scaffolding Domain , 1997, The Journal of Biological Chemistry.
[119] D. Mochly‐Rosen,et al. A Protein Kinase C Translocation Inhibitor as an Isozyme-selective Antagonist of Cardiac Function* , 1996, The Journal of Biological Chemistry.
[120] K. Garlid. Cation transport in mitochondria--the potassium cycle. , 1996, Biochimica et biophysica acta.
[121] V. Yarov-Yarovoy,et al. The Mitochondrial K Channel as a Receptor for Potassium Channel Openers (*) , 1996, The Journal of Biological Chemistry.
[122] J. Downey,et al. Pretreatment with endothelin-1 mimics ischemic preconditioning against infarction in isolated rabbit heart. , 1996, Journal of molecular and cellular cardiology.
[123] G. Grover,et al. Glyburide-reversible cardioprotective effects of calcium-independent phospholipase A2 inhibition in ischemic rat hearts. , 1996, Cardiovascular research.
[124] J. Downey,et al. Myocardial preconditioning promises to be a novel approach to the treatment of ischemic heart disease. , 1996, Annual review of medicine.
[125] M. Lisanti,et al. Evidence for a Regulated Interaction between Heterotrimeric G Proteins and Caveolin , 1995, The Journal of Biological Chemistry.
[126] J. Downey,et al. Pretreatment with angiotensin II activates protein kinase C and limits myocardial infarction in isolated rabbit hearts. , 1995, Journal of molecular and cellular cardiology.
[127] D. Mochly‐Rosen,et al. An autoregulatory region in protein kinase C: the pseudoanchoring site. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[128] J. Downey,et al. Signal transduction in ischemic preconditioning. , 1995, Zeitschrift fur Kardiologie.
[129] M. Lisanti,et al. Caveolae, caveolin and caveolin-rich membrane domains: a signalling hypothesis. , 1994, Trends in cell biology.
[130] M. Barbacid,et al. Zinc finger domains and phorbol ester pharmacophore. Analysis of binding to mutated form of protein kinase C zeta and the vav and c-raf proto-oncogene products. , 1994, The Journal of biological chemistry.
[131] B. Soifer,et al. Cardioprotection provided by adenosine receptor activation is abolished by blockade of the KATP channel. , 1994, The American journal of physiology.
[132] C. Cairns,et al. Preconditioning against myocardial dysfunction after ischemia and reperfusion by an alpha 1-adrenergic mechanism. , 1993, Circulation research.
[133] A. Beavis,et al. Evidence for the allosteric regulation of the mitochondrial K+/H+ antiporter by matrix protons. , 1990, The Journal of biological chemistry.
[134] G. Grover,et al. Anti-ischemic effects of the potassium channel activators pinacidil and cromakalim and the reversal of these effects with the potassium channel blocker glyburide. , 1989, The Journal of pharmacology and experimental therapeutics.
[135] F. Prinzen,et al. Uptake and Tissue Content of Fatty Acids in Dog Myocardium under Normoxic and Ischemic Conditions , 1982, Circulation research.
[136] R. Jennings,et al. Mitochondrial Structure and Function in Acute Myocardial Ischemic Injury , 1976, Circulation research.