Conditional ablation of macrophages halts progression of crescentic glomerulonephritis.
暂无分享,去创建一个
J. Duffield | P. Tipping | T. Kipari | J. Cailhier | Spike Clay | R. Lang | J. Bonventre | J. Hughes | S. Clay
[1] J. Cailhier,et al. Conditional Macrophage Ablation Demonstrates That Resident Macrophages Initiate Acute Peritoneal Inflammation1 , 2005, The Journal of Immunology.
[2] S. Forbes,et al. Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair. , 2005, The Journal of clinical investigation.
[3] K. Meldrum,et al. Unilateral ureteral obstruction induces renal tubular cell production of tumor necrosis factor-alpha independent of inflammatory cell infiltration. , 2004, The Journal of urology.
[4] J. Iredale,et al. Engagement of αvβ3 Integrin Regulates Proliferation and Apoptosis of Hepatic Stellate Cells* , 2004, Journal of Biological Chemistry.
[5] C. Alpers,et al. Late onset of treatment with a chemokine receptor CCR1 antagonist prevents progression of lupus nephritis in MRL-Fas(lpr) mice. , 2004, Journal of the American Society of Nephrology : JASN.
[6] C. Haslett,et al. Eat Me or Die , 2003, Science.
[7] R. Atkins,et al. Macrophages act as effectors of tissue damage in acute renal allograft rejection , 2003, Transplantation.
[8] P. Bruneval,et al. Glomerular epithelial-mesenchymal transdifferentiation in pauci-immune crescentic glomerulonephritis. , 2003, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[9] S. Mckercher,et al. Wound Healing in the PU.1 Null Mouse—Tissue Repair Is Not Dependent on Inflammatory Cells , 2003, Current Biology.
[10] H. Groux,et al. The complex role of interleukin-10 in autoimmunity. , 2003, Journal of autoimmunity.
[11] N. Vaziri,et al. Microvascular and tubulointerstitial injury associated with chronic hypoxia-induced hypertension. , 2003, Kidney international.
[12] V. Koteliansky,et al. Monocytes may promote myofibroblast accumulation and apoptosis in Alport renal fibrosis. , 2003, Kidney international.
[13] S. Fulda,et al. Macrophages induce apoptosis in proximal tubule cells , 2003, Pediatric Nephrology.
[14] V. Kelley,et al. Reduced Macrophage Recruitment, Proliferation, and Activation in Colony-Stimulating Factor-1-Deficient Mice Results in Decreased Tubular Apoptosis During Renal Inflammation 1 , 2003, The Journal of Immunology.
[15] I. Fishbein,et al. Macrophage Depletion by Clodronate-Containing Liposomes Reduces Neointimal Formation After Balloon Injury in Rats and Rabbits , 2002, Circulation.
[16] P. Tipping,et al. IFN-γ Production by Intrinsic Renal Cells and Bone Marrow-Derived Cells Is Required for Full Expression of Crescentic Glomerulonephritis in Mice1 , 2002, The Journal of Immunology.
[17] John P. Iredale,et al. Inhibition of Apoptosis of Activated Hepatic Stellate Cells by Tissue Inhibitor of Metalloproteinase-1 Is Mediated via Effects on Matrix Metalloproteinase Inhibition , 2002, The Journal of Biological Chemistry.
[18] B. Thornhill,et al. Selectins mediate macrophage infiltration in obstructive nephropathy in newborn mice. , 2002, Kidney international.
[19] K. Park,et al. Prevention of Kidney Ischemia/Reperfusion-induced Functional Injury, MAPK and MAPK Kinase Activation, and Inflammation by Remote Transient Ureteral Obstruction* , 2002, The Journal of Biological Chemistry.
[20] Carl F. Ware,et al. Suppression by apoptotic cells defines tumor necrosis factor-mediated induction of glomerular mesangial cell apoptosis by activated macrophages. , 2001, The American journal of pathology.
[21] R. Schnabel,et al. Engulfment genes cooperate with ced-3 to promote cell death in Caenorhabditis elegans , 2001, Nature.
[22] H. Horvitz,et al. Phagocytosis promotes programmed cell death in C. elegans , 2001, Nature.
[23] H. Brühl,et al. Expression and Characterization of the Chemokine Receptors CCR2 and CCR5 in Mice1 , 2001, The Journal of Immunology.
[24] P. Rémy,et al. A new morphologic index for the evaluation of renal biopsies in lupus nephritis. , 2000, Kidney international.
[25] B. Antus,et al. Influence of alternatively and classically activated macrophages on fibrogenic activities of human fibroblasts. , 2000, Cellular immunology.
[26] K. Matsushima,et al. Distinct Expression of CCR1 and CCR5 in Glomerular and Interstitial Lesions of Human Glomerular Diseases , 2000, American Journal of Nephrology.
[27] M. Kaplan,et al. Cutting Edge: Differential Expression of Chemokines in Th1 and Th2 Cells Is Dependent on Stat6 But Not Stat41 , 2000, The Journal of Immunology.
[28] J. Duffield,et al. Activated Macrophages Direct Apoptosis and Suppress Mitosis of Mesangial Cells1 , 2000, The Journal of Immunology.
[29] B. Rollins,et al. Monocyte Chemoattractant Protein 1–Dependent Leukocytic Infiltrates Are Responsible for Autoimmune Disease in Mrl-Faslpr Mice , 1999, The Journal of experimental medicine.
[30] V. Fadok,et al. Transcriptional and translational regulation of inflammatory mediator production by endogenous TGF-beta in macrophages that have ingested apoptotic cells. , 1999, Journal of immunology.
[31] D. Salant,et al. Lack of chemokine receptor CCR1 enhances Th1 responses and glomerular injury during nephrotoxic nephritis. , 1999, The Journal of clinical investigation.
[32] C. Alpers,et al. Obstructive uropathy in the mouse: role of osteopontin in interstitial fibrosis and apoptosis. , 1999, Kidney international.
[33] R. Lang,et al. Macrophages kill capillary cells in G1 phase of the cell cycle during programmed vascular regression. , 1999, Development.
[34] S. Goerdt,et al. Other functions, other genes: alternative activation of antigen-presenting cells. , 1999, Immunity.
[35] R. Atkins,et al. Local macrophage proliferation in human glomerulonephritis. , 1998, Kidney international.
[36] P. Tipping,et al. Crescentic glomerulonephritis in CD4- and CD8-deficient mice. Requirement for CD4 but not CD8 cells. , 1998, The American journal of pathology.
[37] M. Kitamura,et al. TGF-beta1 as an endogenous defender against macrophage-triggered stromelysin gene expression in the glomerulus. , 1998, Journal of immunology.
[38] R. Lang,et al. Macrophages induce apoptosis in normal cells in vivo. , 1997, Development.
[39] P. Tipping,et al. Mechanisms of T cell‐induced glomerular injury in anti‐glomeruler basement membrane (GBM) glomerulonephritis in rats , 1997, Clinical and experimental immunology.
[40] J. Schalkwijk,et al. Decreased PMN accumulation and glomerular damage by clodronate liposome treatment in PMN-dependent anti-GBM nephritis in mice. , 1997, Experimental nephrology.
[41] C. McClain,et al. Kupffer cell depletion by gadolinium chloride enhances liver regeneration after partial hepatectomy in rats. , 1996, The American journal of physiology.
[42] R. Atkins,et al. Local macrophage proliferation in the progression of glomerular and tubulointerstitial injury in rat anti-GBM glomerulonephritis. , 1995, Kidney international.
[43] R. Lang,et al. Apoptosis during macrophage-dependent ocular tissue remodelling. , 1994, Development.
[44] J. Hughes,et al. Mesangial cell apoptosis: the major mechanism for resolution of glomerular hypercellularity in experimental mesangial proliferative nephritis. , 1994, The Journal of clinical investigation.
[45] G. Ding,et al. Glomerular macrophages and the mesangial proliferative response in the experimental nephrotic syndrome. , 1992, The American journal of pathology.
[46] J. Diamond,et al. Sublethal X-irradiation during acute puromycin nephrosis prevents late renal injury: role of macrophages. , 1991, The American journal of physiology.
[47] A. Gown,et al. Localization of PDGF-B protein in macrophages in all phases of atherogenesis. , 1990, Science.
[48] P. Tipping,et al. Macrophage-induced glomerular fibrin deposition in experimental glomerulonephritis in the rabbit. , 1985, The Journal of clinical investigation.
[49] J. Klippel,et al. Prognostic factors in lupus nephritis. Contribution of renal histologic data. , 1983, The American journal of medicine.
[50] J. Duffield,et al. Quantitative morphometry of lupus nephritis: the significance of collagen, tubular space, and inflammatory infiltrate. , 2005, Kidney international.
[51] R. Atkins,et al. Heterogeneity of antigen expression explains controversy over glomerular macrophage accumulation in mouse glomerulonephritis. , 2003, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[52] J. Duffield. The inflammatory macrophage: a story of Jekyll and Hyde. , 2003, Clinical science.
[53] R. Atkins,et al. Adoptive transfer studies demonstrate that macrophages can induce proteinuria and mesangial cell proliferation. , 2003, Kidney international.
[54] C. Haslett,et al. Cell biology. Eat me or die. , 2003, Science.
[55] N. Franc. Phagocytosis of apoptotic cells in mammals, caenorhabditis elegans and Drosophila melanogaster: molecular mechanisms and physiological consequences. , 2002, Frontiers in bioscience : a journal and virtual library.
[56] P. O'Connor. Mammalian G1 and G2 phase checkpoints. , 1997, Cancer surveys.
[57] O'Connor Pm. Mammalian G1 and G2 phase checkpoints. , 1997 .
[58] N. Van Rooijen. The liposome-mediated macrophage 'suicide' technique. , 1989, Journal of immunological methods.
[59] R. Atkins,et al. Cellular composition of crescents in human rapidly progressive glomerulonephritis identified using monoclonal antibodies. , 1984, American journal of nephrology.
[60] R. Ross,et al. The role of the macrophage in wound repair. A study with hydrocortisone and antimacrophage serum. , 1975, The American journal of pathology.
[61] J. Mitchison. Cell Biology , 1964, Nature.