C4d Deposits on the Surface of RBCs in Trauma Patients and Interferes With Their Function*
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S. Shevkoplyas | N. Shapiro | G. Tsokos | M. Tsokos | I. Ghiran | Z. Paz | J. D. Dalle Lucca | L. Kannan | T. Muroya
[1] D. Rittirsch,et al. Role of Complement in Multiorgan Failure , 2012, Clinical & developmental immunology.
[2] Jiaquan Xu,et al. Deaths: preliminary data for 2011. , 2012 .
[3] P. Marik,et al. The immune response to surgery and trauma: Implications for treatment , 2012, The journal of trauma and acute care surgery.
[4] G. Wanner,et al. Danger Signals Activating the Immune Response after Trauma , 2012, Mediators of inflammation.
[5] M. Huber-Lang,et al. Molecular mechanisms of inflammation and tissue injury after major trauma-is complement the "bad guy"? , 2011, Journal of Biomedical Science.
[6] Sergey S Shevkoplyas,et al. Systemic lupus erythematosus serum deposits C4d on red blood cells, decreases red blood cell membrane deformability, and promotes nitric oxide production. , 2011, Arthritis and rheumatism.
[7] S. Shevkoplyas,et al. Ligation of complement receptor 1 increases erythrocyte membrane deformability. , 2010, Blood.
[8] Subra Suresh,et al. Shape and Biomechanical Characteristics of Human Red Blood Cells in Health and Disease , 2010, MRS bulletin.
[9] J. Ahearn,et al. Erythrocyte C3d and C4d for monitoring disease activity in systemic lupus erythematosus. , 2010, Arthritis and rheumatism.
[10] P. Gál,et al. Calcium-dependent Conformational Flexibility of a CUB Domain Controls Activation of the Complement Serine Protease C1r* , 2010, The Journal of Biological Chemistry.
[11] J. Asara,et al. C3b deposition on human erythrocytes induces the formation of a membrane skeleton-linked protein complex. , 2009, The Journal of clinical investigation.
[12] E. Deitch,et al. Trauma-hemorrhagic shock-induced red blood cell damage leads to decreased microcirculatory blood flow* , 2009, Critical care medicine.
[13] John D Lambris,et al. Complement component C5a mediates hemorrhage-induced intestinal damage. , 2008, The Journal of surgical research.
[14] N. Mohandas,et al. Red cell membrane: past, present, and future. , 2008, Blood.
[15] M. Cohen,et al. ROLE OF THE ALTERNATIVE PATHWAY IN THE EARLY COMPLEMENT ACTIVATION FOLLOWING MAJOR TRAUMA , 2007, Shock.
[16] E. Deitch,et al. Augmented erythrocyte band-3 phosphorylation in septic mice. , 2007, Biochimica et biophysica acta.
[17] Thomas Lauer,et al. Red blood cells express a functional endothelial nitric oxide synthase. , 2006, Blood.
[18] S. Krishnaswami,et al. Measurement of erythrocyte C4d and complement receptor 1 in systemic lupus erythematosus. , 2004, Arthritis and rheumatism.
[19] S. Zaets,et al. Alterations of red blood cell shape in patients with severe trauma. , 2004, The Journal of trauma.
[20] W. Baldwin,et al. Beyond C4d: Other Complement‐Related Diagnostic Approaches to Antibody‐Mediated Rejection , 2004, American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons.
[21] O. Götze,et al. Complement Activation During Hemorrhagic Shock and Resuscitation in Swine , 2003, Shock.
[22] Rosalinda B Wenby,et al. Effects of Nitric Oxide on Red Blood Cell Deformability Blood Sampling , 2022 .
[23] Alexander Barbul,et al. Ca2+ promotes erythrocyte band 3 tyrosine phosphorylation via dissociation of phosphotyrosine phosphatase from band 3. , 2002, The Biochemical journal.
[24] E. Fosse,et al. Complement activation in injured patients occurs immediately and is dependent on the severity of the trauma. , 1998, Injury.
[25] A. Brovelli,et al. Tyrosine phosphorylation of band 3 protein in Ca2+/A23187-treated human erythrocytes. , 1996, The Biochemical journal.
[26] N. Mohandas,et al. Modulation of Erythrocyte Membrane Mechanical Function by β-Spectrin Phosphorylation and Dephosphorylation (*) , 1995, The Journal of Biological Chemistry.
[27] Dl Hoyert,et al. National Vital Statistics Reports NCHS.pdf , 2012 .
[28] O. Baskurt,et al. Nitric oxide, erythrocytes and exercise. , 2011, Clinical hemorheology and microcirculation.
[29] O. Baskurt,et al. Nitric oxide generation in red blood cells induced by mechanical stress. , 2010, Clinical hemorheology and microcirculation.