Iodoacetic acid exposure alters the transcriptome in mouse ovarian antral follicles.
暂无分享,去创建一个
J. Flaws | M. Plewa | Alison M. Neff | M. Laws | A. Gonsioroski | J. Drnevich | Vasiliki E. Mourikes
[1] Q. Guo,et al. The RNA-Binding Motif Protein Family in Cancer: Friend or Foe? , 2021, Frontiers in Oncology.
[2] S. Richardson,et al. Are Disinfection Byproducts (DBPs) Formed in My Cup of Tea? Regulated, Priority, and Unknown DBPs. , 2021, Environmental science & technology.
[3] J. Flaws,et al. Iodoacetic acid, a water disinfection byproduct, disrupts hypothalamic and pituitary reproductive regulatory factors and induces toxicity in the female pituitary. , 2021, Toxicological sciences : an official journal of the Society of Toxicology.
[4] Gang Liu,et al. RNA-Binding Motif Protein 11 (RBM11) Serves as a Prognostic Biomarker and Promotes Ovarian Cancer Progression , 2021, Disease markers.
[5] J. Flaws,et al. Iodoacetic acid affects estrous cyclicity, ovarian gene expression, and hormone levels in mice , 2021, Biology of Reproduction.
[6] J. Qiao,et al. RBM14 Modulates Tubulin Acetylation and Regulates Spindle Morphology During Meiotic Maturation in Mouse Oocytes , 2021, Frontiers in Cell and Developmental Biology.
[7] Yawei Wang,et al. Occurrence and Distribution of Disinfection Byproducts in Domestic Wastewater Effluent, Tap Water, and Surface Water during the SARS-CoV-2 Pandemic in China , 2021, Environmental science & technology.
[8] B. G.,et al. Meta-Analysis of Small for Gestational Age Births and Disinfection Byproduct Exposures. , 2020, Environmental research.
[9] J. Flaws,et al. Iodoacetic acid disrupts mouse oocyte maturation by inducing oxidative stress and spindle abnormalities , 2020, bioRxiv.
[10] Naveen Patel,et al. Disinfection by-products in drinking water: Occurrence, toxicity and abatement. , 2020, Environmental pollution.
[11] G. Amy,et al. Chloramination of iodide-containing waters: Formation of iodinated disinfection byproducts and toxicity correlation with total organic halides of treated waters. , 2019, The Science of the total environment.
[12] J. Flaws,et al. Iodoacetic Acid Inhibits Follicle Growth and Alters Expression of Genes that Regulate Apoptosis, the Cell Cycle, Estrogen Receptors, and Ovarian Steroidogenesis in Mouse Ovarian Follicles. , 2019, Reproductive toxicology.
[13] G. Amy,et al. Formation of iodinated trihalomethanes and noniodinated disinfection byproducts during chloramination of algal organic matter extracted from Microcystis aeruginosa. , 2019, Water research.
[14] M. Felipe-Sotelo,et al. Disinfection byproducts potentially responsible for the association between chlorinated drinking water and bladder cancer: A review. , 2019, Water research.
[15] K. Robien,et al. Ingested nitrate, disinfection by-products, and risk of colon and rectal cancers in the Iowa Women's Health Study cohort. , 2019, Environment international.
[16] Alireza Hadj Khodabakhshi,et al. Metascape provides a biologist-oriented resource for the analysis of systems-level datasets , 2019, Nature Communications.
[17] S. Richardson,et al. Formation of Iodinated Disinfection Byproducts (I-DBPs) in Drinking Water: Emerging Concerns and Current Issues. , 2019, Accounts of chemical research.
[18] J. Wright,et al. Exposure to disinfectant by-products and the risk of stillbirth in Massachusetts , 2018, Occupational and Environmental Medicine.
[19] Rutao Liu,et al. Drinking water disinfection byproduct iodoacetic acid interacts with catalase and induces cytotoxicity in mouse primary hepatocytes. , 2018, Chemosphere.
[20] Yunfeng Zou,et al. Iodoacetic Acid Disrupting the Thyroid Endocrine System in Vitro and in Vivo. , 2018, Environmental science & technology.
[21] P. Kaufman,et al. Ki-67: more than a proliferation marker , 2018, Chromosoma.
[22] David F Wilson. Oxidative phosphorylation: regulation and role in cellular and tissue metabolism , 2017, The Journal of physiology.
[23] T. Ternes,et al. The impact of iodinated X-ray contrast agents on formation and toxicity of disinfection by-products in drinking water. , 2017, Journal of environmental sciences.
[24] M. Plewa,et al. CHO cell cytotoxicity and genotoxicity analyses of disinfection by-products: An updated review. , 2017, Journal of environmental sciences.
[25] Li Li,et al. Role of the PI3K-Akt Signaling Pathway in the Pathogenesis of Polycystic Ovary Syndrome. , 2017 .
[26] C. Lobe,et al. The role of Notch signalling in ovarian angiogenesis , 2017, Journal of Ovarian Research.
[27] Rob Patro,et al. Salmon provides fast and bias-aware quantification of transcript expression , 2017, Nature Methods.
[28] J. Flaws,et al. Effects of an Environmentally Relevant Phthalate Mixture on Cultured Mouse Antral Follicles , 2016, Toxicological sciences : an official journal of the Society of Toxicology.
[29] J. Wright,et al. Disinfection By-Product Exposures and the Risk of Specific Cardiac Birth Defects , 2016, Environmental health perspectives.
[30] J. Flaws,et al. Monohaloacetic acid drinking water disinfection by-products inhibit follicle growth and steroidogenesis in mouse ovarian antral follicles in vitro. , 2016, Reproductive toxicology.
[31] Aaron T. L. Lun,et al. From reads to genes to pathways: differential expression analysis of RNA-Seq experiments using Rsubread and the edgeR quasi-likelihood pipeline , 2016, F1000Research.
[32] Måns Magnusson,et al. MultiQC: summarize analysis results for multiple tools and samples in a single report , 2016, Bioinform..
[33] J. Dupont,et al. Insulin signalling and glucose transport in the ovary and ovarian function during the ovarian cycle , 2016, The Biochemical journal.
[34] Terence P. Speed,et al. How data analysis affects power, reproducibility and biological insight of RNA-seq studies in complex datasets , 2015, Nucleic acids research.
[35] Rupesh K. Gupta,et al. Di(2-ethylhexyl) phthalate inhibits antral follicle growth, induces atresia, and inhibits steroid hormone production in cultured mouse antral follicles. , 2015, Toxicology and applied pharmacology.
[36] Abbas Ali Mahdi,et al. PI3K/PTEN/Akt and TSC/mTOR signaling pathways, ovarian dysfunction, and infertility: an update. , 2014, Journal of molecular endocrinology.
[37] A. Drewnowski,et al. Water and beverage consumption among adults in the United States: cross-sectional study using data from NHANES 2005–2010 , 2013, BMC Public Health.
[38] M. Xia,et al. Human cell toxicogenomic analysis linking reactive oxygen species to the toxicity of monohaloacetic acid drinking water disinfection byproducts. , 2013, Environmental science & technology.
[39] M. Plewa,et al. Pyruvate remediation of cell stress and genotoxicity induced by haloacetic acid drinking water disinfection by‐products , 2013, Environmental and molecular mutagenesis.
[40] E. S. Hunter,et al. Comprehensive assessment of a chlorinated drinking water concentrate in a rat multigenerational reproductive toxicity study. , 2013, Environmental science & technology.
[41] Gengsheng He,et al. Drinking water disinfection byproduct iodoacetic acid induces tumorigenic transformation of NIH3T3 cells. , 2013, Environmental science & technology.
[42] Laura N. Vandenberg,et al. Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses. , 2012, Endocrine reviews.
[43] Davis J. McCarthy,et al. Differential expression analysis of multifactor RNA-Seq experiments with respect to biological variation , 2012, Nucleic acids research.
[44] M. Conti,et al. Natriuretic Peptide Signaling Regulates Female Fertility at the Level of the Ovary. , 2011 .
[45] M. Plewa,et al. Biological mechanism for the toxicity of haloacetic acid drinking water disinfection byproducts. , 2011, Environmental science & technology.
[46] J. E. Simmons,et al. Mammalian cell cytotoxicity and genotoxicity of the haloacetic acids, a major class of drinking water disinfection by‐products , 2010, Environmental and molecular mutagenesis.
[47] M. Attene-Ramos,et al. Comparative human cell toxicogenomic analysis of monohaloacetic acid drinking water disinfection byproducts. , 2010, Environmental science & technology.
[48] M. Robinson,et al. A scaling normalization method for differential expression analysis of RNA-seq data , 2010, Genome Biology.
[49] Rupesh K. Gupta,et al. Di-(2-ethylhexyl) phthalate and mono-(2-ethylhexyl) phthalate inhibit growth and reduce estradiol levels of antral follicles in vitro. , 2010, Toxicology and applied pharmacology.
[50] Kui Liu,et al. Molecular mechanisms underlying the activation of mammalian primordial follicles. , 2009, Endocrine reviews.
[51] Stephen M. Hedrick,et al. MAPK3/1 (ERK1/2) in Ovarian Granulosa Cells Are Essential for Female Fertility , 2009, Science.
[52] Brad T. Sherman,et al. Bioinformatics enrichment tools: paths toward the comprehensive functional analysis of large gene lists , 2008, Nucleic acids research.
[53] John J. Evans,et al. Occurrence and mammalian cell toxicity of iodinated disinfection byproducts in drinking water. , 2008, Environmental science & technology.
[54] How-Ran Guo,et al. Water disinfection by-products and the risk of specific birth defects: a population-based cross-sectional study in Taiwan , 2008, Environmental health : a global access science source.
[55] D. DeMarini,et al. Occurrence, genotoxicity, and carcinogenicity of regulated and emerging disinfection by-products in drinking water: a review and roadmap for research. , 2007, Mutation research.
[56] P. Rogerson,et al. Case-Control Study of the Effects of Trihalomethanes on Urinary Bladder Cancer Risk , 2007, Archives of environmental & occupational health.
[57] R. Melnick,et al. Toxicity and carcinogenicity of the water disinfection byproduct, dibromoacetic acid, in rats and mice. , 2007, Toxicology.
[58] E. Cemeli,et al. Modulation of the cytotoxicity and genotoxicity of the drinking water disinfection byproduct lodoacetic acid by suppressors of oxidative stress. , 2006, Environmental science & technology.
[59] Yin-Tak Woo,et al. Chemical and biological characterization of newly discovered iodoacid drinking water disinfection byproducts. , 2004, Environmental science & technology.
[60] D. Veeramachaneni,et al. Chronic exposure to dibromoacetic acid, a water disinfection byproduct, diminishes primordial follicle populations in the rabbit. , 2004, Toxicological sciences : an official journal of the Society of Toxicology.
[61] M. Kogevinas,et al. Disinfection Byproducts and Bladder Cancer: A Pooled Analysis , 2004, Epidemiology.
[62] John C Lipscomb,et al. A Feasibility Study of Cumulative Risk Assessment Methods for Drinking Water Disinfection By-Product Mixtures , 2004, Journal of toxicology and environmental health. Part A.
[63] R. Calderon,et al. The epidemiology of chemical contaminants of drinking water. , 2000, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[64] P. Monget,et al. Folliculogenesis , 2019, Encyclopedia of Endocrine Diseases.
[65] U. Luderer. Ovarian toxicity from reactive oxygen species. , 2014, Vitamins and hormones.
[66] Laura N. Vandenberg,et al. Low-dose effects of hormones and endocrine disruptors. , 2014, Vitamins and hormones.
[67] S. Richardson,et al. Drinking Water Disinfection By-products , 2011 .
[68] E. S. Hunter,et al. Pregnancy loss and eye malformations in offspring of F344 rats following gestational exposure to mixtures of regulated trihalomethanes and haloacetic acids. , 2011, Reproductive toxicology.
[69] Brad T. Sherman,et al. Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources , 2008, Nature Protocols.
[70] A. Voss,et al. Ovarian Oxytocin: Periovulatory Production and Effects , 2000 .
[71] Y. Benjamini,et al. Controlling the false discovery rate: a practical and powerful approach to multiple testing , 1995 .