P43/pro-EMAPII: a potential biomarker for discriminating traumatic versus ischemic brain injury.
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F. Tortella | R. Hayes | J. Dave | Kevin K. W. Wang | A. Ottens | X. Lu | M. Liu | C. Yao | A. Williams
[1] F. Tortella,et al. Detection of protein biomarkers using high-throughput immunoblotting following focal ischemic or penetrating ballistic-like brain injuries in rats , 2008, Brain injury.
[2] F. Tortella,et al. Acute and delayed neuroinflammatory response following experimental penetrating ballistic brain injury in the rat , 2007, Journal of Neuroinflammation.
[3] H. Schluesener,et al. FTY720 attenuates accumulation of EMAP-II+ and MHC-II+ monocytes in early lesions of rat traumatic brain injury , 2007, Journal of cellular and molecular medicine.
[4] F. Tortella,et al. Penetrating ballistic-like brain injury in the rat: differential time courses of hemorrhage, cell death, inflammation, and remote degeneration. , 2006, Journal of neurotrauma.
[5] G. Ling,et al. Severity level and injury track determine outcome following a penetrating ballistic-like brain injury in the rat , 2006, Neuroscience Letters.
[6] A. Eggermont,et al. Endothelial monocyte-activating polypeptide-II and its functions in (patho)physiological processes. , 2006, Cytokine & growth factor reviews.
[7] M. Schwarz,et al. Identification of protease-sensitive sites in Human Endothelial-Monocyte Activating Polypeptide II protein. , 2006, Experimental cell research.
[8] James G. Tobin,et al. Pu Workshop Letter , 2006 .
[9] F. Tortella,et al. Down-regulation of the sodium channel Na(v)1.1 alpha-subunit following focal ischemic brain injury in rats: in situ hybridization and immunohistochemical analysis. , 2005, Life sciences.
[10] F. Tortella,et al. Characterization of a new rat model of penetrating ballistic brain injury. , 2005, Journal of neurotrauma.
[11] Sunghoon Kim,et al. The novel cytokine p43 stimulates dermal fibroblast proliferation and wound repair. , 2005, The American journal of pathology.
[12] E. Chavakis,et al. Cathepsin L is required for endothelial progenitor cell–induced neovascularization , 2005, Nature Medicine.
[13] P. Symonds,et al. Colorectal Cancer Cells Induce Lymphocyte Apoptosis by an Endothelial Monocyte-Activating Polypeptide-II-Dependent Mechanism 1 , 2004, The Journal of Immunology.
[14] J. Schwab,et al. Spinal cord injury induces lesional expression of the proinflammatory and antiangiogenic cytokine EMAP II. , 2003, Journal of neurotrauma.
[15] J. Richt,et al. Accumulation of the proinflammatory cytokine endothelial-monocyte-activating polypeptide II in ramified microglial cells in brains of Borna virus infected Lewis rats , 2003, Neuroscience Letters.
[16] J. Schwab,et al. Lesional expression of a proinflammatory and antiangiogenic cytokine EMAP II confined to endothelium and microglia/macrophages during secondary damage following experimental traumatic brain injury , 2003, Journal of Neuroimmunology.
[17] D. Chuang,et al. Regulation of c‐Jun N‐terminal kinase, p38 kinase and AP‐1 DNA binding in cultured brain neurons: roles in glutamate excitotoxicity and lithium neuroprotection , 2003, Journal of neurochemistry.
[18] Sunghoon Kim,et al. Dose-dependent Biphasic Activity of tRNA Synthetase-associating Factor, p43, in Angiogenesis* , 2002, The Journal of Biological Chemistry.
[19] F. Michetti,et al. Expression of EMAP-II by Activated Monocytes/Microglial Cells in Different Regions of the Rat Hippocampus after Trimethyltin-Induced Brain Damage , 2002, Experimental Neurology.
[20] Doo Yeon Kim,et al. Cellular and molecular pathways of ischemic neuronal death. , 2002, Journal of biochemistry and molecular biology.
[21] Sunghoon Kim,et al. A Cofactor of tRNA Synthetase, p43, Is Secreted to Up-regulate Proinflammatory Genes* , 2001, The Journal of Biological Chemistry.
[22] X. C. Lu,et al. Neuroprotection (focal ischemia) and neurotoxicity (electroencephalographic) studies in rats with AHN649, a 3-amino analog of dextromethorphan and low-affinity N-methyl-D-aspartate antagonist. , 1999, The Journal of pharmacology and experimental therapeutics.
[23] W. Stetler-Stevenson,et al. Matrix metalloproteinases in angiogenesis: a moving target for therapeutic intervention. , 1999, The Journal of clinical investigation.
[24] W. Risau,et al. Regulation of endothelial monocyte-activating polypeptide II release by apoptosis. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[25] M. Mirande,et al. The p43 Component of the Mammalian Multi-synthetase Complex Is Likely To Be the Precursor of the Endothelial Monocyte-activating Polypeptide II Cytokine* , 1997, The Journal of Biological Chemistry.
[26] H. Schluesener,et al. Localization of endothelial‐monocyte‐activating polypeptide II (EMAP II), a novel proinflammatory cytokine, to lesions of experimental autoimmune encephalomyelitis, neuritis and uveitis: Expression by monocytes and activated microglial cells , 1997, Glia.
[27] R. J. Mullen,et al. NeuN, a neuronal specific nuclear protein in vertebrates. , 1992, Development.
[28] J. Schaper,et al. Regulation of EMAP II by hypoxia. , 2003, The American journal of pathology.
[29] G. Pasinetti,et al. From cDNA microarrays to high-throughput proteomics. Implications in the search for preventive initiatives to slow the clinical progression of Alzheimer's disease dementia. , 2001, Restorative neurology and neuroscience.
[30] R. De Simone,et al. Journal of Neuroinflammation Activation of Α7 Nicotinic Acetylcholine Receptor by Nicotine Selectively Up-regulates Cyclooxygenase-2 and Prostaglandin E 2 in Rat Microglial Cultures Brain Macrophagesinflammationtnfil-10prostaglandin E 2 , 2022 .