Defective Metabolic Signaling in Adenylate Kinase AK1 Gene Knock-out Hearts Compromises Post-ischemic Coronary Reflow*
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Andre Terzic | Darko Pucar | A. Terzic | D. Pucar | B. Wieringa | P. Dzeja | P. Bast | Peter Bast | Petras P Dzeja | Be Wieringa
[1] A. Lajtha,et al. 6.3 Mitochondria-Nucleus Energetic Communication: Role for Phosphotransfer Networks in Processing Cellular Information , 2007 .
[2] B. Viollet,et al. Activation of AMP kinase α1 subunit induces aortic vasorelaxation in mice , 2007 .
[3] M. Rezaee,et al. Impaired perfusion after myocardial infarction is due to reperfusion-induced deltaPKC-mediated myocardial damage. , 2007, Cardiovascular research.
[4] Abel Lajtha,et al. Handbook of Neurochemistry and Molecular Neurobiology , 2007 .
[5] Joseph F. Clark,et al. Creatine Kinase Activity Is Associated With Blood Pressure , 2006, Circulation.
[6] Susumu Seino,et al. Gene knockout of the KCNJ8‐encoded Kir6.1 KATP channel imparts fatal susceptibility to endotoxemia , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[7] T. Garland,et al. AMP-Activated Protein Kinase Is Involved in Endothelial NO Synthase Activation in Response to Shear Stress , 2006, Arteriosclerosis, thrombosis, and vascular biology.
[8] A. Terzic,et al. Cardiac system bioenergetics: metabolic basis of the Frank‐Starling law , 2006, The Journal of physiology.
[9] R. Terjung,et al. Contraction-mediated phosphorylation of AMPK is lower in skeletal muscle of adenylate kinase-deficient mice. , 2006, Journal of applied physiology.
[10] J. Makielski,et al. Downloaded from http://circres.ahajournals.org / by guest on February 24, 2013Spontaneous Coronary Vasospasm in K ATP Mutant Mice Arises From a Smooth Muscle–Extrinsic Process , 2006 .
[11] C. Wyatt,et al. Does AMP-activated Protein Kinase Couple Inhibition of Mitochondrial Oxidative Phosphorylation by Hypoxia to Calcium Signaling in O2-sensing Cells?* , 2005, Journal of Biological Chemistry.
[12] P. Guns,et al. Pharmacological characterization of nucleotide P2Y receptors on endothelial cells of the mouse aorta , 2005, British journal of pharmacology.
[13] R. Terjung,et al. Skeletal muscle contractile performance and ADP accumulation in adenylate kinase-deficient mice. , 2005, American journal of physiology. Cell physiology.
[14] C. C. Hale,et al. Metabolic activation of AMP kinase in vascular smooth muscle. , 2005, Journal of applied physiology.
[15] D. Hardie. The AMP-activated protein kinase pathway – new players upstream and downstream , 2004, Journal of Cell Science.
[16] J. Ingwall. Transgenesis and cardiac energetics: new insights into cardiac metabolism. , 2004, Journal of molecular and cellular cardiology.
[17] E. Feigl,et al. Matching coronary blood flow to myocardial oxygen consumption. , 2004, Journal of applied physiology.
[18] T. Wallimann,et al. Compartmentation of ATP synthesis and utilization in smooth muscle: roles of aerobic glycolysis and creatine kinase , 1994, Molecular and Cellular Biochemistry.
[19] Jan W. P. Kuiper,et al. Two structurally distinct and spatially compartmentalized adenylate kinases are expressed from the AK1 gene in mouse brain , 2004, Molecular and Cellular Biochemistry.
[20] Andre Terzic,et al. Phosphotransfer dynamics in skeletal muscle from creatine kinase gene-deleted mice , 2004, Molecular and Cellular Biochemistry.
[21] Carmen Drahl,et al. Mapping hypoxia-induced bioenergetic rearrangements and metabolic signaling by 18O-assisted 31P NMR and 1H NMR spectroscopy , 2004, Molecular and Cellular Biochemistry.
[22] A. Terzic,et al. Nucleotide-gated KATP channels integrated with creatine and adenylate kinases: Amplification, tuning and sensing of energetic signals in the compartmentalized cellular environment , 2004, Molecular and Cellular Biochemistry.
[23] M. Shattock,et al. Responses to ischaemia and reperfusion in the mouse isolated perfused heart and the phenomenon of ‘contractile cycling’ , 2003, Clinical and experimental pharmacology & physiology.
[24] J. Connell,et al. Direct Activation of AMP-activated Protein Kinase Stimulates Nitric-oxide Synthesis in Human Aortic Endothelial Cells* , 2003, Journal of Biological Chemistry.
[25] Edwin Janssen,et al. Impaired Intracellular Energetic Communication in Muscles from Creatine Kinase and Adenylate Kinase (M-CK/AK1) Double Knock-out Mice* , 2003, Journal of Biological Chemistry.
[26] Jan B Hoek,et al. Hexokinase II: the integration of energy metabolism and control of apoptosis. , 2003, Current medicinal chemistry.
[27] A. Terzic,et al. Phosphotransfer networks and cellular energetics , 2003, Journal of Experimental Biology.
[28] A. Terzic,et al. Cellular remodeling in heart failure disrupts KATP channel‐dependent stress tolerance , 2003, The EMBO journal.
[29] A. Terzic,et al. Adenylate Kinase 1 Deficiency Induces Molecular and Structural Adaptations to Support Muscle Energy Metabolism* , 2003, The Journal of Biological Chemistry.
[30] R. Boucher,et al. Human Airway Ecto-adenylate Kinase , 2003, The Journal of Biological Chemistry.
[31] A. Terzic,et al. Energetic communication between mitochondria and nucleus directed by catalyzed phosphotransfer , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[32] Andre Terzic,et al. Coupling of Cell Energetics with Membrane Metabolic Sensing , 2002, The Journal of Biological Chemistry.
[33] J. Headrick,et al. Coronary function and adenosine receptor-mediated responses in ischemic-reperfused mouse heart. , 2002, Cardiovascular research.
[34] T. Shibasaki,et al. Mouse model of Prinzmetal angina by disruption of the inward rectifier Kir6.1 , 2002, Nature Medicine.
[35] J. Balschi,et al. The Relationship between AMP-activated Protein Kinase Activity and AMP Concentration in the Isolated Perfused Rat Heart* , 2002, The Journal of Biological Chemistry.
[36] A. Terzic,et al. Cellular Energetics in the Preconditioned State , 2001, The Journal of Biological Chemistry.
[37] H. Ito. No reflow phenomenon in coronary heart disease. , 2001, Journal of cardiology.
[38] A. Terzic,et al. Adenylate kinase phosphotransfer communicates cellular energetic signals to ATP-sensitive potassium channels , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[39] S. Jalkanen,et al. Extracellular ATP formation on vascular endothelial cells is mediated by ecto‐nucleotide kinase activities via phosphotransfer reactions , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[40] Edwin Janssen,et al. Compromised Energetics in the Adenylate Kinase AK1Gene Knockout Heart under Metabolic Stress* , 2000, The Journal of Biological Chemistry.
[41] Arend Heerschap,et al. Adenylate kinase 1 gene deletion disrupts muscle energetic economy despite metabolic rearrangement , 2000, The EMBO journal.
[42] A. Terzic,et al. Failing energetics in failing hearts , 2000, Current cardiology reports.
[43] A. Terzic,et al. Adenylate kinase-catalyzed phosphotransfer in the myocardium : increased contribution in heart failure. , 1999, Circulation research.
[44] A. Terzic,et al. Phosphotransfer reactions in the regulation of ATP‐sensitive K+ channels , 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[45] E. Marra,et al. Mitochondrial energy metabolism in the left ventricular tissue of spontaneously hypertensive rats: abnormalities in both adeninenucleotide and phosphate translocators and enzyme adenylate-kinase and creatine-phosphokinase activities. , 1998, Clinical and experimental hypertension.
[46] R. Bache,et al. ATP-sensitive K+ channels, adenosine, and nitric oxide-mediated mechanisms account for coronary vasodilation during exercise. , 1998, Circulation research.
[47] J. Schrader,et al. Hypoxia-induced inhibition of adenosine kinase potentiates cardiac adenosine release. , 1997, Circulation research.
[48] P. Dzeja,et al. Suppression of Creatine Kinase-catalyzed Phosphotransfer Results in Increased Phosphoryl Transfer by Adenylate Kinase in Intact Skeletal Muscle* , 1996, The Journal of Biological Chemistry.
[49] J P Delahaye,et al. The risk of infective endocarditis after cardiac surgical and interventional procedures. , 1995, European heart journal.
[50] C. Jones,et al. Role of nitric oxide in the coronary microvascular responses to adenosine and increased metabolic demand. , 1995, Circulation.
[51] J. Daut,et al. KATP channels and basal coronary vascular tone. , 1994, Cardiovascular research.
[52] J. Schrader,et al. Rapid turnover of the AMP-adenosine metabolic cycle in the guinea pig heart. , 1993, Circulation research.
[53] M. Yamada,et al. Tissue-specific and developmentally regulated expression of the genes encoding adenylate kinase isozymes. , 1993, Journal of biochemistry.
[54] N D Goldberg,et al. Kinetics and compartmentation of energy metabolism in intact skeletal muscle determined from 18O labeling of metabolite phosphoryls. , 1991, The Journal of biological chemistry.
[55] H. Takenaka,et al. Multiforms of mammalian adenylate kinase and its monoclonal antibody against AK1. , 1990, Enzyme.
[56] R. Paul. Smooth muscle energetics. , 1989, Annual review of physiology.
[57] S. Dawis,et al. Adenosine triphosphate utilization rates and metabolic pool sizes in intact cells measured by transfer of 18O from water. , 1989, Biophysical journal.
[58] S. Bessman,et al. Myokinase and contractile function of glycerinated muscle fibers. , 1986, Biochemical medicine and metabolic biology.
[59] R. Berne. The role of adenosine in the regulation of coronary blood flow. , 1980, Circulation research.