Mechanism of dilation to reactive oxygen species in human coronary arterioles.
暂无分享,去创建一个
[1] F. Fonnum,et al. Evaluation of the probes 2',7'-dichlorofluorescin diacetate, luminol, and lucigenin as indicators of reactive species formation. , 2003, Biochemical pharmacology.
[2] Takashi Saito,et al. Role for Hydrogen Peroxide in Flow-Induced Dilation of Human Coronary Arterioles , 2003, Circulation research.
[3] J. Joseph,et al. Oxidative Stress–Induced Iron Signaling Is Responsible for Peroxide-Dependent Oxidation of Dichlorodihydrofluorescein in Endothelial Cells: Role of Transferrin Receptor–Dependent Iron Uptake in Apoptosis , 2003, Circulation research.
[4] T. Vanden Hoek,et al. ROS and NO trigger early preconditioning: relationship to mitochondrial KATP channel. , 2003, American journal of physiology. Heart and circulatory physiology.
[5] D. Harrison,et al. NAD(P)H Oxidase-derived Hydrogen Peroxide Mediates Endothelial Nitric Oxide Production in Response to Angiotensin II* , 2002, The Journal of Biological Chemistry.
[6] Q. Chai,et al. Peroxynitrite Inhibits Ca2+-Activated K+ Channel Activity in Smooth Muscle of Human Coronary Arterioles , 2002, Circulation research.
[7] D. Mottet,et al. Regulation of gene expression by oxygen: NF-kappaB and HIF-1, two extremes. , 2002, Free radical biology & medicine.
[8] P. Vanhoutte,et al. Oxygen‐derived free radicals mediate endothelium‐dependent contractions to acetylcholine in aortas from spontaneously hypertensive rats , 2002, British journal of pharmacology.
[9] U. Laufs,et al. Cellular Antioxidant Effects of Atorvastatin In Vitro and In Vivo , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[10] Hiroaki Shimokawa,et al. Hydrogen peroxide is an endothelium-derived hyperpolarizing factor in human mesenteric arteries. , 2002, Biochemical and biophysical research communications.
[11] W. Dröge. Free radicals in the physiological control of cell function. , 2002, Physiological reviews.
[12] T. Meinertz,et al. Endothelial Dysfunction, Oxidative Stress, and Risk of Cardiovascular Events in Patients With Coronary Artery Disease , 2001, Circulation.
[13] S. Goldman,et al. Oxidative stress contributes to vascular endothelial dysfunction in heart failure. , 2001, American journal of physiology. Heart and circulatory physiology.
[14] L. Oberley,et al. H2O2-induced O⨪2Production by a Non-phagocytic NAD(P)H Oxidase Causes Oxidant Injury* , 2001, The Journal of Biological Chemistry.
[15] I. Fridovich,et al. Methods of detection of vascular reactive species: nitric oxide, superoxide, hydrogen peroxide, and peroxynitrite. , 2001, Circulation research.
[16] D. Gutterman,et al. High Glucose Impairs Voltage-Gated K+ Channel Current in Rat Small Coronary Arteries , 2001, Circulation research.
[17] J. Keaney,et al. Iron Chelation Improves Endothelial Function in Patients With Coronary Artery Disease , 2001, Circulation.
[18] M. Azadniv,et al. Neutrophils in lung inflammation: Which reactive oxygen species are being measured? , 2001, Inhalation toxicology.
[19] Takashi Saito,et al. Flow-Induced Dilation of Human Coronary Arterioles: Important Role of Ca2+-Activated K+ Channels , 2001, Circulation.
[20] K. Kamata,et al. Effect of chronic insulin treatment on NO production and endothelium-dependent relaxation in aortae from established STZ-induced diabetic rats. , 2001, Atherosclerosis.
[21] T. Itoh,et al. Mechanisms of hydrogen peroxide-induced relaxation in rabbit mesenteric small artery. , 2001, European journal of pharmacology.
[22] T. Münzel,et al. Antioxidants and endothelial dysfunction in Hyperlipidemia , 2001, Current Hypertension Reports.
[23] S. Seto,et al. Alteration of antioxidants during the progression of heart disease in streptozotocin-induced diabetic rats , 2001, Free radical research.
[24] H Shimokawa,et al. Hydrogen peroxide is an endothelium-derived hyperpolarizing factor in mice. , 2000, The Journal of clinical investigation.
[25] M. Takumida,et al. Effect of hydrogen peroxide on guinea pig nasal mucosa vasculature. , 2000, Japanese journal of pharmacology.
[26] H. Miura,et al. Human coronary arteriolar dilation to adrenomedullin: role of nitric oxide and K(+) channels. , 2000, American journal of physiology. Heart and circulatory physiology.
[27] R. Ziegelstein,et al. Hypoxia/Reoxygenation Stimulates Intracellular Calcium Oscillations in Human Aortic Endothelial Cells , 2000, Circulation.
[28] D. Stepp,et al. Nitric oxide exerts feedback inhibition on EDHF-induced coronary arteriolar dilation in vivo. , 2000, American journal of physiology. Heart and circulatory physiology.
[29] H. Nohl,et al. H(2)O(2) detection from intact mitochondria as a measure for one-electron reduction of dioxygen requires a non-invasive assay system. , 1999, Biochimica et biophysica acta.
[30] O. Carretero,et al. Reactive oxygen species: role in the relaxation induced by bradykinin or arachidonic acid via EDHF in isolated porcine coronary arteries. , 1999, Journal of cardiovascular pharmacology.
[31] H. Miura,et al. Human coronary arteriolar dilation to bradykinin depends on membrane hyperpolarization: contribution of nitric oxide and Ca2+-activated K+ channels. , 1999, Circulation.
[32] M. Reid,et al. Exogenous reactive oxygen and nitric oxide alter intracellular oxidant status of skeletal muscle fibres. , 1999, Acta physiologica Scandinavica.
[33] H. Miura,et al. Human coronary arteriolar dilation to arachidonic acid depends on cytochrome P-450 monooxygenase and Ca2+-activated K+ channels. , 1998, Circulation research.
[34] D. Gutterman,et al. Pharmacologic activation of the human coronary microcirculation in vitro: endothelium-dependent dilation and differential responses to acetylcholine. , 1998, Cardiovascular research.
[35] D. Mattson,et al. Iron attenuates nitric oxide level and iNOS expression in endotoxin‐treated mice , 1998, FEBS letters.
[36] A. Imrich,et al. Fluorescence-based measurement of nitric oxide synthase activity in activated rat macrophages using dichlorofluorescin. , 1997, Nitric oxide : biology and chemistry.
[37] K. Prasad,et al. Mechanism of hydroxyl radical-induced modulation of vascular tone. , 1997, Free radical biology & medicine.
[38] K. Prasad,et al. Hydroxyl radical--a mediator of acetylcholine-induced vascular relaxation. , 1996, Journal of molecular and cellular cardiology.
[39] V. Ullrich,et al. Peroxynitrite formed by simultaneous generation of nitric oxide and superoxide selectively inhibits bovine aortic prostacyclin synthase , 1996, FEBS letters.
[40] G. Paolisso,et al. Oxidative Stress and Diabetic Vascular Complications , 1996, Diabetes Care.
[41] H. Ischiropoulos,et al. Evaluation of 2',7'-dichlorofluorescin and dihydrorhodamine 123 as fluorescent probes for intracellular H2O2 in cultured endothelial cells. , 1993, Archives of biochemistry and biophysics.
[42] C. Piantadosi,et al. Extracellular superoxide dismutase, nitric oxide, and central nervous system O2 toxicity. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[43] J. T. Wachsman,et al. Hydrogen peroxide stimulates rat colonic prostaglandin production and alters electrolyte transport. , 1990, The Journal of clinical investigation.
[44] C. White,et al. Superoxide dismutase and catalase conjugated to polyethylene glycol increases endothelial enzyme activity and oxidant resistance. , 1988, The Journal of biological chemistry.
[45] J. McCord,et al. Oxygen-derived free radicals in postischemic tissue injury. , 1985, The New England journal of medicine.
[46] B. Matkovics,et al. The Effect of Diabetes on the Activities of the Peroxide Metabolism Enzymes , 1982, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[47] F. Murad,et al. Activation of guanylate cyclase by superoxide dismutase and hydroxyl radical: a physiological regulator of guanosine 3',5'-monophosphate formation. , 1977, Proceedings of the National Academy of Sciences of the United States of America.