Effects of sex hormones on inflammatory response in male and female vascular endothelial cells

[1]  P. Libby,et al.  Progress and challenges in translating the biology of atherosclerosis , 2011, Nature.

[2]  G. Norata,et al.  The androgen derivative 5alpha-androstane-3beta,17beta-diol inhibits tumor necrosis factor alpha and lipopolysaccharide induced inflammatory response in human endothelial cells and in mice aorta. , 2010, Atherosclerosis.

[3]  F. Saad,et al.  The effects of testosterone on risk factors for, and the mediators of, the atherosclerotic process. , 2009, Atherosclerosis.

[4]  G. Rosano,et al.  Gender differences in the cardiovascular effect of sex hormones , 2009, Nature Reviews Cardiology.

[5]  M. Koupparis,et al.  Structural characteristics of anabolic androgenic steroids contributing to binding to the androgen receptor and to their anabolic and androgenic activities Applied modifications in the steroidal structure , 2009, Steroids.

[6]  U. Jeschke,et al.  Human umbilical vascular endothelial cells express estrogen receptor beta (ERβ) and progesterone receptor A (PR-A), but not ERα and PR-B , 2008, Histochemistry and Cell Biology.

[7]  M. Lachat,et al.  Differential Effects of 17&bgr;-Estradiol on Function and Expression of Estrogen Receptor &agr;, Estrogen Receptor &bgr;, and GPR30 in Arteries and Veins of Patients With Atherosclerosis , 2007 .

[8]  M. Lachat,et al.  Differential effects of 17beta-estradiol on function and expression of estrogen receptor alpha, estrogen receptor beta, and GPR30 in arteries and veins of patients with atherosclerosis. , 2007, Hypertension.

[9]  R. Lathe,et al.  Erratum: An endocrine pathway in the prostate, ERβ, AR, 5α-androstane-3β,17β-diol, and CYP7B1, regulates prostate growth (Proceedings of the National Academy of Sciences of the United States of America (October 15, 2002) 99, 21 (13589-13594) (DOI: 10.1073/pnas.162477299)) , 2006 .

[10]  G. Norata,et al.  Dihydrotestosterone decreases tumor necrosis factor-alpha and lipopolysaccharide-induced inflammatory response in human endothelial cells. , 2006, The Journal of clinical endocrinology and metabolism.

[11]  B. Katzenellenbogen,et al.  The androgen derivative 5alpha-androstane-3beta,17beta-diol inhibits prostate cancer cell migration through activation of the estrogen receptor beta subtype. , 2005, Cancer research.

[12]  R. Karas,et al.  Molecular and Cellular Basis of Cardiovascular Gender Differences , 2005, Science.

[13]  S. Chensue,et al.  Tissue-specific effect of estradiol on endothelial cell-dependent lymphocyte recruitment. , 2004, Microvascular research.

[14]  M. Mori,et al.  Suppression by 17beta-estradiol of monocyte adhesion to vascular endothelial cells is mediated by estrogen receptors. , 2004, Life sciences.

[15]  David Handelsman,et al.  Dihydrotestosterone promotes vascular cell adhesion molecule-1 expression in male human endothelial cells via a nuclear factor-kappaB-dependent pathway. , 2004, Endocrinology.

[16]  A. von Eckardstein,et al.  Androgens and coronary artery disease. , 2003, Endocrine reviews.

[17]  P. Shaul,et al.  Estrogen Causes Dynamic Alterations in Endothelial Estrogen Receptor Expression , 2002, Circulation research.

[18]  R. Lathe,et al.  An endocrine pathway in the prostate, ERβ, AR, 5α-androstane-3β,17β-diol, and CYP7B1, regulates prostate growth , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[19]  Mark J. Evans,et al.  Estrogen Receptors (cid:1) and (cid:2) Have Similar Activities in Multiple Endothelial Cell Pathways , 2002 .

[20]  Hong Zhao,et al.  Effects of testosterone and 17-β-estradiol on TNF-α-induced E-selectin and VCAM-1 expression in endothelial cells , 2002 .

[21]  T. Mukherjee,et al.  Testosterone attenuates expression of vascular cell adhesion molecule-1 by conversion to estradiol by aromatase in endothelial cells: Implications in atherosclerosis , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[22]  M. Evans,et al.  Estrogen receptors alpha and beta have similar activities in multiple endothelial cell pathways. , 2002, Endocrinology.

[23]  Hong Zhao,et al.  Effects of testosterone and 17-beta-estradiol on TNF-alpha-induced E-selectin and VCAM-1 expression in endothelial cells. Analysis of the underlying receptor pathways. , 2002, Life sciences.

[24]  R. Lathe,et al.  An endocrine pathway in the prostate, ERbeta, AR, 5alpha-androstane-3beta,17beta-diol, and CYP7B1, regulates prostate growth. , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[25]  L. Ghiadoni,et al.  Mechanisms responsible for endothelial dysfunction associated with acute estrogen deprivation in normotensive women. , 2000, Circulation.

[26]  C. Plass,et al.  Methylation of the estrogen receptor-alpha gene promoter is selectively increased in proliferating human aortic smooth muscle cells. , 2000, Cardiovascular research.

[27]  J. Issa,et al.  Methylation of the estrogen receptor gene is associated with aging and atherosclerosis in the cardiovascular system. , 1999, Cardiovascular research.

[28]  David Handelsman,et al.  Androgen exposure increases human monocyte adhesion to vascular endothelium and endothelial cell expression of vascular cell adhesion molecule-1. , 1999, Circulation.

[29]  A. Akane Sex determination by PCR analysis of the X-Y amelogenin gene. , 1998, Methods in molecular biology.

[30]  R. Pardi,et al.  Effects of 17beta-estradiol on cytokine-induced endothelial cell adhesion molecule expression. , 1996, The Journal of clinical investigation.