Missorting of cathepsin B into the secretory compartment of CI-MPR/IGFII-deficient mice does not induce spontaneous trypsinogen activation but leads to enhanced trypsin activity during experimental pancreatitis--without affecting disease severity.
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M. Lerch | J. Schnekenburger | W. Domschke | M. Sendler | D. Hahn | T. Meister | R. Niehues | D. Hahn | M. Lerch | Juergen Schnekenburger