Simultaneous time-varying systemic appearance of oral and hepatic glucose in adults monitored with stable isotopes.
暂无分享,去创建一个
J. Eagles | J. Brown | F. Mellon | G. Livesey | R. Faulks | M. Roe | J. Dainty | P. Wilson | G Livesey | P D Wilson | J R Dainty | J C Brown | R M Faulks | M A Roe | T A Newman | J Eagles | F A Mellon | R H Greenwood | R. Greenwood | T. A. Newman
[1] G. Reaven. Banting lecture 1988 , 1997 .
[2] P Vicini,et al. The hot but not the cold minimal model allows precise assessment of insulin sensitivity in NIDDM subjects. , 1996, The American journal of physiology.
[3] J. Delarue,et al. Effects of fish oil on metabolic responses to oral fructose and glucose loads in healthy humans. , 1996, The American journal of physiology.
[4] P. Whincup,et al. Prospective study of risk factors for development of non-insulin dependent diabetes in middle aged British men , 1995, BMJ.
[5] F. Féry,et al. Glucose Metabolism During the Starved-To-Fed Transition in Obese Patients With NIDDM , 1994, Diabetes.
[6] A Mari,et al. Glucose absorption and production following oral glucose: comparison of compartmental and arteriovenous-difference methods. , 1994, Metabolism: clinical and experimental.
[7] B. Williams,et al. Insulin resistance: the shape of things to come , 1994, The Lancet.
[8] P. Klein,et al. The use of uniformly labelled substrates and mass isotopomer analysis to study intermediary metabolism , 1994, Proceedings of the Nutrition Society.
[9] N. Mcintyre,et al. Metabolic handling of orally administered glucose in cirrhosis. , 1993, Journal of Clinical Investigation.
[10] A Mari,et al. Estimation of the rate of appearance in the non-steady state with a two-compartment model. , 1992, The American journal of physiology.
[11] T. R. Hennessy,et al. Incomplete suppression of hepatic glucose production in non‐insulin dependent diabetes mellitus measured with [6,6‐2H2]glucose enriched glucose infusion during hyperinsulinaemic euglycaemic clamps , 1992, European journal of clinical investigation.
[12] D. Kelley,et al. Role of reduced suppression of glucose production and diminished early insulin release in impaired glucose tolerance. , 1992, The New England journal of medicine.
[13] M. Eggstein,et al. Selected ion monitoring gas chromatography/mass spectrometry using uniformly labelled (13C)glucose for determination of glucose turnover in man. , 1991, Biological mass spectrometry.
[14] T. Jenssen,et al. Contribution of Abnormal Muscle and Liver Glucose Metabolism to Postprandial Hyperglycemia in NIDDM , 1990, Diabetes.
[15] W. Rühle,et al. Band-gap dynamics and nonlinear propagation of optical pulses in CdTe , 1990 .
[16] R. Wolfe,et al. Role of the glucose cycle in control of net glucose flux in exercising humans. , 1990, Journal of applied physiology.
[17] T. Jenssen,et al. Contribution of abnormal muscle and liver glucose metabolism to postprandial hyperglycemia in NIDDM. , 1990, Diabetes.
[18] J. Levy,et al. Hepatic glucose output in humans measured with labeled glucose to reduce negative errors. , 1989, American Journal of Physiology.
[19] G. Reaven. Role of Insulin Resistance in Human Disease , 1988, Diabetes.
[20] G Sonnenberg,et al. Skeletal muscle glycolysis, oxidation, and storage of an oral glucose load. , 1988, The Journal of clinical investigation.
[21] C Cobelli,et al. Non-steady state: error analysis of Steele's model and developments for glucose kinetics. , 1987, The American journal of physiology.
[22] R. Rizza,et al. Postprandial hyperglycemia in patients with noninsulin-dependent diabetes mellitus. Role of hepatic and extrahepatic tissues. , 1986, The Journal of clinical investigation.
[23] R. DeFronzo,et al. The Disposal of an Oral Glucose Load in Healthy Subjects: A Quantitative Study , 1985, Diabetes.
[24] R. Rizza,et al. Abnormal meal carbohydrate disposition in insulin-dependent diabetes. Relative contributions of endogenous glucose production and initial splanchnic uptake and effect of intensive insulin therapy. , 1984, The Journal of clinical investigation.
[25] J. Radziuk. Hepatic glycogen formation by direct uptake of glucose following oral glucose loading in man. , 1979, Canadian journal of physiology and pharmacology.
[26] R. Wolfe,et al. The realiability of rates of glucose appearance in vivo calculated from constant tracer infusions. , 1978, The Biochemical journal.
[27] T. McDonald,et al. Initial splanchnic extraction of ingested glucose in normal man. , 1978, Metabolism: clinical and experimental.
[28] K.,et al. Experimental validation of measurements of glucose turnover in nonsteady state. , 1978, The American journal of physiology.
[29] M Berman,et al. Insulin control of glucose metabolism in man: a new kinetic analysis. , 1975, The Journal of clinical investigation.
[30] K H Norwich,et al. Experimental validation of nonsteady rate measurements using a tracer infusion method and inulin as tracer and tracee. , 1974, Canadian journal of physiology and pharmacology.
[31] R. J. Bondar,et al. Evaluation of glucose-6-phosphate dehydrogenase from Leuconostoc mesenteroides in the hexokinase method for determining glucose in serum. , 1974, Clinical chemistry.
[32] Rachel Jenkins. Health of the nation. , 1969, British medical journal.
[33] R. Steele,et al. INFLUENCES OF GLUCOSE LOADING AND OF INJECTED INSULIN ON HEPATIC GLUCOSE OUTPUT * , 1959, Annals of the New York Academy of Sciences.
[34] I. L. Schwartz. Measurement of extracellular fluid by means of a constant infusion technique without collection of urine. , 1950, The American journal of physiology.