Mucosal-activated invariant T cells do not exhibit significant lung recruitment and proliferation profiles in macaques in response to infection with Mycobacterium tuberculosis CDC1551.

[1]  M. Kuroda,et al.  High Turnover of Tissue Macrophages Contributes to Tuberculosis Reactivation in Simian Immunodeficiency Virus-Infected Rhesus Macaques , 2018, The Journal of infectious diseases.

[2]  D. Kalman,et al.  In vivo inhibition of tryptophan catabolism reorganizes the tuberculoma and augments immune-mediated control of Mycobacterium tuberculosis , 2017, Proceedings of the National Academy of Sciences.

[3]  A. Lackner,et al.  Nonpathologic Infection of Macaques by an Attenuated Mycobacterial Vaccine Is Not Reactivated in the Setting of HIV Co-Infection. , 2017, The American journal of pathology.

[4]  A. Lackner,et al.  Translational Research in the Nonhuman Primate Model of Tuberculosis. , 2017, ILAR journal.

[5]  O. Lantz,et al.  MAIT cells in infectious diseases. , 2017, Current opinion in immunology.

[6]  J. Bader,et al.  Hypoxia Sensing and Persistence Genes Are Expressed during the Intragranulomatous Survival of Mycobacterium tuberculosis , 2017, American journal of respiratory cell and molecular biology.

[7]  Thurston H. Y. Dang,et al.  MR1 Restricted Mucosal-Associated Invariant T (MAIT) Cells Respond to Mycobacterial Vaccination and Infection in Nonhuman Primates , 2016, Mucosal Immunology.

[8]  W. Jacobs,et al.  CD4+ T-cell–independent mechanisms suppress reactivation of latent tuberculosis in a macaque model of HIV coinfection , 2016, Proceedings of the National Academy of Sciences.

[9]  Xinjing Wang,et al.  Mucosal-associated invariant T cells from patients with tuberculosis exhibit impaired immune response. , 2016, The Journal of infection.

[10]  A. Sette,et al.  The TB-specific CD4(+) T cell immune repertoire in both cynomolgus and rhesus macaques largely overlap with humans. , 2015, Tuberculosis.

[11]  A. Lackner,et al.  Mucosal vaccination with attenuated Mycobacterium tuberculosis induces strong central memory responses and protects against tuberculosis , 2015, Nature Communications.

[12]  Josh Eckels,et al.  A tuberculosis ontology for host systems biology. , 2015, Tuberculosis.

[13]  T. Niu,et al.  The DosR Regulon Modulates Adaptive Immunity and Is Essential for Mycobacterium tuberculosis Persistence. , 2015, American journal of respiratory and critical care medicine.

[14]  E. Shankar,et al.  Attrition of TCR Vα7.2+ CD161++ MAIT Cells in HIV-Tuberculosis Co-Infection Is Associated with Elevated Levels of PD-1 Expression , 2015, PloS one.

[15]  P. Klenerman,et al.  MAIT cells are licensed through granzyme exchange to kill bacterially sensitized targets , 2014, Mucosal Immunology.

[16]  A. Lackner,et al.  Aerosol Vaccination with AERAS-402 Elicits Robust Cellular Immune Responses in the Lungs of Rhesus Macaques but Fails To Protect against High-Dose Mycobacterium tuberculosis Challenge , 2014, The Journal of Immunology.

[17]  M. Lacey,et al.  Identification of biomarkers for tuberculosis susceptibility via integrated analysis of gene expression and longitudinal clinical data , 2014, Front. Genet..

[18]  M. Selman,et al.  S100A8/A9 proteins mediate neutrophilic inflammation and lung pathology during tuberculosis. , 2013, American journal of respiratory and critical care medicine.

[19]  L. Morici,et al.  Post-Exposure Therapeutic Efficacy of COX-2 Inhibition against Burkholderia pseudomallei , 2013, PLoS neglected tropical diseases.

[20]  S. Lawn,et al.  Diagnostic and prognostic value of serum C-reactive protein for screening for HIV-associated tuberculosis. , 2013, The international journal of tuberculosis and lung disease : the official journal of the International Union against Tuberculosis and Lung Disease.

[21]  A. Lackner,et al.  Granuloma correlates of protection against tuberculosis and mechanisms of immune modulation by Mycobacterium tuberculosis. , 2013, Journal of Infectious Diseases.

[22]  M. Selman,et al.  CXCR5⁺ T helper cells mediate protective immunity against tuberculosis. , 2013, The Journal of clinical investigation.

[23]  J. Flynn,et al.  Metronidazole prevents reactivation of latent Mycobacterium tuberculosis infection in macaques , 2012, Proceedings of the National Academy of Sciences.

[24]  A. Lackner,et al.  The non‐human primate model of tuberculosis , 2012, Journal of medical primatology.

[25]  M. Kuroda,et al.  The Mycobacterium tuberculosis stress response factor SigH is required for bacterial burden as well as immunopathology in primate lungs. , 2012, The Journal of infectious diseases.

[26]  J. Blanchard,et al.  Reactivation of latent tuberculosis in rhesus macaques by coinfection with simian immunodeficiency virus , 2011, Journal of medical primatology.

[27]  D. Kaushal,et al.  Improved Xenobiotic Metabolism and Reduced Susceptibility to Cancer in Gluten-Sensitive Macaques upon Introduction of a Gluten-Free Diet , 2011, PloS one.

[28]  Toshihiro Ito,et al.  CCR6 as a mediator of immunity in the lung and gut. , 2011, Experimental cell research.

[29]  O. Lantz,et al.  Human MAIT cells are xenobiotic-resistant, tissue-targeted, CD161hi IL-17-secreting T cells. , 2011, Blood.

[30]  D. Kaushal,et al.  Transcriptional Reprogramming in Nonhuman Primate (Rhesus Macaque) Tuberculosis Granulomas , 2010, PloS one.

[31]  O. Lantz,et al.  Antimicrobial activity of mucosal-associated invariant T cells , 2010, Nature Immunology.

[32]  Matthew S. Cook,et al.  Human Mucosal Associated Invariant T Cells Detect Bacterially Infected Cells , 2010, PLoS biology.

[33]  Nicholas A. Be,et al.  Genetic requirements for the survival of tubercle bacilli in primates. , 2010, The Journal of infectious diseases.

[34]  David G. Russell,et al.  Tuberculosis: What We Don’t Know Can, and Does, Hurt Us , 2010, Science.

[35]  B. Haynes,et al.  A Critical Role for CD8 T Cells in a Nonhuman Primate Model of Tuberculosis , 2009, PLoS pathogens.

[36]  Z. Sun,et al.  The roles of CCR6 in migration of Th17 cells and regulation of effector T-cell balance in the gut , 2009, Mucosal Immunology.

[37]  M. Raviglione The new Stop TB Strategy and the Global Plan to Stop TB, 2006-2015. , 2007, Bulletin of the World Health Organization.

[38]  Z. Xing,et al.  Activation of CD8 T Cells by Mycobacterial Vaccination Protects against Pulmonary Tuberculosis in the Absence of CD4 T Cells1 , 2004, The Journal of Immunology.

[39]  I. Orme,et al.  CD4 is required for the development of a protective granulomatous response to pulmonary tuberculosis. , 2002, Cellular immunology.

[40]  R. V. van Neerven,et al.  The CC-chemokine receptor 5 (CCR5) is a marker of, but not essential for the development of human Th1 cells. , 1999, Tissue antigens.

[41]  J. Flynn,et al.  An essential role for interferon gamma in resistance to Mycobacterium tuberculosis infection , 1993, The Journal of experimental medicine.

[42]  M. Colonna,et al.  Innate lymphoid cells: A new paradigm in immunology , 2015, Science.

[43]  D. Kaushal,et al.  Humoral and lung immune responses to Mycobacterium tuberculosis infection in a primate model of protection. , 2014, Trials in vaccinology.

[44]  S. Behar,et al.  Infection Mycobacterium tuberculosis Development of Central Memory during T Cells and the + Antigen-Specific CD8 , 2006 .

[45]  I. Orme,et al.  Materials and Methods Briefdefinitive Report Disseminated Tuberculosis in Interferon 7 Gene-disrupted Mice , 2022 .

[46]  M. Selman,et al.  IMMUNOPATHOLOGY AND INFECTIOUS DISEASES LAG 3 Expression in Active Mycobacterium tuberculosis Infections , 2022 .