Large-scale genome-wide association studies in east Asians identify new genetic loci influencing metabolic traits
暂无分享,去创建一个
Taesung Park | Cheng Hu | Bermseok Oh | Kuchan Kimm | Yukinori Okada | Naoyuki Kamatani | Michiaki Kubo | Weiping Jia | Koichi Matsuda | Chang Bum Hong | Min Jin Go | Yoon Shin Cho | Dong Joon Kim | Atsushi Takahashi | Y. J. Kim | Daehee Kang | Soeui Kim | Y. Okada | Toshihiro Tanaka | K. Matsuda | D. Kang | Y. S. Cho | Jong-Young Lee | B. Han | M. Kubo | B. Oh | M. Go | A. Takahashi | N. Kamatani | Cheng Hu | W. Jia | T. Park | Ji-Young Lee | C. Shin | Eun-Jung Hong | Young-Jin Kim | Joo-Yeon Hwang | Ji-Hyun Kim | Rong Zhang | N. Cho | Y. Kim | K. Kimm | Jong-Young Lee | Yun Kyoung Kim | Toshihiro Tanaka | Ji-Young Lee | Chol Shin | Rong Zhang | Hyung Lae Kim | Young-Jin Kim | Nam H. Cho | Bok Ghee Han | J. Oh | N. Kim | Hyung L. Kim | Haesook Min | Ji Hee Oh | C. Hong | Dong Joon Kim | So-Hyun Kim | H. Min | Yeonjung Kim | Ji-Hyun Kim | Yeonjung Kim | Joo Yeon Hwang | Nam Hee Kim | Eun Jung Hong | Dong-Joon Kim | Y. Cho | H. Kim | Ji Young Lee | M. Kubo
[1] Luigi Ferrucci,et al. Common variation in the beta-carotene 15,15'-monooxygenase 1 gene affects circulating levels of carotenoids: a genome-wide association study. , 2009, American journal of human genetics.
[2] Uwe Völker,et al. New loci associated with kidney function and chronic kidney disease , 2010, Nature Genetics.
[3] Yusuke Nakamura,et al. [BioBank Japan project]. , 2005, Nihon rinsho. Japanese journal of clinical medicine.
[4] Yusuke Nakamura,et al. Genome-wide association study of hematological and biochemical traits in a Japanese population , 2010, Nature Genetics.
[5] Tien Yin Wong,et al. Meta-analysis of genome-wide association studies identifies common variants associated with blood pressure variation in east Asians , 2011, Nature Genetics.
[6] N. Kato,et al. Polymorphism of OAS‐1 determines liver fibrosis progression in hepatitis C by reduced ability to inhibit viral replication , 2009, Liver international : official journal of the International Association for the Study of the Liver.
[7] Judy H. Cho,et al. Finding the missing heritability of complex diseases , 2009, Nature.
[8] C Polychronakos,et al. Type 1 diabetes and the OAS gene cluster: association with splicing polymorphism or haplotype? , 2005, Journal of Medical Genetics.
[9] N. Cook,et al. Loci related to metabolic-syndrome pathways including LEPR,HNF1A, IL6R, and GCKR associate with plasma C-reactive protein: the Women's Genome Health Study. , 2008, American journal of human genetics.
[10] Manuel A. R. Ferreira,et al. PLINK: a tool set for whole-genome association and population-based linkage analyses. , 2007, American journal of human genetics.
[11] Christian Gieger,et al. New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk , 2010, Nature Genetics.
[12] U. Francke. Williams-Beuren syndrome: genes and mechanisms. , 1999, Human molecular genetics.
[13] V. Salomaa,et al. Excess of rare variants in genes identified by genome-wide association study of hypertriglyceridemia , 2010, Nature Genetics.
[14] A. Hofman,et al. Large-scale genomic studies reveal central role of ABO in sP-selectin and sICAM-1 levels. , 2010, Human molecular genetics.
[15] E. Rimm,et al. Genetic variants in ABO blood group region, plasma soluble E-selectin levels and risk of type 2 diabetes. , 2010, Human molecular genetics.
[16] S. Heath,et al. Common susceptibility alleles are unlikely to contribute as strongly as the FV and ABO loci to VTE risk: results from a GWAS approach. , 2009, Blood.
[17] Liuda Ziaugra,et al. SNP Genotyping Using the Sequenom MassARRAY iPLEX Platform , 2009, Current protocols in human genetics.
[18] Ying Wang,et al. Genome-wide association study in a Chinese Han population identifies nine new susceptibility loci for systemic lupus erythematosus , 2009, Nature Genetics.
[19] C. Ting,et al. A genome-wide association study identifies new loci for ACE activity: potential implications for response to ACE inhibitor , 2010, The Pharmacogenomics Journal.
[20] L. Wasserman,et al. Genomic control, a new approach to genetic-based association studies. , 2001, Theoretical population biology.
[21] Dolores Corella,et al. Six new loci associated with blood low-density lipoprotein cholesterol, high-density lipoprotein cholesterol or triglycerides in humans , 2008, Nature Genetics.
[22] Luigi Ferrucci,et al. Population-based genome-wide association studies reveal six loci influencing plasma levels of liver enzymes. , 2008, American journal of human genetics.
[23] P. Ridker,et al. Association of 77 polymorphisms in 52 candidate genes with blood pressure progression and incident hypertension: the Women's Genome Health Study , 2008, Journal of hypertension.
[24] Taesung Park,et al. A large-scale genome-wide association study of Asian populations uncovers genetic factors influencing eight quantitative traits , 2009, Nature Genetics.
[25] S. MacMahon,et al. Use of the Friedewald formula to estimate LDL-cholesterol in patients with chronic renal failure on dialysis. , 1997, Clinical chemistry.
[26] Yurii S. Aulchenko,et al. Multiple loci associated with indices of renal function and chronic kidney disease , 2009, Nature Genetics.
[27] Geoffrey S. Tobias,et al. Genome-wide association study identifies variants in the ABO locus associated with susceptibility to pancreatic cancer , 2009, Nature Genetics.
[28] R. A. Bailey,et al. Robust associations of four new chromosome regions from genome-wide analyses of type 1 diabetes , 2007, Nature Genetics.
[29] Christian Gieger,et al. A genome-wide meta-analysis identifies 22 loci associated with eight hematological parameters in the HaemGen consortium , 2009, Nature Genetics.
[30] Melissa Bondy,et al. Genome-wide association study identifies five susceptibility loci for glioma , 2009, Nature Genetics.
[31] R. Collins,et al. Common variants at 30 loci contribute to polygenic dyslipidemia , 2009, Nature Genetics.
[32] Alex Doney,et al. Genetic variation in GIPR influences the glucose and insulin responses to an oral glucose challenge , 2010, Nature Genetics.
[33] Rick Twee-Hee Ong,et al. varLD: a program for quantifying variation in linkage disequilibrium patterns between populations , 2010, Bioinform..
[34] P. Donnelly,et al. A new multipoint method for genome-wide association studies by imputation of genotypes , 2007, Nature Genetics.
[35] D. Hernandez,et al. Multiple Genetic Loci Influence Serum Urate Levels and Their Relationship With Gout and Cardiovascular Disease Risk Factors , 2010, Circulation. Cardiovascular genetics.
[36] Joshua M. Korn,et al. Integrated genotype calling and association analysis of SNPs, common copy number polymorphisms and rare CNVs , 2008, Nature Genetics.
[37] R. D'Agostino,et al. Gamma Glutamyl Transferase and Metabolic Syndrome, Cardiovascular Disease, and Mortality Risk: The Framingham Heart Study , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[38] S. Yusuf,et al. A polygenic basis for four classical Fredrickson hyperlipoproteinemia phenotypes that are characterized by hypertriglyceridemia , 2009, Human molecular genetics.
[39] Evangelos Evangelou,et al. Heterogeneity in Meta-Analyses of Genome-Wide Association Investigations , 2007, PloS one.
[40] Christian Gieger,et al. Meta-Analysis of 28,141 Individuals Identifies Common Variants within Five New Loci That Influence Uric Acid Concentrations , 2009, PLoS genetics.
[41] R. Collins,et al. Newly identified loci that influence lipid concentrations and risk of coronary artery disease , 2008, Nature Genetics.
[42] S. Yusuf,et al. Polygenic determinants of severe hypertriglyceridemia. , 2008, Human molecular genetics.
[43] A. Gaw. HDL-C and Triglyceride Levels: Relationship to Coronary Heart Disease and Treatment with Statins , 2004, Cardiovascular Drugs and Therapy.
[44] C D Naylor,et al. Meta-analysis of controlled clinical trials. , 1989, The Journal of rheumatology.