GASTROINTESTINAL , HEPATOBILIARY , AND PANCREATIC PATHOLOGY Alcohol Exacerbates LPS-Induced Fibrosis in Subclinical Acute Pancreatitis

[1]  C. Holst,et al.  Progression From Acute to Chronic Pancreatitis: Prognostic Factors, Mortality, and Natural Course , 2011, Pancreas.

[2]  T. Śledziński,et al.  Coordinated Increase in Serum Platelet-Derived Growth Factor-BB and Transforming Growth Factor-β1 in Patients with Chronic Pancreatitis , 2011, Pancreatology.

[3]  R. Pirola,et al.  Withdrawal of alcohol promotes regression while continued alcohol intake promotes persistence of LPS-induced pancreatic injury in alcohol-fed rats , 2010, Gut.

[4]  V. Muddana,et al.  Toll-like receptor 4 polymorphisms in German and US patients are not associated with occurrence or severity of acute pancreatitis , 2010, Gut.

[5]  P. Maisonneuve,et al.  Natural History of Acute Pancreatitis: A Long-Term Population-Based Study , 2009, The American Journal of Gastroenterology.

[6]  G. Kroemer,et al.  Impaired autophagosome-lysosome fusion in the pathogenesis of pancreatitis , 2009, Autophagy.

[7]  M. Büchler,et al.  Impaired autolysosome formation correlates with Lamp-2 depletion: role of apoptosis, autophagy, and necrosis in pancreatitis. , 2009, Gastroenterology.

[8]  R. Takayanagi,et al.  Fractalkine and TGF-beta1 levels reflect the severity of chronic pancreatitis in humans. , 2008, World journal of gastroenterology.

[9]  I. Berger,et al.  Immune‐compromised state in the rat pancreas after chronic alcohol exposure: the role of peroxisome proliferator‐activated receptor γ , 2007, The Journal of pathology.

[10]  P. Maisonneuve,et al.  TIME TRENDS IN INCIDENCE OF ACUTE PANCREATITIS IN LÜNEBURG: A POPULATION-BASED STUDY , 2007 .

[11]  Z. Wajda,et al.  Biliary Versus Alcohol-Related Infected Pancreatic Necrosis Similarities and Differences in the Follow-Up , 2007, Pancreas.

[12]  Yuan Li,et al.  Alterations of Toll-like Receptor 4 Expression on Peripheral Blood Monocytes During the Early Stage of Human Acute Pancreatitis , 2007, Digestive Diseases and Sciences.

[13]  Yuan Li,et al.  Toll-Like Receptor 4 Asp299Gly Polymorphism Is Associated With an Increased Risk of Pancreatic Necrotic Infection in Acute Pancreatitis: A Study in the Chinese Population , 2007, Pancreas.

[14]  H. Friess,et al.  The neurotrophic factor artemin influences the extent of neural damage and growth in chronic pancreatitis , 2006, Gut.

[15]  M. Bachem,et al.  Role of stellate cells in pancreatic fibrogenesis associated with acute and chronic pancreatitis , 2006, Journal of gastroenterology and hepatology.

[16]  C. McClain,et al.  Pancreatic response to endotoxin after chronic alcohol exposure: switch from apoptosis to necrosis? , 2006, American journal of physiology. Gastrointestinal and liver physiology.

[17]  K. Hahm,et al.  Amelioration of Pancreatic Fibrosis in Mice With Defective TGF-β Signaling , 2005, Pancreas.

[18]  M. Apte,et al.  Mechanisms of Pancreatic Fibrosis , 2005, Digestive Diseases.

[19]  Andrew Leask,et al.  TGF‐β signaling and the fibrotic response , 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[20]  J. Wilson,et al.  Stellate Cell Activation in Alcoholic Pancreatitis , 2003, Pancreas.

[21]  G. Klöppel,et al.  Fibrosis of the pancreas: the initial tissue damage and the resulting pattern , 2003, Virchows Archiv.

[22]  J. Iredale,et al.  Expression of transforming growth factor-beta 1 by pancreatic stellate cells and its implications for matrix secretion and turnover in chronic pancreatitis. , 2002, The American journal of pathology.

[23]  C. Bode,et al.  [Alcohol, the gastrointestinal tract and pancreas]. , 2000, Therapeutische Umschau. Revue therapeutique.

[24]  J S Wilson,et al.  Activation of pancreatic stellate cells in human and experimental pancreatic fibrosis. , 1999, The American journal of pathology.

[25]  G. Klöppel.  Progression from acute to chronic pancreatitis. A pathologist's view. , 1999, The Surgical clinics of North America.

[26]  R. Graf,et al.  New concepts in understanding the pathophysiology of chronic pancreatitis , 1999, International journal of pancreatology : official journal of the International Association of Pancreatology.

[27]  R. Schmid,et al.  Identification, culture, and characterization of pancreatic stellate cells in rats and humans. , 1998, Gastroenterology.

[28]  R. Ammann.  A clinically based classification system for alcoholic chronic pancreatitis: summary of an international workshop on chronic pancreatitis. , 1997, Pancreas.

[29]  K. Miyazono,et al.  Localization of transforming growth factor β1 and its latent binding protein in human chronic pancreatitis , 1995 .

[30]  C. Lieber,et al.  The feeding of ethanol in liquid diets. , 1986, Alcoholism, clinical and experimental research.

[31]  H. Sarles,et al.  Observations on 205 confirmed cases of acute pancreatitis, recurring pancreatitis, and chronic pancreatitis. , 1965, Gut.

[32]  宮本 珠生.  Overexpression of Smad6 exacerbates pancreatic fibrosis in murine caerulein-induced chronic pancreatic injuries , 2010 .

[33]  Zhenzhen Wang,et al.  Protection of cerulein-induced pancreatic fibrosis by pancreas-specific expression of Smad7. , 2009, Biochimica et biophysica acta.

[34]  D. Jaffe.  Bacterial Endotoxin: A Trigger Factor for Alcoholic Pancreatitis? Evidence From a Novel, Physiologically Relevant Animal Model , 2008 .

[35]  H. Friess,et al.  Enhanced expression of TGF-betas and their receptors in human acute pancreatitis. , 1998, Annals of surgery.

[36]  G. Klöppel,et al.  The morphological basis for the evolution of acute pancreatitis into chronic pancreatitis. , 1992 .

[37]  C. Bode,et al.  Endotoxemia in patients with alcoholic and non-alcoholic cirrhosis and in subjects with no evidence of chronic liver disease following acute alcohol excess. , 1987, Journal of hepatology.