Latency : Implications for Immune Control-Herpesvirus γ Early Establishment of Woodland ,

[1]  R. Sun,et al.  Murine Gammaherpesvirus 68 Open Reading Frame 31 Is Required for Viral Replication , 2004, Journal of Virology.

[2]  P. Stevenson,et al.  Murine Gammaherpesvirus 68 Lacking gp150 Shows Defective Virion Release but Establishes Normal Latency In Vivo , 2004, Journal of Virology.

[3]  David C. Gondek,et al.  Different Functional Capacities of Latent and Lytic Antigen-Specific CD8 T Cells in Murine Gammaherpesvirus Infection1 , 2004, The Journal of Immunology.

[4]  P. Moore,et al.  Kaposi's sarcoma-associated herpesvirus immunoevasion and tumorigenesis: two sides of the same coin? , 2003, Annual review of microbiology.

[5]  S. Speck,et al.  Long-Term Latent Murine Gammaherpesvirus 68 Infection Is Preferentially Found within the Surface Immunoglobulin D-Negative Subset of Splenic B Cells In Vivo , 2003, Journal of Virology.

[6]  In-Jeong Kim,et al.  Maintenance of Long Term γ-Herpesvirus B Cell Latency Is Dependent on CD40-Mediated Development of Memory B Cells1 , 2003, The Journal of Immunology.

[7]  H. Virgin,et al.  Establishment and Maintenance of Gammaherpesvirus Latency Are Independent of Infective Dose and Route of Infection , 2003, Journal of Virology.

[8]  In-Jeong Kim,et al.  Differential γ-Herpesvirus Distribution in Distinct Anatomical Locations and Cell Subsets During Persistent Infection in Mice1 , 2003, The Journal of Immunology.

[9]  P. Stevenson,et al.  Murine Gammaherpesvirus 68 Lacking Thymidine Kinase Shows Severe Attenuation of Lytic Cycle Replication In Vivo but Still Establishes Latency , 2003, Journal of Virology.

[10]  In-Jeong Kim,et al.  γ-Herpesvirus Latency Is Preferentially Maintained in Splenic Germinal Center and Memory B Cells , 2002, The Journal of experimental medicine.

[11]  H. Virgin,et al.  Antibody to a Lytic Cycle Viral Protein Decreases Gammaherpesvirus Latency in B-Cell-Deficient Mice , 2002, Journal of Virology.

[12]  J. Yewdell,et al.  Viral interference with antigen presentation , 2002, Nature Immunology.

[13]  U. Koszinowski,et al.  K3-mediated evasion of CD8+ T cells aids amplification of a latent γ-herpesvirus , 2002, Nature Immunology.

[14]  A. Rickinson,et al.  Epitope-specific Evolution of Human CD8+ T Cell Responses from Primary to Persistent Phases of Epstein-Barr Virus Infection , 2002, The Journal of experimental medicine.

[15]  P. Doherty,et al.  Dissecting the host response to a gamma-herpesvirus. , 2001, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.

[16]  P. Murphy.  Viral exploitation and subversion of the immune system through chemokine mimicry , 2001, Nature Immunology.

[17]  H. Virgin,et al.  Ongoing Viral Replication Is Required for Gammaherpesvirus 68-Induced Vascular Damage , 2000, Journal of Virology.

[18]  Antonio Alcami,et al.  Viral mechanisms of immune evasion , 2000, Immunology Today.

[19]  J. Cohen,et al.  Epstein-Barr virus infection. , 2000, The New England journal of medicine.

[20]  J. Sample,et al.  Latent Murine γ-Herpesvirus Infection Is Established in Activated B Cells, Dendritic Cells, and Macrophages1 , 2000, The Journal of Immunology.

[21]  W. Halford,et al.  Optimized Viral Dose and Transient Immunosuppression Enable Herpes Simplex Virus ICP0-Null Mutants To Establish Wild-Type Levels of Latency In Vivo , 2000, Journal of Virology.

[22]  A. Rickinson,et al.  T-cell memory: lessons from Epstein-Barr virus infection in man. , 2000, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.

[23]  A. Rickinson,et al.  Epstein–Barr virus: Co-opting B-cell memory and migration , 2000, Current Biology.

[24]  M. Blackman,et al.  T-Cell Vaccination Alters the Course of Murine Herpesvirus 68 Infection and the Establishment of Viral Latency in Mice , 1999, Journal of Virology.

[25]  D. Thorley-Lawson,et al.  A model for persistent infection with Epstein-Barr virus: the stealth virus of human B cells. , 1999, Life sciences.

[26]  J. Altman,et al.  A gamma-herpesvirus sneaks through a CD8(+) T cell response primed to a lytic-phase epitope. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[27]  H. Virgin,et al.  Macrophages Are the Major Reservoir of Latent Murine Gammaherpesvirus 68 in Peritoneal Cells , 1999, Journal of Virology.

[28]  Carl Liu,et al.  Three Distinct Regions of the Murine Gammaherpesvirus 68 Genome Are Transcriptionally Active in Latently Infected Mice , 1999, Journal of Virology.

[29]  E. Usherwood,et al.  Lung Epithelial Cells Are a Major Site of Murine Gammaherpesvirus Persistence , 1998, Journal of Experimental Medicine.

[30]  P. Stevenson,et al.  Long-Term Persistence of Activated Cytotoxic T Lymphocytes after Viral Infection of the Central Nervous System , 1998, The Journal of experimental medicine.

[31]  E. De Clercq,et al.  In Vitro and In Vivo Inhibition of Murine Gamma Herpesvirus 68 Replication by Selected Antiviral Agents , 1998, Antimicrobial Agents and Chemotherapy.

[32]  R. Burger,et al.  An Immunosuppressed Mouse Model of Lethal Murine Gammaherpesvirus 68 Infection for Studying Potential Treatment of Epstein—Barr Virus Infection in Man , 1997 .

[33]  P. Latreille,et al.  Copyright © 1997, American Society for Microbiology Complete Sequence and Genomic Analysis of Murine , 1997 .

[34]  R. Ahmed,et al.  Regression of papillomas induced by cottontail rabbit papillomavirus is associated with infiltration of CD8+ cells and persistence of viral DNA after regression , 1997, Journal of virology.

[35]  J. Stewart,et al.  Characterization of tumor cell lines derived from murine gammaherpesvirus-68-infected mice , 1996, Journal of virology.

[36]  D. Thorley-Lawson,et al.  Detection of the latent form of Epstein-Barr virus DNA in the peripheral blood of healthy individuals , 1996, Journal of virology.

[37]  L. Young,et al.  Epstein-Barr virus latency in blood mononuclear cells: analysis of viral gene transcription during primary infection and in the carrier state , 1994, Journal of virology.

[38]  A. Nash,et al.  The Effect of Acyclovir on the Acute and Latent Murine Gammaherpesvirus-68 Infection of Mice , 1994 .

[39]  S. Jonjić,et al.  The conditions of primary infection define the load of latent viral genome in organs and the risk of recurrent cytomegalovirus disease , 1994, The Journal of experimental medicine.

[40]  U. Nater,et al.  Epstein-Barr virus. , 1991, The Journal of family practice.

[41]  D. Thorley-Lawson,et al.  B cell activation and the establishment of Epstein-Barr virus latency , 1988, The Journal of experimental medicine.

[42]  T. Gardella,et al.  Detection of circular and linear herpesvirus DNA molecules in mammalian cells by gel electrophoresis , 1984, Journal of virology.

[43]  M. Blackman,et al.  Persistent (cid:2) -herpesvirus Infections: What Can We Learn from an Experimental Mouse Model? , 2002 .

[44]  D. Thorley-Lawson,et al.  Detection and discrimination of latent and replicative herpesvirus infection at the single cell level in vivo. , 2001, Methods in molecular biology.

[45]  In-Jeong Kim,et al.  Vaccination Against Murine γ-Herpesvirus Infection , 2001 .

[46]  H. Ploegh,et al.  Viral subversion of the immune system. , 2000, Annual review of immunology.

[47]  J. Stewart,et al.  Murine gamma-herpesvirus 68 glycoprotein 150 protects against virus-induced mononucleosis: a model system for gamma-herpesvirus vaccination. , 1999, Vaccine.

[48]  P. Doherty,et al.  Progressive Loss of Cd8 + T Cell-mediated Control of a ~/-herpesvirus in the Absence of Cd4 + T Cells , 1996 .

[49]  E. Kieff.  Epstein-Barr virus and its replication , 1996 .