The significance of biochemical and structural changes in the development of the myocardiopathy of the Syrian hamster.

[1]  A. Lochner,et al.  Contractility and tension development of the myopathic hamster (Bio 14-6) heart. , 1969, Cardiovascular research.

[2]  J. R. Baker,et al.  SPONTANEOUS, HEREDITARY MYOCARDIAL DEGENERATION AND CONGESTIVE HEART FAILURE IN A STRAIN OF SYRIAN HAMSTERS * , 1969, Annals of the New York Academy of Sciences.

[3]  L. Sordahl,et al.  Reevaluation of Oxidative Phosphorylation in Cardiac Mitochondria from Normal Animals and Animals in Heart Failure , 1968, Circulation research.

[4]  E. Bajusz,et al.  SECTION OF BIOLOGICAL AND MEDICAL SCIENCES: A NEW DISEASE MODEL OF CHRONIC CONGESTIVE HEART FAILURE: STUDIES ON ITS PATHOGENESIS* , 1968 .

[5]  M. Blanchaer,et al.  SECTION OF BIOLOGICAL AND MEDICAL SCIENCES: OXIDATIVE PHOSPHORYLATION BY MITOCHONDRIA ISOLATED FROM HEARTS OF BIO 14.6 MYOPATHIC HAMSTERS*,† , 1968 .

[6]  T. Kelley.  Altered lipoprotein lipase and lipid metabolism in the myocardium of BIO 14.6 myopathic hamsters. , 1968, Transactions of the New York Academy of Sciences.

[7]  A. Schwartz,et al.  SECTION OF BIOLOGICAL AND MEDICAL SCIENCES: RESPIRATORY CONTROL AND CALCIUM TRANSPORT IN HEART MITOCHONDRIA FROM THE CARDIOMYOPATHIC SYRIAN HAMSTER*,† , 1968 .

[8]  M. Blanchaer,et al.  Respiration and oxidative phosphorylation by muscle and heart mitochondria of hamsters with hereditary myocardiopathy and polymyopathy. , 1968, Canadian journal of biochemistry.

[9]  E. Bajusz.  Interrelationships between reparative processes in myocardium and the development of congestive heart failure. , 1968, Revue canadienne de biologie.

[10]  A. Lochner,et al.  Oxidative phosphorylation and glycolysis in the hereditary muscular dystrophy of the Syrian hamster. , 1967, Clinical science.

[11]  E. Braunwald,et al.  Normal Oxidative Phosphorylation in Mitochondria from the Failing Heart , 1967 .

[12]  E. Braunwald,et al.  Myocardial High Energy Phosphate Stores in Cardiac Hypertrophy and Heart Failure , 1967, Circulation research.

[13]  A. Lochner,et al.  Work Performance of the Isolated Perfused Beating Heart in the Hereditary Myocardiopathy of the Syrian Hamster , 1967, Circulation research.

[14]  J. R. Baker,et al.  HEREDITARY MYOPATHY IN THE SYRIAN HAMSTER: STUDIES ON PATHOGENESIS * , 1966, Annals of the New York Academy of Sciences.

[15]  L. Opie.  Effect of extracellular pH on function and metabolism of isolated perfused rat heart. , 1965, The American journal of physiology.

[16]  L. Opie.  Coronary flow rate and perfusion pressure as determinants of mechanical function and oxidative metabolism of isolated perfused rat heart. , 1965, The Journal of physiology.

[17]  L. Opie,et al.  HYPERTHERMIC DAMAGE TO ISOLATED RAT HEART TISSUE. , 1965, Clinical science.

[18]  G. Reed,et al.  HIGH ENERGY PHOSPHATE COMPOUNDS IN THE MYOCARDIUM DURING EXPERIMENTAL CONGESTIVE HEART FAILURE. PURINE AND PYRIMIDINE NUCLEOTIDES, CREATINE, AND CREATINE PHOSPHATE IN NORMAL AND IN FAILING HEARTS. , 1965, The Journal of clinical investigation.

[19]  L. Opie,et al.  OXIDATIVE PHOSPHORYLATION IN HEREDITARY MYOCARDIOPATHY IN THE SYRIAN HAMSTER. , 1964, Lancet.

[20]  Olson Re.  ABNORMALITIES OF MYOCARDIAL METABOLISM. , 1964 .

[21]  E. Sonnenblick,et al.  Comparison of the Ultrastructural Basis of the Contractile Process in Heart and Skeletal Muscle , 1964, Circulation research.

[22]  J. Baker,et al.  Serum Phosphocreatine Kinase in Hereditary Muscular Dystrophy and Cardiac Necrosis of Syrian Golden Hamsters.∗ , 1964, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[23]  M. Argus,et al.  Oxidative Rates and Phosphorylation in Sarcosomes from Experimentally-Induced Failing Rat Heart.∗ , 1964, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[24]  A. Wollenberger,et al.  Some metabolic characteristics of mitochondria from chronically overloaded, hypertrophied hearts. , 1963, Experimental and molecular pathology.

[25]  M. Feinstein,et al.  Effects of Experimental Congestive Heart Failure, Ouabain, and Asphyxia on the High‐Energy Phosphate and Creatine Content of the Guinea Pig Heart , 1962, Circulation research.

[26]  K. S. Lee,et al.  Study of Heart Mitochondria and Glycolytic Metabolism in Experimentally Induced Cardiac Failure , 1962, Circulation research.

[27]  R. Furchgott,et al.  High Energy Phosphates and the Force of Contraction of Cardiac Muscle , 1961, Circulation.

[28]  N. M. Buckley,et al.  Cardiac Nucleotides and Derivatives in Acute and Chronic Ventricular Failure of the Dog Heart , 1961 .

[29]  M. Gertler,et al.  OXIDATIVE PHOSPHORYLATION STUDIES IN NORMAL AND EXPERIMENTALLY PRODUCED CONGESTIVE HEART FAILURE IN GUINEA PIGS: A COMPARISON , 1959, Annals of the New York Academy of Sciences.

[30]  E. Braunwald,et al.  Hemodynamic determinants of oxygen consumption of the heart with special reference to the tension-time index. , 1957, The American journal of physiology.

[31]  H. Perry,et al.  A Journal of the American -heart Association Studies on the Control of Hypertension by Hyphex v. Effects on the Course of the Malignant Stage , 2022 .

[32]  E. C. Slater,et al.  Respiratory granules of heart muscle. , 1953, The Biochemical journal.

[33]  E. C. Slater.  A method of measuring the yield of oxidative phosphorylation. , 1953 .

[34]  W. Schwartz,et al.  MYOCARDIAL METABOLISM IN CONGESTIVE HEART FAILURE , 1951, Journal of chronic diseases.

[35]  C. H. Fiske,et al.  THE COLORIMETRIC DETERMINATION OF PHOSPHORUS , 1925 .

[36]  C. Chidsey,et al.  Biochemical studies of energy production in the failing human heart. , 1966, The Journal of clinical investigation.

[37]  E. C. Slater,et al.  STUDIES IN VITRO ON SINGLE BEATING HEART CELLS. 8. THE EFFECT OF OLIGOMYCIN, DINITROPHENOL, AND OUABAIN ON THE BEATING RATE. , 1965, Biochimica et biophysica acta.

[38]  F. Homburger,et al.  THE EARLY HISTOPATHOLOGICAL LESION OF MUSCULAR DYSTROPHY IN THE SYRIAN GOLDEN HAMSTER. , 1965, The Journal of pathology and bacteriology.