Application of Periocheck to Periodontal Therapy. II. Comparison of Treatments after Gingival Curettage.

Periocheck(R) is a chairside colorimetric bacteriological diagnostic kit that measures peptidases from Treponema denticola, Porphyromonas gingivalis and Bacteroides forsythus. Fifty-eight periodontitis -affected sites (47 sites in posterior teeth , 11 sites in anterior teeth) in 31 patients showing Periocheck activity two weeks after gingival curettage were subjected to intrapocket antibiotic therapy (IPAT), gingival curettage or were left untreated. The IPAT sites were treated with Periocline(R) once a week for 4 weeks and the curettage sites were subjected to a second gingival curettage, while the control sites were left untreated. All sites were then subjected to Periocheck 6, 10, 14 and 18 weeks after treatment. The results at 18 weeks disclosed a smaller Periocheck-positive fraction at IPAT sites (27%) than after curettage (58%), coinciding with the occurrence of BOP. This demonstrates that IPAT is more effective than a curettage. Because Periocheck-positive fractions tended to increase during the period from 6 to 18 weeks, our findings underline the necessity of periodic postoperative bacteriological checking, in which Periocheck should be practical and useful.

[1]  J. Lindhe,et al.  The effect of supragingival plaque control on the subgingival microbiota in subjects with periodontal disease. , 1992, Journal of clinical periodontology.

[2]  R. Attström,et al.  The effect of supragingival plaque control on the composition of the subgingival flora in periodontal pockets. , 1992, Journal of clinical periodontology.

[3]  T. Kato,et al.  A sensitive enzymatic method (SK-013) for detection and quantification of specific periodontopathogens. , 1992, Journal of periodontal research.

[4]  J. Moriarty,et al.  Closed versus open debridement of facial grade II molar furcations. , 1991, Journal of clinical periodontology.

[5]  A. Jacobson Proceedings of the World Workshop in Clinical Periodontics Princeton. American Academy of Periodontology (1989) , 1990 .

[6]  S. Socransky,et al.  The predominant cultivable microbiota of active and inactive lesions of destructive periodontal diseases. , 1988, Journal of clinical periodontology.

[7]  J. Egelberg,et al.  The effect of plaque control and root debridement in molar teeth. , 1987, Journal of clinical periodontology.

[8]  P. Baehni,et al.  The effect of supragingival plaque control on the composition of the subgingival microflora in human periodontitis. , 1987, Journal of clinical periodontology.

[9]  N. Lang,et al.  Bleeding on probing. A predictor for the progression of periodontal disease? , 1986, Journal of clinical periodontology.

[10]  J. Hardie,et al.  The effect of supragingival plaque control on the subgingival microflora. , 1985, Journal of clinical periodontology.

[11]  R. McHugh,et al.  Molar and nonmolar teeth compared over 6 1/2 years following two methods of periodontal therapy. , 1984, Journal of periodontology.

[12]  J. Lindhe,et al.  Some microbiological and histopathological features of periodontal disease in man. , 1980, Journal of periodontology.

[13]  S. Ramfjord,et al.  Comparison of results following three modalities of periodontal therapy related to tooth type and initial pocket depth. , 1980, Journal of clinical periodontology.

[14]  H. Löe,et al.  PERIODONTAL DISEASE IN PREGNANCY. II. CORRELATION BETWEEN ORAL HYGIENE AND PERIODONTAL CONDTION. , 1964, Acta odontologica Scandinavica.

[15]  R. C. Bower,et al.  Furcation morphology relative to periodontal treatment. Furcation entrance architecture. , 1979, Journal of periodontology.