Deciphering the dynamic molecular program of radiation-induced endothelial senescence.
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I. Morilla | F. Milliat | V. Paget | M. Benadjaoud | O. Guipaud | M. Mondini | Bruno L'homme | F. Soysouvanh | A. François | V. Buard | G. Tarlet | G. Gruel | Morgane dos Santos | A. Bertho | E. Deutsch | Henrique Santos de Andrade | Bruno L’homme
[1] Stephen L. Brown,et al. Therapy-Induced Senescence: Opportunities to Improve Anticancer Therapy , 2021, Journal of the National Cancer Institute.
[2] D. Bernard,et al. Elimination of Senescent Endothelial Cells: Good or Bad Idea? , 2021, Trends in cell biology.
[3] K. Kwon,et al. CD9 induces cellular senescence and aggravates atherosclerotic plaque formation , 2020, Cell Death & Differentiation.
[4] E. Deutsch,et al. Radiotherapy–immunotherapy combinations – perspectives and challenges , 2020, Molecular oncology.
[5] W. Quax,et al. Regulation of Survival Networks in Senescent Cells: From Mechanisms to Interventions. , 2019, Journal of molecular biology.
[6] R. Baumgartner,et al. Prevention of radiotherapy-induced arterial inflammation by interleukin-1 blockade , 2019, European heart journal.
[7] Yun Wang,et al. Protective effect of rapamycin on endothelial-to-mesenchymal transition in HUVECs through the Notch signaling pathway. , 2019, Vascular pharmacology.
[8] F. Milliat,et al. Lung Stereotactic Arc Therapy in Mice: Development of Radiation Pneumopathy and Influence of HIF-1α Endothelial Deletion. , 2019, International journal of radiation oncology, biology, physics.
[9] J. Mallm,et al. HMGB2 Loss upon Senescence Entry Disrupts Genomic Organization and Induces CTCF Clustering across Cell Types. , 2018, Molecular cell.
[10] F. Milliat,et al. The importance of the vascular endothelial barrier in the immune-inflammatory response induced by radiotherapy. , 2018, The British journal of radiology.
[11] J. Gil,et al. Mechanisms and functions of cellular senescence. , 2018, The Journal of clinical investigation.
[12] E. O'Duibhir,et al. Paracrine cellular senescence exacerbates biliary injury and impairs regeneration , 2018, Nature Communications.
[13] M. Iruela-Arispe,et al. HIF-1α Deletion in the Endothelium, but Not in the Epithelium, Protects From Radiation-Induced Enteritis , 2017, Cellular and molecular gastroenterology and hepatology.
[14] D. Citrin,et al. Inhibition of Bcl-2/xl With ABT-263 Selectively Kills Senescent Type II Pneumocytes and Reverses Persistent Pulmonary Fibrosis Induced by Ionizing Radiation in Mice. , 2017, International journal of radiation oncology, biology, physics.
[15] S. Melov,et al. Unmasking Transcriptional Heterogeneity in Senescent Cells , 2017, Current Biology.
[16] M. Iruela-Arispe,et al. Endothelial Hey2 deletion reduces endothelial-to-mesenchymal transition and mitigates radiation proctitis in mice , 2017, Scientific Reports.
[17] F. Paris,et al. Ionizing radiation induces long‐term senescence in endothelial cells through mitochondrial respiratory complex II dysfunction and superoxide generation , 2017, Free radical biology & medicine.
[18] L. Zender,et al. The senescence-associated secretory phenotype induces cellular plasticity and tissue regeneration , 2017, Genes & development.
[19] James B. Mitchell,et al. Mammalian Target of Rapamycin Inhibition With Rapamycin Mitigates Radiation-Induced Pulmonary Fibrosis in a Murine Model. , 2016, International journal of radiation oncology, biology, physics.
[20] M. Boerma,et al. Ionizing Radiation-Induced Endothelial Cell Senescence and Cardiovascular Diseases , 2016, Radiation Research.
[21] M. Iruela-Arispe,et al. In vivo evidence for an endothelium-dependent mechanism in radiation-induced normal tissue injury , 2015, Scientific Reports.
[22] F. Milliat,et al. Identification of Endothelial-to-Mesenchymal Transition as a Potential Participant in Radiation Proctitis. , 2015, The American journal of pathology.
[23] M. Humbert,et al. Endothelial-to-Mesenchymal Transition in Pulmonary Hypertension , 2015, Circulation.
[24] J. Hoeijmakers,et al. An essential role for senescent cells in optimal wound healing through secretion of PDGF-AA. , 2014, Developmental cell.
[25] D. Harats,et al. Interleukin-1 deficiency prolongs ovarian lifespan in mice , 2014, Proceedings of the National Academy of Sciences.
[26] K. Raj,et al. Premature aging induced by radiation exhibits pro-atherosclerotic effects mediated by epigenetic activation of CD44 expression , 2014, Aging cell.
[27] F. Milliat,et al. PAI-1-Dependent Endothelial Cell Death Determines Severity of Radiation-Induced Intestinal Injury , 2012, PloS one.
[28] C. A. de la Motte,et al. Inflammation-induced endothelial-to-mesenchymal transition: a novel mechanism of intestinal fibrosis. , 2011, The American journal of pathology.
[29] R. Sen,et al. Faculty Opinions recommendation of Cell surface-bound IL-1alpha is an upstream regulator of the senescence-associated IL-6/IL-8 cytokine network. , 2010 .
[30] J. Campisi,et al. Protocols to detect senescence-associated beta-galactosidase (SA-βgal) activity, a biomarker of senescent cells in culture and in vivo , 2009, Nature Protocols.
[31] F. Milliat,et al. Effects of pharmacological inhibition and genetic deficiency of plasminogen activator inhibitor-1 in radiation-induced intestinal injury. , 2009, International journal of radiation oncology, biology, physics.
[32] D. Peeper,et al. Senescence-messaging secretome: SMS-ing cellular stress , 2009, Nature Reviews Cancer.
[33] J. Erusalimsky. Vascular endothelial senescence: from mechanisms to pathophysiology. , 2009, Journal of applied physiology.
[34] E. Deutsch,et al. Gastrointestinal , Hepatobiliary and Pancreatic Pathology Essential Role of Plasminogen Activator Inhibitor Type-1 in Radiation Enteropathy , 2010 .
[35] J. Erusalimsky. From Mechanisms to Pathophysiology , 2008 .
[36] Xueli Yuan,et al. Endothelial-to-mesenchymal transition contributes to cardiac fibrosis , 2007, Nature Medicine.
[37] Zvi Fuks,et al. Endothelial Apoptosis as the Primary Lesion Initiating Intestinal Radiation Damage in Mice , 2001, Science.