Erythrocyte glycohydrolases in subjects with trisomy 21: Could Down's syndrome be a model of accelerated ageing?

[1]  M. Rondanelli,et al.  Erythrocyte membrane alterations during ageing affect β-d-glucuronidase and neutral sialidase in elderly healthy subjects , 2005, Experimental Gerontology.

[2]  F. Licastro,et al.  Altered Cytokine and Acute Phase Response Protein Levels in the Blood of Children with Downs Syndrome: Relationship with Dementia of Alzheimer's Type , 2005, International journal of immunopathology and pharmacology.

[3]  G. Cervato,et al.  Biochemical assessments of oxidative stress, erythrocyte membrane fluidity and antioxidant status in professional soccer players and sedentary controls , 2003, European journal of clinical investigation.

[4]  R. Wersto,et al.  Red cell perturbations by amyloid β-protein , 2003 .

[5]  M. Maggiora,et al.  Effect of 4‐hydroxynonenal, a lipid peroxidation product, on exocytosis in HL‐60 cells , 2002, Cell biochemistry and function.

[6]  A. Manto,et al.  Early increase of oxidative stress and reduced antioxidant defenses in patients with uncomplicated type 1 diabetes: a case for gender difference. , 2002, Diabetes care.

[7]  L. Mazzanti,et al.  Acidic and neutral sialidase in the erythrocyte membrane of type 2 diabetic patients. , 2002, Blood.

[8]  G. Hart,et al.  O-Glycosylation of Nuclear and Cytosolic Proteins , 2000, The Journal of Biological Chemistry.

[9]  L. Massaccesi,et al.  Membrane anchoring and surface distribution of glycohydrolases of human erythrocyte membranes , 2000, FEBS letters.

[10]  L. Mazzanti,et al.  ERYTHROCYTE PLASMA MEMBRANES OBTAINED FROM CENTENARIANS SHOW DIFFERENT FUNCTIONAL PROPERTIES , 2000, Journal of the American Geriatrics Society.

[11]  A. Burlina,et al.  Alterations in the activity of several glycohydrolases in red blood cell membrane from type 2 diabetes mellitus patients. , 1999, Metabolism: clinical and experimental.

[12]  P. Viani,et al.  A fluorescence method for the determination of plasma susceptibility to lipid peroxidation. , 1999, Clinical biochemistry.

[13]  D. McClain,et al.  Insulin and glucosamine infusions increase O-linked N-acetyl-glucosamine in skeletal muscle proteins in vivo. , 1998, Metabolism: clinical and experimental.

[14]  P. Coleman,et al.  Reduction of O-Linked N-Acetylglucosamine-Modified Assembly Protein-3 in Alzheimer’s Disease , 1998, The Journal of Neuroscience.

[15]  M. Smith,et al.  Early glycoxidation damage in brains from Down's syndrome. , 1998, Biochemical and biophysical research communications.

[16]  N. Cairns,et al.  Evidence against increased oxidative DNA-damage in Down syndrome , 1997, Neuroscience Letters.

[17]  A. Fiorilli,et al.  Presence in human erythrocyte membranes of a novel form of sialidase acting optimally at neutral pH. , 1997, Blood.

[18]  B. Yankner,et al.  Apoptosis and increased generation of reactive oxygen species in Down's syndrome neurons in vitro , 1995, Nature.

[19]  M. Mathes,et al.  β‐Amyloid precursor protein is modified with O‐linked N‐acetylglucosamine , 1995, Journal of neuroscience research.

[20]  G. Hart,et al.  Purification and characterization of an O-GlcNAc selective N-acetyl-beta-D-glucosaminidase from rat spleen cytosol. , 1994, The Journal of biological chemistry.

[21]  K. Emerk,et al.  Simple colorimetric method for determination of peroxide. , 1993, Clinical chemistry.

[22]  G. Bosman,et al.  Erythrocyte membrane changes of individuals with Down's Syndrome in various stages of Alzheimer-type dementia , 1993, Neurobiology of Aging.

[23]  L. Mazzanti,et al.  Effects of diabetes mellitus on structural and functional properties of erythrocyte membranes. , 1993, Membrane biochemistry.

[24]  G. Bosman,et al.  Erythrocyte membrane characteristics indicate abnormal cellular aging in patients with Alzheimer's disease , 1991, Neurobiology of Aging.

[25]  S. Incerpi,et al.  Membrane Lipid Alterations and Na+-Pumping Activity in Erythrocytes From IDDM and NIDDM Subjects , 1989, Diabetes.

[26]  M. Stringer,et al.  Lipid peroxides and atherosclerosis. , 1989, BMJ.

[27]  D. Mann,et al.  The pattern of acquisition of plaques and tangles in the brains of patients under 50 years of age with Down's syndrome , 1989, Journal of the Neurological Sciences.

[28]  A. Sadovnick,et al.  Causes of death to age 30 in Down syndrome. , 1988, American journal of human genetics.

[29]  G. Hart,et al.  Erythrocytes contain cytoplasmic glycoproteins. O-linked GlcNAc on Band 4.1. , 1987, The Journal of biological chemistry.

[30]  H. Wiśniewski,et al.  Occurrence of neuropathological changes and dementia of Alzheimer's disease in Down's syndrome , 1985, Annals of neurology.

[31]  G. Bartosz,et al.  Aging of the erythrocyte. XX. Decreased red cell life span in Down's syndrome. , 1982, Cell biology international reports.

[32]  P. Sinet.  Metabolism of Oxygen Derivatives in Down's Syndrome * , 1982, Annals of the New York Academy of Sciences.

[33]  G. Mariani,et al.  Human erythrocyte separation according to age on a discontinuous "Percoll" density gradient. , 1982, Clinica chimica acta; international journal of clinical chemistry.

[34]  L. Caimi,et al.  Enzymes of lysosomal origin in human plasma and serum: assay conditions and parameters influencing the assay. , 1980, Clinica chimica acta; international journal of clinical chemistry.

[35]  L. Caimi,et al.  Optimal conditions for the assay of fibroblast neuraminidase with different natural substrates. , 1979, Biochimica et biophysica acta.

[36]  J. Fehr,et al.  Comparison of red cell creatine level and reticulocyte count in appraising the severity of hemolytic processes. , 1979, Blood.

[37]  J. Kaplan,et al.  International Committee for Standardization in Haematology: Recommended Methods for Red‐Cell Enzyme Analysis * , 1977, British journal of haematology.

[38]  M. M. Bradford.  A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.

[39]  J. Sever,et al.  EPIDEMIOLOGY OF MONGOLISM IN THE COLLABORATIVE PROJECT * † , 1970 .

[40]  Oliver H. Lowry,et al.  Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.

[41]  M. Corsi,et al.  Reactive oxygen metabolites and prooxidant status in children with Down's syndrome. , 2001, International journal of clinical pharmacology research.

[42]  I. Kola,et al.  Reactive oxygen species and their contribution to pathology in Down syndrome. , 1997, Advances in pharmacology.

[43]  A. Kantar,et al.  Alterations in erythrocyte membrane fluidity in children with trisomy 21: a fluorescence study , 1992, Biology of the cell.

[44]  A. Mosca,et al.  Clinical utility of fractionating erythrocytes into "Percoll" density gradients. , 1991, Advances in experimental medicine and biology.

[45]  H. Goulet,et al.  Fluorescence studies on aged and young erythrocyte populations. , 1991, Cellular and molecular biology.

[46]  C. Jeandel,et al.  Lipid peroxidation and free radical scavengers in Alzheimer's disease. , 1989, Gerontology.

[47]  D. Segre,et al.  Immunological aspects of aging , 1981 .

[48]  T. Steck,et al.  Preparation of impermeable ghosts and inside-out vesicles from human erythrocyte membranes. , 1974, Methods in enzymology.