Cardiotrophin-1 protects the human myocardium from ischemic injury. Comparison with the first and second window of protection by ischemic preconditioning.

[1]  G. Ertl,et al.  Experimental heart failure in rats: effects on cardiovascular circadian rhythms and on myocardial beta-adrenergic signaling. , 2000, Cardiovascular research.

[2]  N. Standen,et al.  Evidence for mitochondrial K ATP channels as effectors of human myocardial preconditioning. , 2000, Cardiovascular research.

[3]  N. Standen,et al.  Preconditioning the human myocardium by simulated ischemia: studies on the early and delayed protection. , 2000, Cardiovascular research.

[4]  Jiahuai Han,et al.  PKC-dependent activation of p46/p54 JNKs during ischemic preconditioning in conscious rabbits. , 1999, American journal of physiology. Heart and circulatory physiology.

[5]  Y. Fujio,et al.  Hypoxic stress induces cardiotrophin-1 expression in cardiac myocytes. , 1999, Biochemical and biophysical research communications.

[6]  J. Davies,et al.  An immunoluminometric assay for cardiotrophin-1: a newly identified cytokine is present in normal human plasma and is increased in heart failure. , 1999, Biochemical and biophysical research communications.

[7]  D. Latchman.  Cardiotrophin‐1 (CT‐1): a novel hypertrophic and cardioprotective agent , 1999, International journal of experimental pathology.

[8]  D. Yellon,et al.  Myocardial adaptation to ischaemia--the preconditioning phenomenon. , 1999, International journal of cardiology.

[9]  Beranek Jt.  Why growth hormone therapy is efficient in heart failure , 1999 .

[10]  E. Marbán,et al.  Modulation of mitochondrial ATP-dependent K+ channels by protein kinase C. , 1998, Circulation research.

[11]  Yongge Liu,et al.  Mitochondrial ATP-dependent potassium channels: novel effectors of cardioprotection? , 1998, Circulation.

[12]  J. Levasseur,et al.  Dissociation of heat shock proteins expression with ischemic tolerance by whole body hyperthermia in rat heart. , 1998, Journal of molecular and cellular cardiology.

[13]  A. Stephanou,et al.  Cardiotrophin-1 induces heat shock protein accumulation in cultured cardiac cells and protects them from stressful stimuli. , 1998, Journal of molecular and cellular cardiology.

[14]  K. Chien,et al.  Cardiotrophin 1 (CT-1) Inhibition of Cardiac Myocyte Apoptosis via a Mitogen-activated Protein Kinase-dependent Pathway , 1997, The Journal of Biological Chemistry.

[15]  S. Chevalier,et al.  Signaling of the Cardiotrophin-1 Receptor , 1997, The Journal of Biological Chemistry.

[16]  A. Ashworth,et al.  Stimulation of the stress-activated mitogen-activated protein kinase subfamilies in perfused heart. p38/RK mitogen-activated protein kinases and c-Jun N-terminal kinases are activated by ischemia/reperfusion. , 1996, Circulation research.

[17]  J. Heath,et al.  Cardiotrophin-1 Activates a Distinct Form of Cardiac Muscle Cell Hypertrophy , 1996, The Journal of Biological Chemistry.

[18]  D. Buxton,et al.  Stimulation of c-Jun kinase and mitogen-activated protein kinase by ischemia and reperfusion in the perfused rat heart. , 1996, Biochemical and biophysical research communications.

[19]  W. Wood,et al.  Cardiotrophin-1 , 1995, The Journal of Biological Chemistry.

[20]  K. Chien,et al.  Expression cloning of cardiotrophin 1, a cytokine that induces cardiac myocyte hypertrophy. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[21]  R. Jennings,et al.  Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium. , 1986, Circulation.