Guilt by association: HLA-B27 and ankylosing spondylitis.

[1]  M. A. Saper,et al.  Specificity pockets for the side chains of peptide antigens in HLA-Aw68 , 1990, Nature.

[2]  J. Castro.  HLA-B27 and HLA-A2 subtypes: structure, evolution and function , 1989 .

[3]  H. Rammensee,et al.  Clonal anergy induced in mature Vβ6+ T lymphocytes on immunizing Mls-1b mice with Mls-1a expressing cells , 1989, Nature.

[4]  J. Levy,et al.  Physical association between MHC class I molecules and immunogenic peptides , 1989, Nature.

[5]  H. Waldmann,et al.  Induction of classical transplantation tolerance in the adult , 1989, The Journal of experimental medicine.

[6]  P. Parham,et al.  Direct binding of influenza peptides to class I HLA molecules , 1989, Nature.

[7]  H. von Boehmer,et al.  The thymus selects the useful, neglects the useless and destroys the harmful. , 1989, Immunology today.

[8]  J. Edmonds,et al.  Cross-reacting bacterial determinants in ankylosing spondylitis. , 1988, The American journal of medicine.

[9]  J. Reveille,et al.  Restriction fragment length polymorphism analysis in ankylosing spondylitis. , 1988, The American journal of medicine.

[10]  F. El-Zaatari,et al.  In vitro mutagenesis of HLA-B27. Substitution of an unpaired cysteine residue in the alpha 1 domain causes loss of antibody-defined epitopes. , 1988, The Journal of clinical investigation.

[11]  J. H. Chen,et al.  A Yersinia pseudotuberculosis protein which cross-reacts with HLA-B27. , 1987, Journal of immunology.

[12]  M. A. Saper,et al.  Structure of the human class I histocompatibility antigen, HLA-A2 , 1987, Nature.

[13]  A. Berns,et al.  HLA-restricted recognition of viral antigens in HLA transgenic mice , 1987, Nature.

[14]  M. Oldstone.  Molecular mimicry and autoimmune disease , 1987, Cell.

[15]  P. Schwimmbeck,et al.  Autoantibodies to HLA B27 in the sera of HLA B27 patients with ankylosing spondylitis and Reiter's syndrome. Molecular mimicry with Klebsiella pneumoniae as potential mechanism of autoimmune disease , 1987, The Journal of experimental medicine.

[16]  P. Iványi,et al.  Distribution of HLA-B27 subtypes in patients with ankylosing spondylitis: the disease is associated with a common determinant of the various B27 molecules. , 1987, Annals of the rheumatic diseases.

[17]  T. Glant,et al.  Proteoglycan-induced arthritis in BALB/c mice. Clinical features and histopathology. , 1987, Arthritis and rheumatism.

[18]  E. Unanue,et al.  Identification of the T-cell and Ia contact residues of a T-cell antigenic epitope , 1987, Nature.

[19]  J. Edmonds,et al.  Cytotoxic T lymphocytes against disease-associated determinant(s) in ankylosing spondylitis , 1986, The Journal of experimental medicine.

[20]  A. Keat.  Is spondylitis caused by Klebsiella? , 1986, Immunology today.

[21]  R. Knobler,et al.  Encephalitogenic T cell clones specific for myelin basic protein. An unusual bias in antigen recognition , 1985, The Journal of experimental medicine.

[22]  J. Woody,et al.  Primary structure of papain-solubilized human histocompatibility antigen HLA-B27. , 1985, Biochemistry.

[23]  B. Benacerraf,et al.  Suppressor cells and immunoregulation. , 1984, Annual review of immunology.

[24]  I. Chalmers,et al.  HLA-B27 haplotypes in family studies of ankylosing spondylitis. , 1983, Arthritis and rheumatism.

[25]  A. McMichael,et al.  Recognition of HLA-B27 and related antigen by a monoclonal antibody. , 1982, Human immunology.

[26]  J. Edmonds,et al.  A factor(s) in Klebsiella culture filtrates specifically modifies an HLA-B27-associated cell-surface component , 1980, Nature.

[27]  R D Sturrock,et al.  Ankylosing spondylitis and HL-A 27. , 1973, Lancet.

[28]  P I Terasaki,et al.  High association of an HL-A antigen, W27, with ankylosing spondylitis. , 1973, The New England journal of medicine.