NLRP3 inflammasome-mediated Pyroptosis induce Notch signal activation in endometriosis angiogenesis

[1]  Shaparak Najibi,et al.  Focus on the role of NLRP3 inflammasome in the pathology of endometriosis: a review on molecular mechanisms and possible medical applications , 2022, Naunyn-Schmiedeberg's Archives of Pharmacology.

[2]  Yi Huang,et al.  PEG2-Induced Pyroptosis Regulates the Expression of HMGB1 and Promotes hEM15A Migration in Endometriosis , 2022, International journal of molecular sciences.

[3]  H. Qi,et al.  TOP2A deficit-induced abnormal decidualization leads to recurrent implantation failure via the NF-κB signaling pathway , 2022, Reproductive Biology and Endocrinology.

[4]  Shiyu Ma,et al.  Berberine alleviates NLRP3 inflammasome induced endothelial junction dysfunction through Ca2+ signalling in inflammatory vascular injury. , 2022, Phytomedicine : international journal of phytotherapy and phytopharmacology.

[5]  Chun Fan,et al.  Isoliquiritigenin alleviates P. gingivalis-LPS/ATP-induced pyroptosis by inhibiting NF-κB/ NLRP3/GSDMD signals in human gingival fibroblasts. , 2021, International immunopharmacology.

[6]  Hua Zhou,et al.  TOB1 modulates the decidualization of human endometrial stromal cells via the Notch pathway , 2021, Journal of Assisted Reproduction and Genetics.

[7]  Libo Zhu,et al.  NLRP3 Inflammasome Activation of Mast Cells by Estrogen via the Nuclear-Initiated Signaling Pathway Contributes to the Development of Endometriosis , 2021, Frontiers in Immunology.

[8]  Jin Huang,et al.  Neuritin promotes angiogenesis through inhibition of DLL4/Notch signaling pathway. , 2021, Acta biochimica et biophysica Sinica.

[9]  Yuli Jin,et al.  E3 ubiquitin ligase TRIM24 deficiency promotes NLRP3/caspase‐1/IL‐1β‐mediated pyroptosis in endometriosis , 2021, Cell biology international.

[10]  F. Di Nicuolo,et al.  Alpha-Lipoic Acid Plays a Role in Endometriosis: New Evidence on Inflammasome-Mediated Interleukin Production, Cellular Adhesion and Invasion , 2021, Molecules.

[11]  D. Lambrechts,et al.  IL1β Promotes Immune Suppression in the Tumor Microenvironment Independent of the Inflammasome and Gasdermin D , 2020, Cancer Immunology Research.

[12]  S. Missmer,et al.  Comparison of cytokines in the peritoneal fluid and conditioned medium of adolescents and adults with and without endometriosis , 2020, American journal of reproductive immunology.

[13]  A. Harris,et al.  RHOQ is induced by DLL4 and regulates angiogenesis by determining the intracellular route of the Notch intracellular domain , 2020, Angiogenesis.

[14]  Yong-mei Song,et al.  Interleukin-6 (IL-6) activates the NOTCH1 signaling pathway through E-proteins in endometriotic lesions. , 2020, The Journal of clinical endocrinology and metabolism.

[15]  Chunjie Fan,et al.  Contribution of TGF-Beta-Mediated NLRP3-HMGB1 Activation to Tubulointerstitial Fibrosis in Rat With Angiotensin II-Induced Chronic Kidney Disease , 2020, Frontiers in Cell and Developmental Biology.

[16]  I. Shih,et al.  The Origin and Pathogenesis of Endometriosis. , 2020, Annual review of pathology.

[17]  Z. Asemi,et al.  The role of inflammation, oxidative stress, angiogenesis, and apoptosis in the pathophysiology of endometriosis: Basic science and new insights based on gene expression , 2019, Journal of cellular physiology.

[18]  P. Santulli,et al.  Rethinking mechanisms, diagnosis and management of endometriosis , 2019, Nature Reviews Endocrinology.

[19]  Yuan He,et al.  The NLRP3 Inflammasome: An Overview of Mechanisms of Activation and Regulation , 2019, International journal of molecular sciences.

[20]  Wen-Jie Zhou,et al.  Anti-inflammatory cytokines in endometriosis , 2019, Cellular and Molecular Life Sciences.

[21]  Yang Zhang,et al.  Aspirin alleviates endothelial gap junction dysfunction through inhibition of NLRP3 inflammasome activation in LPS-induced vascular injury , 2019, Acta pharmaceutica Sinica. B.

[22]  K. Meade,et al.  Non-canonical Inflammasome-Mediated IL-1β Production by Primary Endometrial Epithelial and Stromal Fibroblast Cells Is NLRP3 and Caspase-4 Dependent , 2019, Front. Immunol..

[23]  S. Rath,et al.  A high level of TGF-B1 promotes endometriosis development via cell migration, adhesiveness, colonization, and invasiveness†. , 2018, Biology of reproduction.

[24]  Li Shen,et al.  MicroRNA‐34a promotes CMECs apoptosis and upregulate inflammatory cytokines, thus worsening CMECs damage and inhibiting angiogenesis by negatively targeting the Notch signaling pathway , 2018, Journal of cellular biochemistry.

[25]  N. Galazis,et al.  Vascularization in endometriosis and its clinical applications. , 2018, Human Reproduction Update.

[26]  Ling Zhang,et al.  Notch activity mediates oestrogen-induced stromal cell invasion in endometriosis. , 2018, Reproduction.

[27]  M. Menger,et al.  Notch signaling controls sprouting angiogenesis of endometriotic lesions , 2018, Angiogenesis.

[28]  L. Kiesel,et al.  The endometrial stem cell markers notch-1 and numb are associated with endometriosis. , 2017, Reproductive biomedicine online.

[29]  K. Osteen,et al.  Therapeutically Targeting the Inflammasome Product in a Chimeric Model of Endometriosis-Related Surgical Adhesions , 2017, Reproductive Sciences.

[30]  Rong-zhen Liu,et al.  Lentiviral vector-mediated down-regulation of Notch1 in endometrial stem cells results in proliferation and migration in endometriosis , 2016, Molecular and Cellular Endocrinology.

[31]  Lingli Jiang,et al.  Inflammation and endometriosis. , 2016, Frontiers in bioscience.

[32]  D. Lambrechts,et al.  Vascular endothelial growth factor pathway in endometriosis: genetic variants and plasma biomarkers. , 2016, Fertility and sterility.

[33]  S. Young,et al.  IL-17A Contributes to the Pathogenesis of Endometriosis by Triggering Proinflammatory Cytokines and Angiogenic Growth Factors , 2015, The Journal of Immunology.

[34]  S. Young,et al.  Decreased Notch pathway signaling in the endometrium of women with endometriosis impairs decidualization. , 2015, The Journal of clinical endocrinology and metabolism.

[35]  G. Rabinovich,et al.  Targeting galectin‐1‐induced angiogenesis mitigates the severity of endometriosis , 2014, The Journal of pathology.

[36]  C. Calhaz-Jorge,et al.  Angiogenesis as a therapeutic target in endometriosis. , 2014, Acta medica portuguesa.

[37]  P. Vercellini,et al.  Endometriosis: pathogenesis and treatment , 2014, Nature Reviews Endocrinology.

[38]  M. Mattson,et al.  Evidence that collaboration between HIF-1α and Notch-1 promotes neuronal cell death in ischemic stroke , 2014, Neurobiology of Disease.

[39]  I. Shapiro,et al.  Inflammatory Cytokines Induce NOTCH Signaling in Nucleus Pulposus Cells , 2013, The Journal of Biological Chemistry.

[40]  Robert N. Taylor,et al.  Mechanistic and Therapeutic Implications of Angiogenesis in Endometriosis , 2009, Reproductive sciences.

[41]  U. Lendahl,et al.  Interaction with factor inhibiting HIF-1 defines an additional mode of cross-coupling between the Notch and hypoxia signaling pathways , 2008, Proceedings of the National Academy of Sciences.

[42]  Tae Funakoshi,et al.  2-methoxyestradiol inhibits hypoxia-inducible factor-1{alpha} and suppresses growth of lesions in a mouse model of endometriosis. , 2008, The American journal of pathology.

[43]  R. D'Amato,et al.  Angiogenesis and antiangiogenic therapy in endometriosis. , 2007, Microvascular research.

[44]  U. Lendahl,et al.  Hypoxia requires notch signaling to maintain the undifferentiated cell state. , 2005, Developmental cell.

[45]  T. Okamoto,et al.  Induction of Notch signaling by tumor necrosis factor in rheumatoid synovial fibroblasts , 2003, Oncogene.

[46]  V. Jonušienė,et al.  Notch and Endometrial Cancer. , 2021, Advances in experimental medicine and biology.

[47]  T. Gridley.  Notch signaling in the vasculature. , 2010, Current topics in developmental biology.

[48]  Lei-Chin Chen,et al.  Distinct regulation of cyclooxygenase-2 by interleukin-1beta in normal and endometriotic stromal cells. , 2005, The Journal of clinical endocrinology and metabolism.