Peroxynitrite inhibits myofibrillar protein function in an in vitro assay of motility.
暂无分享,去创建一个
[1] M. Regnier,et al. Myosin S2 is not required for effects of myosin binding protein‐C on motility , 2007, FEBS letters.
[2] Yaqin Xu,et al. Alterations to myofibrillar protein function in nonischemic regions of the heart early after myocardial infarction. , 2007, American journal of physiology. Heart and circulatory physiology.
[3] B. Hambly,et al. Myosin binding protein-C: enigmatic regulator of cardiac contraction. , 2007, The international journal of biochemistry & cell biology.
[4] B. Pesse,et al. Peroxynitrite is a major trigger of cardiomyocyte apoptosis in vitro and in vivo. , 2006, Free radical biology & medicine.
[5] A. Rojas,et al. Oxidative stress at the vascular wall. Mechanistic and pharmacological aspects. , 2006, Archives of medical research.
[6] S. Simão,et al. Inhibition of skeletal muscle S1-myosin ATPase by peroxynitrite. , 2006, Biochemistry.
[7] T. Malinski. Understanding nitric oxide physiology in the heart: a nanomedical approach. , 2005, The American journal of cardiology.
[8] W. Paulus,et al. Peroxynitrite-induced alpha-actinin nitration and contractile alterations in isolated human myocardial cells. , 2005, Cardiovascular research.
[9] E. Moilanen,et al. Nitric oxide production and signaling in inflammation. , 2005, Current drug targets. Inflammation and allergy.
[10] Csaba Szabó,et al. Poly(ADP-ribose) polymerase and the therapeutic effects of its inhibitors , 2005, Nature Reviews Drug Discovery.
[11] D. Allen,et al. Reactive oxygen species reduce myofibrillar Ca2+ sensitivity in fatiguing mouse skeletal muscle at 37°C , 2005 .
[12] D. Allen,et al. Reactive oxygen species reduce myofibrillar Ca2+ sensitivity in fatiguing mouse skeletal muscle at 37 degrees C. , 2005, The Journal of physiology.
[13] J. Pelling,et al. Proteomic identification of 3-nitrotyrosine-containing rat cardiac proteins: effects of biological aging. , 2005, American journal of physiology. Heart and circulatory physiology.
[14] W. Guilford,et al. The tail of myosin reduces actin filament velocity in the in vitro motility assay. , 2004, Cell motility and the cytoskeleton.
[15] S. Thompson,et al. Some precautions in using chelators to buffer metals in biological solutions. , 2004, Cell calcium.
[16] Steven R Tannenbaum,et al. Reactive nitrogen species in the chemical biology of inflammation. , 2004, Archives of biochemistry and biophysics.
[17] J. Sellers,et al. The in vitro motility activity of β-cardiac myosin depends on the nature of the β-myosin heavy chain gene mutation in hypertrophic cardiomyopathy , 1997, Journal of Muscle Research & Cell Motility.
[18] R. K. Wright,et al. Smooth, cardiac and skeletal muscle myosin force and motion generation assessed by cross-bridge mechanical interactions in vitro , 1994, Journal of Muscle Research & Cell Motility.
[19] Kenji Sunagawa,et al. Myosin light chain isoforms modify force-generating ability of cardiac myosin by changing the kinetics of actin-myosin interaction. , 2003, Cardiovascular research.
[20] S. Hazen,et al. Association of nitrotyrosine levels with cardiovascular disease and modulation by statin therapy. , 2003, JAMA.
[21] H. Ischiropoulos,et al. Oxidative stress and nitration in neurodegeneration: cause, effect, or association? , 2003, The Journal of clinical investigation.
[22] J. Bauer,et al. Peroxynitrite-induced inhibition and nitration of cardiac myofibrillar creatine kinase. , 2002, Biochimie.
[23] D. Giustarini,et al. Methionine oxidation as a major cause of the functional impairment of oxidized actin. , 2002, Free radical biology & medicine.
[24] Rifat Hasan,et al. Peroxynitrite oxidation of tubulin sulfhydryls inhibits microtubule polymerization. , 2002, Archives of biochemistry and biophysics.
[25] J. Bauer,et al. Intracellular distribution of peroxynitrite during doxorubicin cardiomyopathy: evidence for selective impairment of myofibrillar creatine kinase , 2002, British journal of pharmacology.
[26] B Balachandran,et al. Role of Oxidative Stress and Antioxidants in Neurodegenerative Diseases , 2002, Nutritional neuroscience.
[27] D. Giustarini,et al. Actin carbonylation: from a simple marker of protein oxidation to relevant signs of severe functional impairment. , 2001, Free radical biology & medicine.
[28] I. Kulikovskaya,et al. Changes in cardiac contractility related to calcium-mediated changes in phosphorylation of myosin-binding protein C. , 2001, Biophysical journal.
[29] D. V. Van Wagoner,et al. Impaired Myofibrillar Energetics and Oxidative Injury During Human Atrial Fibrillation , 2001, Circulation.
[30] S. Powell,et al. Actin is oxidized during myocardial ischemia. , 2001, Free radical biology & medicine.
[31] Christen M. Coyle,et al. Peroxynitrite induced nitration and inactivation of myofibrillar creatine kinase in experimental heart failure. , 2001, Cardiovascular research.
[32] J. Zweier,et al. Nitric oxide and peroxynitrite in postischemic myocardium. , 2001, Antioxidants & redox signaling.
[33] H. Westerblad,et al. Contractile response of skeletal muscle to low peroxide concentrations: myofibrillar calcium sensitivity as a likely target for redox-modulation. , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[34] Peitan Liu,et al. Peroxynitrite attenuates hepatic ischemia-reperfusion injury. , 2000, American journal of physiology. Cell physiology.
[35] P. Ferdinandy,et al. Peroxynitrite is a major contributor to cytokine-induced myocardial contractile failure. , 2000, Circulation research.
[36] J. Bauer,et al. Cardiac peroxynitrite formation and left ventricular dysfunction following doxorubicin treatment in mice. , 2000, The Journal of pharmacology and experimental therapeutics.
[37] L. Viera,et al. Peroxynitrite irreversibly decreases diastolic and systolic function in cardiac muscle. , 1999, Free radical biology & medicine.
[38] D. Sawyer,et al. Cytokine-mediated apoptosis in cardiac myocytes: the role of inducible nitric oxide synthase induction and peroxynitrite generation. , 1999, Circulation research.
[39] D. Warshaw,et al. Tropomyosin directly modulates actomyosin mechanical performance at the level of a single actin filament. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[40] A. Dimarco,et al. Peroxynitrite induces contractile dysfunction and lipid peroxidation in the diaphragm. , 1999, Journal of applied physiology.
[41] M. Tien,et al. Peroxynitrite-mediated modification of proteins at physiological carbon dioxide concentration: pH dependence of carbonyl formation, tyrosine nitration, and methionine oxidation. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[42] B. Lopez,et al. Peroxynitrite aggravates myocardial reperfusion injury in the isolated perfused rat heart. , 1997, Cardiovascular research.
[43] S. Cuzzocrea,et al. Protection against myocardial ischemia and reperfusion injury by 3-aminobenzamide, an inhibitor of poly (ADP-ribose) synthetase. , 1997, Cardiovascular research.
[44] H. Maeda,et al. Activation of human neutrophil procollagenase by nitrogen dioxide and peroxynitrite: a novel mechanism for procollagenase activation involving nitric oxide. , 1997, Archives of biochemistry and biophysics.
[45] Peitan Liu,et al. Formation of nitric oxide, superoxide, and peroxynitrite in myocardial ischemia-reperfusion injury in rats. , 1997, The American journal of physiology.
[46] R. Schulz,et al. Generation of peroxynitrite contributes to ischemia-reperfusion injury in isolated rat hearts. , 1997, Cardiovascular research.
[47] J. Sellers,et al. The in vitro motility activity of beta-cardiac myosin depends on the nature of the beta-myosin heavy chain gene mutation in hypertrophic cardiomyopathy. , 1997, Journal of muscle research and cell motility.
[48] J. Zweier,et al. Measurement of Nitric Oxide and Peroxynitrite Generation in the Postischemic Heart , 1996, The Journal of Biological Chemistry.
[49] D. Grier,et al. Methods of Digital Video Microscopy for Colloidal Studies , 1996 .
[50] H. Sugi,et al. Different cardiac myosin isoforms exhibit equal force-generating ability in vitro. , 1996, Biochimica et biophysica acta.
[51] H. Ischiropoulos,et al. Detection and quantitation of nitrotyrosine residues in proteins: in vivo marker of peroxynitrite. , 1996, Methods in enzymology.
[52] W. Lehman,et al. Steric-blocking by tropomyosin visualized in relaxed vertebrate muscle thin filaments. , 1995, Journal of molecular biology.
[53] A. Al-Mehdi,et al. Peroxynitrite‐mediated oxidative protein modifications , 1995, FEBS letters.
[54] J. Beckman,et al. The comparative toxicity of nitric oxide and peroxynitrite to Escherichia coli. , 1995, Archives of biochemistry and biophysics.
[55] P G Anderson,et al. Extensive nitration of protein tyrosines in human atherosclerosis detected by immunohistochemistry. , 1994, Biological chemistry Hoppe-Seyler.
[56] B. Ames,et al. Oxidants, antioxidants, and the degenerative diseases of aging. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[57] K. Trybus,et al. Smooth muscle myosin cross-bridge interactions modulate actin filament sliding velocity in vitro , 1990, The Journal of cell biology.
[58] J. Spudich,et al. Fluorescent actin filaments move on myosin fixed to a glass surface. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[59] J. D. Pardee,et al. [18] Purification of muscle actin , 1982 .
[60] J. Spudich,et al. Purification of muscle actin. , 1982, Methods in enzymology.
[61] M. Rabinowitz,et al. Measurements of half-life of rat cardiac myosin heavy chain with leucyl-tRNA used as precursor pool. , 1977, The Journal of biological chemistry.
[62] N. Alpert,et al. Purification of cardiac myosin. Application to hypertrophied myocardium. , 1975, Biochimica et biophysica acta.
[63] A. Huxley. Muscle structure and theories of contraction. , 1957, Progress in biophysics and biophysical chemistry.