Effects of flecainide on the rate dependence of atrial refractoriness, atrial repolarization and atrioventricular node conduction in anesthetized dogs.
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[1] M. Allessie,et al. Mode of Action of a New Class IC Drug (ORG 7797) Against Atrial Fibrillation in Conscious Doges , 1991, Journal of cardiovascular pharmacology.
[2] K. Lie,et al. The value of class IC antiarrhythmic drugs for acute conversion of paroxysmal atrial fibrillation or flutter to sinus rhythm. , 1990, Journal of the American College of Cardiology.
[3] T. Colatsky,et al. Channel specificity in antiarrhythmic drug action. Mechanism of potassium channel block and its role in suppressing and aggravating cardiac arrhythmias. , 1990, Circulation.
[4] S Nattel,et al. Effects of flecainide and quinidine on human atrial action potentials. Role of rate-dependence and comparison with guinea pig, rabbit, and dog tissues. , 1990, Circulation.
[5] T. Colatsky,et al. Block of delayed rectifier potassium current, IK, by flecainide and E-4031 in cat ventricular myocytes. , 1990, Circulation.
[6] S. Nattel,et al. Electrophysiological Effects of α‐Adrenergic Stimulation , 1990, Pacing and clinical electrophysiology : PACE.
[7] J. Le Heuzey,et al. Cellular electrophysiological effects of flecainide on human atrial fibres. , 1990, Cardiovascular research.
[8] D. Snyders,et al. Class III antiarrhythmic agents have a lot of potential but a long way to go. Reduced effectiveness and dangers of reverse use dependence. , 1990, Circulation.
[9] J. Davy,et al. Comparison of the Cardiac Electrophysiological Effects of Flecainide and Hydroquinidine in Anesthetized Dog: Concentration‐Response Relationship , 1990, Journal of cardiovascular pharmacology.
[10] S. Nattel,et al. Amplification of flecainide-induced ventricular conduction slowing by exercise. A potentially significant clinical consequence of use-dependent sodium channel blockade. , 1989, Circulation.
[11] J. Kingma,et al. Intravenous flecainide versus verapamil for acute conversion of paroxysmal atrial fibrillation or flutter to sinus rhythm. , 1989, The American journal of cardiology.
[12] G. Vassort,et al. Inhibition of ICa in single frog cardiac cells by quinidine, flecainide, ethmozin, and ethacizin. , 1989, The American journal of physiology.
[13] E. Antman,et al. Long-term oral propafenone therapy for suppression of refractory symptomatic atrial fibrillation and atrial flutter. , 1988, Journal of the American College of Cardiology.
[14] P. Coumel,et al. Oral flecainide for prophylaxis of paroxysmal atrial fibrillation. , 1988, The American journal of cardiology.
[15] J. Goy,et al. Restoration of sinus rhythm with flecainide in patients with atrial fibrillation. , 1988, The American journal of cardiology.
[16] I. V. Van Gelder,et al. Acute conversion of atrial fibrillation to sinus rhythm: clinical efficacy of flecainide acetate. Comparison of two regimens. , 1988, European heart journal.
[17] G. Feld,et al. Effects of N-acetylprocainamide and recainam in the pharmacologic conversion and suppression of experimental canine atrial flutter: significance of changes in refractoriness and conduction. , 1988, Journal of cardiovascular pharmacology.
[18] M R Franz,et al. Frequency-dependent effects of quinidine on the relationship between action potential duration and refractoriness in the canine heart in situ. , 1988, Circulation.
[19] G. Klein,et al. Propafenone for prevention of recurrent atrial fibrillation. , 1988, The American journal of cardiology.
[20] B. Gersh,et al. Propafenone for paroxysmal atrial fibrillation. , 1988, The American journal of cardiology.
[21] M. Jenkins,et al. Efficacy and safety of flecainide acetate for atrial tachycardia or fibrillation. , 1987, The American journal of cardiology.
[22] J. Goy,et al. Flecainide versus quinidine for conversion of atrial fibrillation to sinus rhythm. , 1986, The American journal of cardiology.
[23] R. Lal,et al. Treatment of paroxysmal reentrant supraventricular tachycardia with flecainide acetate. , 1986, The American journal of cardiology.
[24] G. Feld,et al. Pharmacologic conversion and suppression of experimental canine atrial flutter: differing effects of d-sotalol, quinidine, and lidocaine and significance of changes in refractoriness and conduction. , 1986, Circulation.
[25] M. Shenasa,et al. Evaluation of flecainide acetate in rapid atrial fibrillation complicating wolff‐parkinson‐white syndrome , 1985, Clinical cardiology.
[26] J. R. Schmid,et al. Electrophysiologic effects of flecainide acetate and its major metabolites in the canine heart. , 1985, The American journal of cardiology.
[27] L. D. Davis,et al. Effects of flecainide on the electrophysiologic properties of isolated canine and rabbit myocardial fibers. , 1985, Journal of the American College of Cardiology.
[28] E. Williams. A Classification of Antiarrhythmic Actions Reassessed After a Decade of New Drugs , 1984 .
[29] J. Ruskin,et al. Electrophysiologic properties of flecainide acetate. , 1984, The American journal of cardiology.
[30] A. Camm,et al. Cardiac electrophysiologic effects of flecainide acetate for paroxysmal reentrant junctional tachycardias. , 1983, The American journal of cardiology.
[31] B. Pitt,et al. Oral flecainide acetate for the treatment of ventricular arrhythmias. , 1981, The New England journal of medicine.
[32] J. Spear,et al. Electrophysiological Effects of a New Antiarrhythmic Agent, Flecainide, on the Intact Canine Heart , 1979, Journal of cardiovascular pharmacology.
[33] E Ruiz-Ceretti,et al. Action Potential Changes under Varied [Na+]0 and [Ca2+]0 Indicating the Existence of Two Inward Currents in Cells of the Rabbit Atrioventricular Node , 1976, Circulation research.
[34] W. Rheinboldt,et al. A COMPUTER MODEL OF ATRIAL FIBRILLATION. , 1964, American heart journal.
[35] E. Pritchett,et al. Flecainide acetate prevents recurrence of symptomatic paroxysmal supraventricular tachycardia. The Flecainide Supraventricular Tachycardia Study Group. , 1991, Circulation.
[36] S. Nattel,et al. Antiarrhythmic actions of diltiazem during experimental atrioventricular reentrant tachycardias. Importance of use-dependent calcium channel-blocking properties. , 1990, Circulation.
[37] J. Billette,et al. Atrioventricular nodal activation during periodic premature stimulation of the atrium. , 1987, The American journal of physiology.
[38] M. Allessie,et al. Experimental evaluation of Moe's multiple wavelet hypothesis of atrial fibrillation , 1985 .
[39] R. Lewis,et al. Chronic epicardial His bundle recordings in awake nonsedated dogs: a new method. , 1983, American heart journal.