The acute phase response stimulates the expression of angiopoietin like protein 4.
暂无分享,去创建一个
K. Feingold | C. Grunfeld | A. Moser | J. Shigenaga | B. Lu
[1] P. Libby. Inflammation in Atherosclerosis , 2012, Arteriosclerosis, thrombosis, and vascular biology.
[2] Robert V Farese,et al. Angiopoietin-like 4 (ANGPTL4, Fasting-induced Adipose Factor) Is a Direct Glucocorticoid Receptor Target and Participates in Glucocorticoid-regulated Triglyceride Metabolism* , 2009, The Journal of Biological Chemistry.
[3] A. Xu,et al. A Highly Conserved Motif within the NH2-terminal Coiled-coil Domain of Angiopoietin-like Protein 4 Confers Its Inhibitory Effects on Lipoprotein Lipase by Disrupting the Enzyme Dimerization* , 2009, Journal of Biological Chemistry.
[4] J. Olefsky,et al. Insulin sensitivity: modulation by nutrients and inflammation. , 2008, The Journal of clinical investigation.
[5] S. Corvera,et al. Enhanced angiogenesis in obesity and in response to PPARgamma activators through adipocyte VEGF and ANGPTL4 production. , 2008, American journal of physiology. Endocrinology and metabolism.
[6] M. Morishima,et al. Bezafibrate improves bacterial lipopolysaccharide-induced dyslipidemia and anorexia in rats. , 2007, Metabolism: clinical and experimental.
[7] G. Hotamisligil,et al. Inflammation and metabolic disorders , 2006, Nature.
[8] T. Olivecrona,et al. Angiopoietin-like protein 4 converts lipoprotein lipase to inactive monomers and modulates lipase activity in adipose tissue , 2006, Proceedings of the National Academy of Sciences.
[9] K. Feingold,et al. Type II nuclear hormone receptors, coactivator, and target gene repression in adipose tissue in the acute-phase response Published, JLR Papers in Press, July 17, 2006. , 2006, Journal of Lipid Research.
[10] Cai Li. Genetics and regulation of angiopoietin-like proteins 3 and 4 , 2006, Current opinion in lipidology.
[11] J. Cha,et al. Differential regulation and properties of angiopoietin-like proteins 3 and 4 Published, JLR Papers in Press, May 1, 2005. DOI 10.1194/jlr.M500005-JLR200 , 2005, Journal of Lipid Research.
[12] Kenneth R Feingold,et al. Effects of infection and inflammation on lipid and lipoprotein metabolism: mechanisms and consequences to the host. , 2004, Journal of lipid research.
[13] J. Pickup. Inflammation and activated innate immunity in the pathogenesis of type 2 diabetes. , 2004, Diabetes care.
[14] K. Feingold,et al. Altered expression of nuclear hormone receptors and coactivators in mouse heart during the acute-phase response. , 2004, American journal of physiology. Endocrinology and metabolism.
[15] Kenichi Yoshida,et al. Angiopoietin-like protein 4 is a potent hyperlipidemia-inducing factor in mice and inhibitor of lipoprotein lipase Published, JLR Papers in Press, September 16, 2002. DOI 10.1194/jlr.C200010-JLR200 , 2002, Journal of Lipid Research.
[16] P. Chambon,et al. Characterization of the Fasting-induced Adipose Factor FIAF, a Novel Peroxisome Proliferator-activated Receptor Target Gene* , 2000, The Journal of Biological Chemistry.
[17] K. Feingold,et al. The Acute Phase Response Is Associated with Retinoid X Receptor Repression in Rodent Liver* , 2000, The Journal of Biological Chemistry.
[18] I. Kushner,et al. Acute-phase proteins and other systemic responses to inflammation. , 1999, The New England journal of medicine.
[19] K. Feingold,et al. Cytokines induce catabolic effects in cultured adipocytes by multiple mechanisms. , 1994, Cytokine.
[20] J. Gauldie,et al. The acute phase response. , 1994, Immunology today.
[21] S. Fried,et al. Cachectin/tumor necrosis factor decreases human adipose tissue lipoprotein lipase mRNA levels, synthesis, and activity. , 1989, Journal of lipid research.
[22] K. Feingold,et al. Effect of tumor necrosis factor administration in vivo on lipoprotein lipase activity in various tissues of the rat. , 1989, Journal of lipid research.
[23] T. Chajek-Shaul,et al. Lipoprotein lipase in heart cell cultures is suppressed by bacterial lipopolysaccharide: an effect mediated by production of tumor necrosis factor. , 1988, Biochimica et biophysica acta.
[24] J. Tavernier,et al. Tissue-specific regulation of guinea pig lipoprotein lipase; effects of nutritional state and of tumor necrosis factor on mRNA levels in adipose tissue, heart and liver. , 1988, Gene.
[25] S. Enerbäck,et al. Regulation of lipoprotein lipase mRNA content in 3T3-L1 cells by tumour necrosis factor. , 1988, The Biochemical journal.
[26] G. Bagby,et al. Triacylglycerol kinetics in endotoxic rats with suppressed lipoprotein lipase activity. , 1987, The American journal of physiology.
[27] J. Tavernier,et al. Multiple effects of tumor necrosis factor on lipoprotein lipase in vivo. , 1987, The Journal of biological chemistry.
[28] M. Kawakami,et al. Human recombinant TNF suppresses lipoprotein lipase activity and stimulates lipolysis in 3T3-L1 cells. , 1987, Journal of biochemistry.
[29] B. Aggarwal,et al. Interferons and tumor necrosis factors have similar catabolic effects on 3T3 L1 cells. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[30] B. Beutler,et al. Purification of cachectin, a lipoprotein lipase-suppressing hormone secreted by endotoxin-induced RAW 264.7 cells , 1985, The Journal of experimental medicine.
[31] A. Cerami,et al. Studies of endotoxin-induced decrease in lipoprotein lipase activity , 1981, The Journal of experimental medicine.
[32] Richard Graham Knowles,et al. Bacterial endotoxin effects on carbohydrate utilization and transport , 2009 .
[33] M. Tabata,et al. The role of angiopoietin-like proteins in angiogenesis and metabolism. , 2008, Trends in cardiovascular medicine.
[34] S. Kersten. Regulation of lipid metabolism via angiopoietin-like proteins. , 2005, Biochemical Society transactions.
[35] L. Chan,et al. Endotoxin-induced hypertriglyceridemia is mediated by suppression of lipoprotein lipase at a post-transcriptional level. , 1993, Journal of lipid research.