Effects of antiepileptic drug therapy on vitamin D status and biochemical markers of bone turnover in children with epilepsy
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G. Brabant | M. Wolters | I. Tuxhorn | R. Lichtinghagen | A. Ströhle | A. Hahn | H. Welkoborsky | S. Nettekoven | B. Trunz | S. Hoffmann | R. Horn | M. Steinert
[1] O. Witte,et al. Long-term anticonvulsant therapy leads to low bone mineral density--evidence for direct drug effects of phenytoin and carbamazepine on human osteoblast-like cells. , 2012, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.
[2] F. Spener,et al. Conjugated linoleic acids as functional food: an insight into their health benefits , 2009, Nutrition & metabolism.
[3] Gert B. M. Mensink,et al. Vitamin D status and health correlates among German adults , 2008, European Journal of Clinical Nutrition.
[4] C. Mølgaard,et al. Pakistani immigrant children and adults in Denmark have severely low vitamin D status , 2008, European Journal of Clinical Nutrition.
[5] D. Papadimitriou,et al. Thyroid dysfunction associated with increased low-density lipoprotein cholesterol in epileptic children treated with carbamazepine monotherapy: a causal relationship? , 2007, European journal of paediatric neurology : EJPN : official journal of the European Paediatric Neurology Society.
[6] P. Vestergaard,et al. Adverse effects of antiepileptic drugs on bone mineral density in children , 2007, Expert opinion on drug safety.
[7] H. Bischoff-Ferrari. The 25-hydroxyvitamin D threshold for better health , 2007, The Journal of Steroid Biochemistry and Molecular Biology.
[8] J. Wark,et al. Anti-epileptic medication and bone health , 2007, Osteoporosis International.
[9] V. Stallings,et al. Vitamin D Status in Children with Intractable Epilepsy, and Impact of the Ketogenic Diet , 2007, Epilepsia.
[10] A. Attilakos,et al. Early effect of sodium valproate and carbamazepine monotherapy on homocysteine metabolism in children with epilepsy , 2006, Epilepsy Research.
[11] H. Valsamis,et al. Antiepileptic drugs and bone metabolism , 2006, Nutrition & metabolism.
[12] M. Holick,et al. The role of vitamin D for bone health and fracture prevention , 2006, Current osteoporosis reports.
[13] R. Vieth. What is the optimal vitamin D status for health? , 2006, Progress in biophysics and molecular biology.
[14] A. Attilakos,et al. Early and persistent increase in serum lipoprotein (a) concentrations in epileptic children treated with carbamazepine and sodium valproate monotherapy , 2006, Epilepsy Research.
[15] W. Willett,et al. Estimation of optimal serum concentrations of 25-hydroxyvitamin D for multiple health outcomes. , 2006, The American journal of clinical nutrition.
[16] P. Watkins,et al. Steroid and xenobiotic receptor and vitamin D receptor crosstalk mediates CYP24 expression and drug-induced osteomalacia. , 2006, The Journal of clinical investigation.
[17] S. Mintzer,et al. Vitamin D Levels and Bone Turnover in Epilepsy Patients Taking Carbamazepine or Oxcarbazepine , 2006, Epilepsia.
[18] Y. Matsinos,et al. Effects of Anticonvulsant Therapy on Vitamin D Status in Children: Prospective Monitoring Study , 2006, Journal of child neurology.
[19] B. Mark,et al. Vitamin D and autoimmune disease--implications for practice from the multiple sclerosis literature. , 2006, Journal of the American Dietetic Association.
[20] M. Topbaş,et al. Effect of Antiepileptic Drugs on Plasma Lipids, Lipoprotein (a), and Liver Enzymes , 2006, Journal of child neurology.
[21] A. Ponsonby,et al. UVR, Vitamin D and Three Autoimmune Diseases—Multiple Sclerosis, Type 1 Diabetes, Rheumatoid Arthritis , 2005, Photochemistry and photobiology.
[22] Kazutoshi Nakamura,et al. Nutrition, mild hyperparathyroidism, and bone mineral density in young Japanese women. , 2005, The American journal of clinical nutrition.
[23] A. Kalueff,et al. Anticonvulsant effects of 1,25-dihydroxyvitamin D in chemically induced seizures in mice , 2005, Brain Research Bulletin.
[24] W. Grant,et al. Benefits and requirements of vitamin D for optimal health: a review. , 2005, Alternative medicine review : a journal of clinical therapeutic.
[25] V. Matkovic,et al. Potential use of biochemical markers of bone turnover for assessing the effect of calcium supplementation and predicting fracture risk. , 2005, Clinical therapeutics.
[26] Pascal G. P. Martin,et al. Possible involvement of pregnane X receptor-enhanced CYP24 expression in drug-induced osteomalacia. , 2005, The Journal of clinical investigation.
[27] J. Bilezikian,et al. Bone mineral density and serum levels of 25 OH vitamin D in chronic users of antiepileptic drugs. , 2004, Arquivos de neuro-psiquiatria.
[28] Ç. Ecevit,et al. Effect of carbamazepine and valproate on bone mineral density. , 2004, Pediatric neurology.
[29] G. Mishra,et al. Effects of usual nutrient intake and vitamin D status on markers of bone turnover in Swiss adolescents , 2004, European Journal of Clinical Nutrition.
[30] S. Karasalihoǧlu,et al. Bone mineral metabolism changes in epileptic children receiving valproic acid , 2004, Journal of paediatrics and child health.
[31] R. Vieth. Why the optimal requirement for Vitamin D3 is probably much higher than what is officially recommended for adults , 2004, The Journal of Steroid Biochemistry and Molecular Biology.
[32] L. Fitzpatrick. Pathophysiology of bone loss in patients receiving anticonvulsant therapy , 2004, Epilepsy & Behavior.
[33] F. Tylavsky,et al. Association of low 25-hydroxyvitamin D concentrations with elevated parathyroid hormone concentrations and low cortical bone density in early pubertal and prepubertal Finnish girls. , 2003, The American journal of clinical nutrition.
[34] B. Otlu,et al. Effects of valproate and carbamazepine on serum levels of homocysteine, vitamin B12, and folic acid , 2003, Brain and Development.
[35] J. Viikari,et al. Vitamin D and attainment of peak bone mass among peripubertal Finnish girls: a 3-y prospective study. , 2002, The American journal of clinical nutrition.
[36] G. Latini,et al. Increased Bone Turnover in Prepubertal, Pubertal, and Postpubertal Patients Receiving Carbamazepine , 2002, Epilepsia.
[37] M. Moustaki,et al. Alkaline phosphatase and its isoenzyme activity for the evaluation of bone metabolism in children receiving anticonvulsant monotherapy , 2002, Seizure.
[38] W. Spain,et al. Antiepileptic drug-induced bone loss in young male patients who have seizures. , 2002, Archives of neurology.
[39] Shlomo Shinnar,et al. Update on the Epidemiology and Prognosis of Pediatric Epilepsy , 2002, Journal of child neurology.
[40] F. Rauch,et al. Analysis of the musculoskeletal system in children and adolescents receiving anticonvulsant monotherapy with valproic acid or carbamazepine. , 2001, Pediatrics.
[41] K. Nakamura,et al. Low serum concentrations of 25-hydroxyvitamin D in young adult Japanese women: a cross sectional study. , 2001, Nutrition.
[42] S. Atkinson,et al. Long‐Term Valproate and Lamotrigine Treatment May Be a Marker for Reduced Growth and Bone Mass in Children with Epilepsy , 2001, Epilepsia.
[43] K. Satoh,et al. Decreased bone mass and increased bone turnover with valproate therapy in adults with epilepsy [RETRACTED] , 2001, Neurology.
[44] M. Kärkkäinen,et al. Vitamin D status affects serum parathyroid hormone concentrations during winter in female adolescents: associations with forearm bone mineral density. , 2001, The American journal of clinical nutrition.
[45] R. Vieth,et al. Efficacy and safety of vitamin D3 intake exceeding the lowest observed adverse effect level. , 2001, The American journal of clinical nutrition.
[46] M. Castro‐Gago,et al. The effects on lipid and apolipoprotein serum levels of long-term carbamazepine, valproic acid and phenobarbital therapy in children with epilepsy , 2000, Epilepsy Research.
[47] F. Chiarelli,et al. Increased bone turnover in epileptic patients treated with carbamazepine , 2000, Annals of neurology.
[48] G Pearce,et al. Moderate exercise during growth in prepubertal boys: changes in bone mass, size, volumetric density, and bone strength: a controlled prospective study. , 1999, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[49] T. Erselcan,et al. Effect of Antiepileptic Drugs on Bone Mineral Density in Children Between Ages 6 and 2 Years , 1999, Clinical pediatrics.
[50] V. Okutan,et al. Evaluation of bone mineral density in children receiving antiepileptic drugs. , 1998, Pediatric neurology.
[51] K. Tsai,et al. Effect of Exercise and Exogenous Glucocorticoid on Serum Level of Intact Parathyroid Hormone , 1997, International journal of sports medicine.
[52] O. Doğu,et al. Serum lipids in epileptic children treated with carbamazepine and valproate , 1997, European Journal of Pediatrics.
[53] M. T. Baer,et al. Vitamin D, calcium, and bone status in children with developmental delay in relation to anticonvulsant use and ambulatory status. , 1997, The American journal of clinical nutrition.
[54] S. Bloomfield,et al. Bone mass and endocrine adaptations to training in spinal cord injured individuals. , 1996, Bone.
[55] M. Holick,et al. McCollum Award Lecture, 1994: vitamin D--new horizons for the 21st century. , 1994, The American journal of clinical nutrition.
[56] M. Välimäki,et al. Bone mineral density measured by dual‐energy X‐ray absorptiometry and novel markers of bone formation and resorption in patients on antiepileptic drugs , 1994, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[57] N. Callaghan,et al. A comparative study of the relative influence of different anticonvulsant drugs, UV exposure and diet on vitamin D and calcium metabolism in out-patients with epilepsy. , 1986, The Quarterly journal of medicine.
[58] P. Gosling. Analytical Reviews in Clinical Biochemistry: Calcium Measurement , 1986, Annals of clinical biochemistry.
[59] D. Lawson,et al. Low plasma 25-hydroxyvitamin D and serum calcium levels in institutionalized epileptic subjects: associated risk factors, consequences and response to treatment with vitamin D. , 1983, The Quarterly journal of medicine.
[60] A. Kaushik,et al. Altered vitamin-D metabolism due to anticonvulsant drugs in epileptic children. , 1982, Indian pediatrics.
[61] K. Kruse,et al. Response of Kidney and Bone to Parathyroid Hormone in Children Receiving Anticonvulsant Drugs , 1982, Neuropediatrics.
[62] J. Sivenius,et al. Osteomalacia in institutionalized epileptic patients on long‐term anticonvulsant therapy , 1981, Acta neurologica Scandinavica.
[63] T. Sato,et al. Factors causing rickets in institutionalised handicapped children on anticonvulsant therapy. , 1981, Archives of disease in childhood.
[64] J. Winnacker,et al. Rickets in children receiving anticonvulsant drugs. Biochemical and hormonal markers. , 1977, American journal of diseases of children.
[65] J. Haddad,et al. Serum 25-Hydroxycalciferol Levels and Bone Mass in Children on Chronic Anticonvulsant Therapy , 1975 .
[66] J. Maxwell,et al. Altered Calcium Metabolism in Epileptic Children on Anticonvulsants , 1971, British medical journal.
[67] A. Richens,et al. Osteomalacia with Long-term Anticonvulsant Therapy in Epilepsy , 1970, British medical journal.
[68] L. Cowan. The epidemiology of the epilepsies in children. , 2002, Mental retardation and developmental disabilities research reviews.
[69] R. Lorentzon,et al. Influence of Heredity and Environment on Bone Density in Adolescent Boys: A Parent–Offspring Study , 1999, Osteoporosis International.
[70] A. von zur Mühlen,et al. Pulse amplitude and frequency modulation of parathyroid hormone in primary hyperparathyroidism. , 1994, The Journal of clinical endocrinology and metabolism.
[71] P. Hauschka,et al. Phenytoin affects osteocalcin secretion from osteoblastic rat osteosarcoma 17/2.8 cells in culture. , 1990, Bone.
[72] P. Berlit,et al. Effect of long-term treatment with antiepileptic drugs on the vitamin status. , 1988, Drug-nutrient interactions.
[73] B. Köhler,et al. Rickets in Children Receiving Anticonvulsant Drugs , 1988 .
[74] B. Lawrence Riggs,et al. Osteoporosis : etiology, diagnosis, and management , 1988 .
[75] L. Arab,et al. B vitamins in epileptics. , 1986, Bibliotheca nutritio et dieta.
[76] C. Christiansen,et al. Pathophysiology behind anticonvulsant osteomalacia. , 1983, Acta neurologica Scandinavica. Supplementum.
[77] P. Berlit,et al. Vitamin status in patients on chronic anticonvulsant therapy. , 1982, International journal for vitamin and nutrition research. Internationale Zeitschrift fur Vitamin- und Ernahrungsforschung. Journal international de vitaminologie et de nutrition.