Neurotoxic effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin in cerebellar granule cells

[1]  S. Choi,et al.  Serotonin transporter mRNA expression in the dorsal raphe nucleus of a tumor bearing mouse , 2005, Experimental & Molecular Medicine.

[2]  R. A. Butler,et al.  Aryl hydrocarbon receptor (AhR)-independent effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on softshell clam (Mya arenaria) reproductive tissue. , 2004, Comparative biochemistry and physiology. Toxicology & pharmacology : CBP.

[3]  P. Kodavanti,et al.  Alterations in brain protein kinase C isoforms following developmental exposure to a polychlorinated biphenyl mixture. , 2003, Brain research. Molecular brain research.

[4]  Masaki Kakeyama,et al.  Perinatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin alters activity-dependent expression of BDNF mRNA in the neocortex and male rat sexual behavior in adulthood. , 2003, Neurotoxicology.

[5]  E. Jakobsson,et al.  A brominated flame retardant, 2,2',4,4',5-pentabromodiphenyl ether: uptake, retention, and induction of neurobehavioral alterations in mice during a critical phase of neonatal brain development. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.

[6]  Anders Fredriksson,et al.  Neonatal exposure to the brominated flame retardant 2,2',4,4',5-pentabromodiphenyl ether causes altered susceptibility in the cholinergic transmitter system in the adult mouse. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.

[7]  F. Fonnum,et al.  The polychlorinated biphenyl mixture aroclor 1254 induces death of rat cerebellar granule cells: the involvement of the N-methyl-D-aspartate receptor and reactive oxygen species. , 2002, Toxicology and applied pharmacology.

[8]  E. Jakobsson,et al.  Brominated flame retardants: a novel class of developmental neurotoxicants in our environment? , 2001, Environmental health perspectives.

[9]  P. Kodavanti,et al.  Possible molecular targets of halogenated aromatic hydrocarbons in neuronal cells. , 2001, Biochemical and biophysical research communications.

[10]  R. Burghardt,et al.  2,3,7,8-Tetrachlorodibenzo-p-dioxin alters hippocampal astroglia-neuronal gap junctional communication. , 2000, Neurotoxicology.

[11]  M. Berridge,et al.  The versatility and universality of calcium signalling , 2000, Nature Reviews Molecular Cell Biology.

[12]  F. Fonnum,et al.  The effects of aliphatic (n-nonane), naphtenic (1,2, 4-trimethylcyclohexane), and aromatic (1,2,4-trimethylbenzene) hydrocarbons on respiratory burst in human neutrophil granulocytes. , 2000, Toxicology and applied pharmacology.

[13]  P. Eriksson,et al.  Neonatal exposure to neurotoxic pesticides increases adult susceptibility: a review of current findings. , 2000, Neurotoxicology.

[14]  S. Lipton,et al.  ■ REVIEW : Excitotoxicity, Free Radicals, Necrosis, and Apoptosis , 1998 .

[15]  G. Keilhoff,et al.  2,7‐Dihydrodichlorofluorescein diacetate as a fluorescent marker for peroxynitrite formation , 1997, FEBS letters.

[16]  T. Taniguchi,et al.  713 Changes in Ca2+-dependent and -independent protein kinase C in Alzheimer's Disease , 1996, Neurobiology of Aging.

[17]  D. Carpenter,et al.  The translocation and involvement of protein kinase C in mossy fiber-CA3 long-term potentiation in hippocampus of the rat brain , 1996, Brain Research.

[18]  A. Puga,et al.  Dioxin induces transcription of fos and jun genes by Ah receptor-dependent and -independent pathways. , 1996, Toxicology and applied pharmacology.

[19]  S. Safe,et al.  Stimulation of calcium uptake in cultured rat hippocampal neurons by 2,3,7,8-tetrachlorodibenzo-p-dioxin. , 1996, Toxicology.

[20]  S. Lipton,et al.  Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function , 1995, Neuron.

[21]  I. Reynolds,et al.  Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[22]  H. Tilson,et al.  Differential effects of polychlorinated biphenyl congeners on phosphoinositide hydrolysis and protein kinase C translocation in rat cerebellar granule cells , 1994, Brain Research.

[23]  Abraham Brouwer,et al.  Effects of Dioxins and Polychlorinated Biphenyls on Thyroid Hormone Status of Pregnant Women and Their Infants , 1994, Pediatric Research.

[24]  H. Manji,et al.  Lithium Decreases Membrane‐Associated Protein Kinase C in Hippocampus: Selectivity for the α Isozyme , 1993, Journal of neurochemistry.

[25]  Kuo-ping Huang,et al.  How is protein kinase C activated in CNS , 1993, Neurochemistry International.

[26]  M. Miras-Portugal,et al.  Positive feedback of glutamate exocytosis by metabotropic presynaptic receptor stimulation , 1992, Nature.

[27]  H. Kawasaki,et al.  Enzymatic properties of a novel phorbol ester receptor/protein kinase, nPKC. , 1989, Journal of biochemistry.

[28]  Y. Nishizuka,et al.  The molecular heterogeneity of protein kinase C and its implications for cellular regulation , 1988, Nature.

[29]  N. Zawia,et al.  Transplacental effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on the temporal modulation of Sp1 DNA binding in the developing cerebral cortex and cerebellum. , 2002, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.

[30]  G. Rimbach,et al.  Molecular aspects of alpha-tocotrienol antioxidant action and cell signalling. , 2001, The Journal of nutrition.

[31]  C. Vogel,et al.  A malignant transformation of human cells by 2,3,7,8-tetrachlorodibenzo-p-dioxin exhibits altered expressions of growth regulatory factors. , 1999, Carcinogenesis.

[32]  P. Eriksson Developmental neurotoxicity of environmental agents in the neonate. , 1997, Neurotoxicology.

[33]  C. Tanaka,et al.  Cellular and intracellular localization of epsilon-subspecies of protein kinase C in the rat brain; presynaptic localization of the epsilon-subspecies. , 1993, Brain research.

[34]  P. Greengard,et al.  Protein phosphorylation and neuronal function. , 1991, Pharmacological reviews.